Hiroaki Kito

521 total citations
29 papers, 401 citations indexed

About

Hiroaki Kito is a scholar working on Molecular Biology, Sensory Systems and Immunology. According to data from OpenAlex, Hiroaki Kito has authored 29 papers receiving a total of 401 indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 8 papers in Sensory Systems and 6 papers in Immunology. Recurrent topics in Hiroaki Kito's work include Ion channel regulation and function (17 papers), Ion Channels and Receptors (8 papers) and Ion Transport and Channel Regulation (5 papers). Hiroaki Kito is often cited by papers focused on Ion channel regulation and function (17 papers), Ion Channels and Receptors (8 papers) and Ion Transport and Channel Regulation (5 papers). Hiroaki Kito collaborates with scholars based in Japan and Egypt. Hiroaki Kito's co-authors include Susumu Ohya, Junko Kajikuri, Hisao Yamamura, Katsuhiko Muraki, Kiyofumi Asai, Takayoshi Suzuki, Noriyuki Hatano, Yuji Imaizumi, Yuji Imaizumi and Masanori Fujii and has published in prestigious journals such as Biochemical and Biophysical Research Communications, International Journal of Molecular Sciences and Journal of Pharmacology and Experimental Therapeutics.

In The Last Decade

Hiroaki Kito

29 papers receiving 399 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hiroaki Kito Japan 13 273 94 60 51 51 29 401
Rebecca Drapp United States 6 237 0.9× 112 1.2× 27 0.5× 26 0.5× 73 1.4× 10 403
Chun-Yin Lo Hong Kong 8 174 0.6× 121 1.3× 22 0.4× 32 0.6× 36 0.7× 10 334
Michael J. Van Kanegan United States 8 315 1.2× 89 0.9× 32 0.5× 117 2.3× 138 2.7× 10 511
Hristina Ivanova Belgium 13 560 2.1× 54 0.6× 64 1.1× 101 2.0× 65 1.3× 18 747
Meenu Madan United States 12 305 1.1× 76 0.8× 24 0.4× 109 2.1× 63 1.2× 16 496
Dongming Hou China 6 221 0.8× 36 0.4× 76 1.3× 19 0.4× 45 0.9× 10 424
Esther Hernández‐SanMiguel Spain 12 390 1.4× 22 0.2× 50 0.8× 151 3.0× 54 1.1× 16 547
Xiahui Tan Australia 11 169 0.6× 73 0.8× 37 0.6× 42 0.8× 110 2.2× 13 361
Christoffer Löf Finland 14 281 1.0× 135 1.4× 38 0.6× 71 1.4× 26 0.5× 27 443
Irina O. Vassilieva Russia 13 276 1.0× 98 1.0× 71 1.2× 51 1.0× 151 3.0× 21 491

Countries citing papers authored by Hiroaki Kito

Since Specialization
Citations

This map shows the geographic impact of Hiroaki Kito's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroaki Kito with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroaki Kito more than expected).

Fields of papers citing papers by Hiroaki Kito

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroaki Kito. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroaki Kito. The network helps show where Hiroaki Kito may publish in the future.

Co-authorship network of co-authors of Hiroaki Kito

This figure shows the co-authorship network connecting the top 25 collaborators of Hiroaki Kito. A scholar is included among the top collaborators of Hiroaki Kito based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroaki Kito. Hiroaki Kito is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ohya, Susumu, et al.. (2022). Downregulation of IL-8 and IL-10 by the Activation of Ca2+-Activated K+ Channel KCa3.1 in THP-1-Derived M2 Macrophages. International Journal of Molecular Sciences. 23(15). 8603–8603. 12 indexed citations
2.
Kajikuri, Junko, et al.. (2021). KCa3.1 inhibition-induced activation of the JNK/c-Jun signaling pathway enhances IL-10 expression in peripherally-induced regulatory T cells. Journal of Pharmacological Sciences. 148(1). 1–5. 6 indexed citations
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Ohya, Susumu, et al.. (2021). KCa1.1 K+ Channel Inhibition Overcomes Resistance to Antiandrogens and Doxorubicin in a Human Prostate Cancer LNCaP Spheroid Model. International Journal of Molecular Sciences. 22(24). 13553–13553. 14 indexed citations
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Kito, Hiroaki, Ryo Tanaka, Junko Kajikuri, et al.. (2019). Possible Contribution of Inflammation-Associated Hypoxia to Increased K2P5.1 K+ Channel Expression in CD4+ T Cells of the Mouse Model for Inflammatory Bowel Disease. International Journal of Molecular Sciences. 21(1). 38–38. 7 indexed citations
7.
Ohya, Susumu, Hiroaki Kito, & Junko Kajikuri. (2019). Ca<sup>2+</sup>-activated K<sup>+</sup> channels as cancer therapeutic targets. Folia Pharmacologica Japonica. 154(3). 108–113. 1 indexed citations
8.
Kajikuri, Junko, et al.. (2018). Histone Deacetylases Enhance Ca2+-Activated K+ Channel KCa3.1 Expression in Murine Inflammatory CD4+ T Cells. International Journal of Molecular Sciences. 19(10). 2942–2942. 12 indexed citations
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Shimizu, Ayaka, et al.. (2016). Defective splicing of the background K+ channel K2P5.1 by the pre-mRNA splicing inhibitor, pladienolide B in lectin-activated mouse splenic CD4+ T cells. Journal of Pharmacological Sciences. 132(3). 205–209. 4 indexed citations
12.
Ohya, Susumu, Hiroaki Kito, Noriyuki Hatano, & Katsuhiko Muraki. (2016). Recent advances in therapeutic strategies that focus on the regulation of ion channel expression. Pharmacology & Therapeutics. 160. 11–43. 38 indexed citations
13.
Inoue, Takahiro, et al.. (2016). Transcriptional repression of HER2 by ANO1 Cl− channel inhibition in human breast cancer cells with resistance to trastuzumab. Biochemical and Biophysical Research Communications. 482(1). 188–194. 18 indexed citations
14.
Kito, Hiroaki, Hisao Yamamura, Yoshiaki Suzuki, et al.. (2015). Regulation of store-operated Ca2+ entry activity by cell cycle dependent up-regulation of Orai2 in brain capillary endothelial cells. Biochemical and Biophysical Research Communications. 459(3). 457–462. 25 indexed citations
15.
Sato, Aya, Hiroaki Kito, Masanori Fujii, et al.. (2015). Pathophysiological significance of the two-pore domain K+ channel K2P5.1 in splenic CD4+CD25− T cell subset from a chemically-induced murine inflammatory bowel disease model. Frontiers in Physiology. 6. 299–299. 11 indexed citations
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Kito, Hiroaki, Hisao Yamamura, Yoshiaki Suzuki, et al.. (2014). Membrane Hyperpolarization Induced by Endoplasmic Reticulum Stress Facilitates Ca2+ Influx to Regulate Cell Cycle Progression in Brain Capillary Endothelial Cells. Journal of Pharmacological Sciences. 125(2). 227–232. 11 indexed citations
18.
Ohya, Susumu, Hiroaki Kito, Yasuhiro Maeda, et al.. (2014). Upregulation of KCa3.1 K+channel in mesenteric lymph node CD4+T lymphocytes from a mouse model of dextran sodium sulfate-induced inflammatory bowel disease. American Journal of Physiology-Gastrointestinal and Liver Physiology. 306(10). G873–G885. 30 indexed citations
19.
Kito, Hiroaki, Daiju Yamazaki, Susumu Ohya, et al.. (2011). Up-regulation of Kir2.1 by ER stress facilitates cell death of brain capillary endothelial cells. Biochemical and Biophysical Research Communications. 411(2). 293–298. 17 indexed citations
20.
Choi, So Yung, et al.. (1992). Effect of cigarette smoking and alcohol drinking on risk of cancers.. 14(1). 35–41. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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