Hiroko Ushikubo

923 total citations
19 papers, 778 citations indexed

About

Hiroko Ushikubo is a scholar working on Molecular Biology, Ophthalmology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Hiroko Ushikubo has authored 19 papers receiving a total of 778 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Ophthalmology and 6 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Hiroko Ushikubo's work include Retinal Diseases and Treatments (8 papers), Retinal Development and Disorders (6 papers) and Retinopathy of Prematurity Studies (5 papers). Hiroko Ushikubo is often cited by papers focused on Retinal Diseases and Treatments (8 papers), Retinal Development and Disorders (6 papers) and Retinopathy of Prematurity Studies (5 papers). Hiroko Ushikubo collaborates with scholars based in Japan and United States. Hiroko Ushikubo's co-authors include Masahiro Ono, Teruo Yasunaga, Takashi Miyata, M Kawakami, Tsutomu Nakahara, Asami Mori, Kenji Sakamoto, Kunio Ishii, Miwa Misawa and Hiroyasu Sakai and has published in prestigious journals such as Journal of Virology, International Journal of Molecular Sciences and European Journal of Pharmacology.

In The Last Decade

Hiroko Ushikubo

19 papers receiving 757 citations

Peers

Hiroko Ushikubo
Lili Gong China
Fei Ji United States
Hiroko Ushikubo
Citations per year, relative to Hiroko Ushikubo Hiroko Ushikubo (= 1×) peers Zhengyu Zhang

Countries citing papers authored by Hiroko Ushikubo

Since Specialization
Citations

This map shows the geographic impact of Hiroko Ushikubo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroko Ushikubo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroko Ushikubo more than expected).

Fields of papers citing papers by Hiroko Ushikubo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroko Ushikubo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroko Ushikubo. The network helps show where Hiroko Ushikubo may publish in the future.

Co-authorship network of co-authors of Hiroko Ushikubo

This figure shows the co-authorship network connecting the top 25 collaborators of Hiroko Ushikubo. A scholar is included among the top collaborators of Hiroko Ushikubo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroko Ushikubo. Hiroko Ushikubo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Watanabe, Koki, Hiroko Ushikubo, Asami Mori, et al.. (2021). Metformin Protects against NMDA-Induced Retinal Injury through the MEK/ERK Signaling Pathway in Rats. International Journal of Molecular Sciences. 22(9). 4439–4439. 20 indexed citations
2.
Mori, Asami, Shiho Arima, Hiroko Ushikubo, et al.. (2018). Retinal neuronal cell loss prevents abnormal retinal vascular growth in a rat model of retinopathy of prematurity. Experimental Eye Research. 168. 115–127. 10 indexed citations
3.
Sakamoto, Kenji, et al.. (2017). Activation inhibitors of nuclear factor kappa B protect neurons against the NMDA-induced damage in the rat retina. Journal of Pharmacological Sciences. 135(2). 72–80. 23 indexed citations
4.
Sakamoto, Kenji, Yuta Murakami, Shohei Sawada, et al.. (2016). Apelin-36 is protective against N-methyl-D-aspartic-acid-induced retinal ganglion cell death in the mice. European Journal of Pharmacology. 791. 213–220. 21 indexed citations
5.
Ushikubo, Hiroko, et al.. (2016). Exposure to high‐concentration oxygen in the neonatal period induces abnormal retinal vascular patterning in mice. Birth Defects Research Part B Developmental and Reproductive Toxicology. 107(6). 216–224. 8 indexed citations
6.
Ushikubo, Hiroko, Asami Mori, Shiho Arima, et al.. (2016). A delay in vascularization induces abnormal astrocyte proliferation and migration in the mouse retina. Developmental Dynamics. 246(3). 186–200. 15 indexed citations
7.
Ushikubo, Hiroko, et al.. (2016). Protective effects of PF‐4708671 against N‐methyl‐d‐aspartic acid‐induced retinal damage in rats. Fundamental and Clinical Pharmacology. 30(6). 529–536. 5 indexed citations
8.
Nakahara, Tsutomu, et al.. (2015). Retinal region-dependent susceptibility of capillaries to high-concentration oxygen exposure and vascular endothelial growth factor receptor inhibition in neonatal mice. Journal of Pharmacological Sciences. 129(2). 107–118. 7 indexed citations
9.
Sakamoto, Masaya, Hiroko Ushikubo, Hirofumi Michimae, et al.. (2015). Expression of UDP-Glucuronosyltransferase 1 (UGT1) and Glucuronidation Activity toward Endogenous Substances in Humanized UGT1 Mouse Brain. Drug Metabolism and Disposition. 43(7). 1071–1076. 22 indexed citations
10.
Nakahara, Tsutomu, et al.. (2015). Short-term treatment with VEGF receptor inhibitors induces retinopathy of prematurity-like abnormal vascular growth in neonatal rats. Experimental Eye Research. 143. 120–131. 21 indexed citations
11.
Ushikubo, Hiroko, et al.. (2015). Protective Effects of Everolimus against <i>N</i>-Methyl-D-aspartic Acid-Induced Retinal Damage in Rats. Biological and Pharmaceutical Bulletin. 38(11). 1765–1771. 12 indexed citations
12.
Nakahara, Tsutomu, et al.. (2015). Preventive Effects of Rapamycin on Inflammation and Capillary Degeneration in a Rat Model of NMDA-Induced Retinal Injury. Biological and Pharmaceutical Bulletin. 38(2). 321–324. 12 indexed citations
13.
Ushikubo, Hiroko, et al.. (2014). 3,3′,4′,5′-Tetrahydroxyflavone Induces Formation of Large Aggregates of Amyloid β Protein. Biological and Pharmaceutical Bulletin. 37(5). 748–754. 10 indexed citations
14.
Nakahara, Tsutomu, et al.. (2014). Anti-angiogenic Effects of Mammalian Target of Rapamycin Inhibitors in a Mouse Model of Oxygen-Induced Retinopathy. Biological and Pharmaceutical Bulletin. 37(11). 1838–1842. 41 indexed citations
15.
Nakahara, Tsutomu, et al.. (2014). Treatment of Newborn Mice with Inhibitors of Vascular Endothelial Growth Factor Receptor Tyrosine Kinase Induces Abnormal Retinal Vascular Patterning. Biological and Pharmaceutical Bulletin. 37(12). 1986–1989. 5 indexed citations
16.
Ushikubo, Hiroko, Sayaka Watanabe, Kazuho Abe, et al.. (2012). 3,3′,4′,5,5′-Pentahydroxyflavone is a potent inhibitor of amyloid β fibril formation. Neuroscience Letters. 513(1). 51–56. 37 indexed citations
17.
Chiba, Yoshihiko, Masahiko Murata, Hiroko Ushikubo, et al.. (2005). Effect of Cigarette Smoke Exposure In Vivo on Bronchial Smooth Muscle Contractility In Vitro in Rats. American Journal of Respiratory Cell and Molecular Biology. 33(6). 574–581. 45 indexed citations
18.
Ono, Masahiro, M Kawakami, & Hiroko Ushikubo. (1987). Stimulation of expression of the human endogenous retrovirus genome by female steroid hormones in human breast cancer cell line T47D. Journal of Virology. 61(6). 2059–2062. 176 indexed citations
19.
Ono, Masahiro, Teruo Yasunaga, Takashi Miyata, & Hiroko Ushikubo. (1986). Nucleotide sequence of human endogenous retrovirus genome related to the mouse mammary tumor virus genome. Journal of Virology. 60(2). 589–598. 288 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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