Hiroshi Itoh

7.9k total citations
150 papers, 6.5k citations indexed

About

Hiroshi Itoh is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Hiroshi Itoh has authored 150 papers receiving a total of 6.5k indexed citations (citations by other indexed papers that have themselves been cited), including 102 papers in Molecular Biology, 22 papers in Cell Biology and 18 papers in Cellular and Molecular Neuroscience. Recurrent topics in Hiroshi Itoh's work include Protein Kinase Regulation and GTPase Signaling (41 papers), Receptor Mechanisms and Signaling (26 papers) and Melanoma and MAPK Pathways (13 papers). Hiroshi Itoh is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (41 papers), Receptor Mechanisms and Signaling (26 papers) and Melanoma and MAPK Pathways (13 papers). Hiroshi Itoh collaborates with scholars based in Japan, United States and Indonesia. Hiroshi Itoh's co-authors include Y Kaziro, Tohru Kozasa, Yoshito Kaziro, Junji Yamauchi, Norikazu Mizuno, Tetsuya Tsukamoto, Kenji Tago, Masao Matsuoka, Motoshi Nagao and Shun Nakamura and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Hiroshi Itoh

146 papers receiving 6.4k citations

Peers

Hiroshi Itoh
Masahiko Hoshijima United States
Bradley A. Schulte United States
Nancy D. Dalton United States
David C. West United Kingdom
Lin Gao China
Liqun He Sweden
Masahiko Hoshijima United States
Hiroshi Itoh
Citations per year, relative to Hiroshi Itoh Hiroshi Itoh (= 1×) peers Masahiko Hoshijima

Countries citing papers authored by Hiroshi Itoh

Since Specialization
Citations

This map shows the geographic impact of Hiroshi Itoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroshi Itoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroshi Itoh more than expected).

Fields of papers citing papers by Hiroshi Itoh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroshi Itoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroshi Itoh. The network helps show where Hiroshi Itoh may publish in the future.

Co-authorship network of co-authors of Hiroshi Itoh

This figure shows the co-authorship network connecting the top 25 collaborators of Hiroshi Itoh. A scholar is included among the top collaborators of Hiroshi Itoh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroshi Itoh. Hiroshi Itoh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kaji, Noriko, et al.. (2025). Functional Analysis of Adhesion GPCR Latrophilin 2 (ADGRL2) in MDAMB‐231 Human Breast Cancer Cells. Genes to Cells. 30(4). e70030–e70030.
2.
Toriyama, Michinori, et al.. (2024). Astrocyte-Specific Inhibition of the Primary Cilium Suppresses C3 Expression in Reactive Astrocyte. Cellular and Molecular Neurobiology. 44(1). 48–48. 2 indexed citations
3.
Kobayashi, Tetsuo, et al.. (2023). CEP164-GLI2 association ensures the hedgehog signaling in pancreatic cancer cells. Biochemical and Biophysical Research Communications. 666. 179–185. 1 indexed citations
4.
Nakamura, Motoki, Michinori Toriyama, Fumitaka Fujita, et al.. (2023). Dendritic cell proliferation by primary cilium in atopic dermatitis. Frontiers in Molecular Biosciences. 10. 1149828–1149828. 7 indexed citations
5.
Morita, Tomoka, et al.. (2023). Cholesterol in the ciliary membrane as a therapeutic target against cancer. Frontiers in Molecular Biosciences. 10. 1160415–1160415. 4 indexed citations
6.
Itoh, Hiroshi, et al.. (2023). Different Modes of Mechanism of Gamma-Mangostin and Alpha-Mangostin to Inhibit Cell Migration of Triple-Negative Breast Cancer Cells Concerning CXCR4 Downregulation and ROS Generation. Iranian journal of pharmaceutical research. 22(1). e138856–e138856. 3 indexed citations
7.
Kobayashi, Tetsuo, et al.. (2022). The atypical small GTPase RABL3 interacts with RAB11 to regulate early ciliogenesis in human cells. Journal of Cell Science. 135(18). 4 indexed citations
8.
Ohashi, Shinya, Mitsuteru Natsuizaka, Seiji Naganuma, et al.. (2011). A NOTCH3-Mediated Squamous Cell Differentiation Program Limits Expansion of EMT-Competent Cells That Express the ZEB Transcription Factors. Cancer Research. 71(21). 6836–6847. 89 indexed citations
9.
Iguchi, Tokuichi, et al.. (2008). Orphan G Protein-coupled Receptor GPR56 Regulates Neural Progenitor Cell Migration via a Gα12/13 and Rho Pathway. Journal of Biological Chemistry. 283(21). 14469–14478. 161 indexed citations
10.
Miyamoto, Yuki, Junji Yamauchi, & Hiroshi Itoh. (2003). Src Kinase Regulates the Activation of a Novel FGD-1-related Cdc42 Guanine Nucleotide Exchange Factor in the Signaling Pathway from the Endothelin A Receptor to JNK. Journal of Biological Chemistry. 278(32). 29890–29900. 53 indexed citations
11.
Takei, Yoshifumi, Kenji Kadomatsu, Hiroshi Itoh, et al.. (2002). 5′-,3′-Inverted Thymidine-modified Antisense Oligodeoxynucleotide Targeting Midkine. Journal of Biological Chemistry. 277(26). 23800–23806. 46 indexed citations
12.
Edamatsu, Hironori, Yoshito Kaziro, & Hiroshi Itoh. (2002). Differential Display of mRNAs Regulated by G-Protein Signaling. Methods in enzymology on CD-ROM/Methods in enzymology. 345. 521–529. 2 indexed citations
13.
Horikawa, Tatsuya, Ashok K. Chakraborty, Koichi Nakagawa, et al.. (2000). Small GTPase Rab3A is Associated with Melanosomes in Melanoma Cells. Pigment Cell Research. 13(5). 332–336. 28 indexed citations
14.
Yamaguchi, Tomohiro, Masami Nagahama, Hiroshi Itoh, et al.. (2000). Regulation of the Golgi structure by the α subunits of heterotrimeric G proteins. FEBS Letters. 470(1). 25–28. 15 indexed citations
15.
Yamauchi, Junji, et al.. (2000). NESK, a Member of the Germinal Center Kinase Family That Activates the c-Jun N-terminal Kinase Pathway and Is Expressed during the Late Stages of Embryogenesis. Journal of Biological Chemistry. 275(27). 20533–20539. 38 indexed citations
16.
Ueda, Hiroshi, Junji Yamauchi, Hiroshi Itoh, et al.. (1999). Phosphorylation of F-actin-Associating G Protein γ12 Subunit Enhances Fibroblast Motility. Journal of Biological Chemistry. 274(17). 12124–12128. 12 indexed citations
17.
Hayashi, Yoshitake, Takashi Itoh, Satoshi Suzuki, et al.. (1999). Inhibitory Role of Plasminogen Activator Inhibitor‐1 in Invasion and Proliferation of HLE Hepatocellular Carcinoma Cells. Japanese Journal of Cancer Research. 90(7). 747–752. 23 indexed citations
18.
Itoh, Hiroshi, et al.. (1997). Expression of S-100 Protein and Neuron-Specific Enolase in the Developing and Adult Human Retina : An Immunohistochemical Study on 24 Cases. Kobe University Repository Kernel (Kobe University). 12(12). 115–122.
19.
Ito, Akiko, Takaya Satoh, Yoshito Kaziro, & Hiroshi Itoh. (1995). G protein βγ subunit activates Ras, Raf, and MAP kinase in HEK 293 cells. FEBS Letters. 368(1). 183–187. 47 indexed citations
20.
Itoh, Hiroshi, et al.. (1991). H∞ Control of a Plant With Redundant Degree of Freedom. 제어로봇시스템학회 국내학술대회 논문집. 1(2). 1493–1496. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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