Hiroaki Hase

1.1k total citations
23 papers, 634 citations indexed

About

Hiroaki Hase is a scholar working on Molecular Biology, Cancer Research and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Hiroaki Hase has authored 23 papers receiving a total of 634 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 9 papers in Cancer Research and 4 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Hiroaki Hase's work include RNA modifications and cancer (11 papers), Cancer-related molecular mechanisms research (5 papers) and Cancer-related gene regulation (5 papers). Hiroaki Hase is often cited by papers focused on RNA modifications and cancer (11 papers), Cancer-related molecular mechanisms research (5 papers) and Cancer-related gene regulation (5 papers). Hiroaki Hase collaborates with scholars based in Japan. Hiroaki Hase's co-authors include Kazutake Tsujikawa, Kaori Kitae, Yuko Ueda, Kentaro Jingushi, Kazuo Harada, Kazumasa Hirata, Norio Nonomura, Kazutoshi Fujita, Motohide Uemura and Wataru Nakata and has published in prestigious journals such as Journal of Biological Chemistry, Scientific Reports and Biochemical and Biophysical Research Communications.

In The Last Decade

Hiroaki Hase

22 papers receiving 629 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hiroaki Hase Japan 12 573 318 52 49 33 23 634
Yasu Jiang China 13 410 0.7× 279 0.9× 46 0.9× 37 0.8× 14 0.4× 19 485
Mengqi Chang China 10 460 0.8× 210 0.7× 61 1.2× 37 0.8× 48 1.5× 17 575
Anpei Hu China 11 531 0.9× 399 1.3× 59 1.1× 38 0.8× 12 0.4× 13 616
Qingya Luo China 9 725 1.3× 427 1.3× 65 1.3× 33 0.7× 90 2.7× 15 779
Ruizhi Fan China 8 267 0.5× 198 0.6× 49 0.9× 24 0.5× 12 0.4× 17 363
Haohao Huang China 9 268 0.5× 164 0.5× 78 1.5× 36 0.7× 14 0.4× 11 350
Yaoyun Tang China 9 329 0.6× 178 0.6× 75 1.4× 44 0.9× 12 0.4× 13 426
Xinquan Wu China 10 313 0.5× 181 0.6× 52 1.0× 22 0.4× 25 0.8× 26 380

Countries citing papers authored by Hiroaki Hase

Since Specialization
Citations

This map shows the geographic impact of Hiroaki Hase's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroaki Hase with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroaki Hase more than expected).

Fields of papers citing papers by Hiroaki Hase

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroaki Hase. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroaki Hase. The network helps show where Hiroaki Hase may publish in the future.

Co-authorship network of co-authors of Hiroaki Hase

This figure shows the co-authorship network connecting the top 25 collaborators of Hiroaki Hase. A scholar is included among the top collaborators of Hiroaki Hase based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroaki Hase. Hiroaki Hase is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hase, Hiroaki, et al.. (2025). CGRPβ suppresses the pathogenesis of ulcerative colitis via the immunoproteasome. Scientific Reports. 15(1). 7224–7224.
2.
Jingushi, Kentaro, Kohei Taniguchi, Kazumasa Komura, et al.. (2025). Cancer‐Specific RNA Modifications in Tumour‐Derived Extracellular Vesicles Promote Tumour Growth. Journal of Extracellular Vesicles. 14(5). e70083–e70083. 1 indexed citations
3.
Nakai, M., Hiroaki Hase, Yutong Zhao, et al.. (2024). RNA-modifying enzyme Alkbh8 is involved in mouse embryonic development. iScience. 27(9). 110777–110777. 2 indexed citations
4.
Honda, Kohei, Hiroaki Hase, Katsuya Okawa, et al.. (2024). ALKBH8 contributes to neurological function through oxidative stress regulation. PNAS Nexus. 3(3). pgae115–pgae115. 9 indexed citations
5.
Jingushi, Kentaro, Atsunari Kawashima, Takuro Saito, et al.. (2024). Cutibacterium acnes‐derived extracellular vesicles promote tumor growth in renal cell carcinoma. Cancer Science. 115(8). 2578–2587. 13 indexed citations
6.
Hase, Hiroaki, et al.. (2023). ALKBH4 is a novel enzyme that promotes translation through modified uridine regulation. Journal of Biological Chemistry. 299(9). 105093–105093. 4 indexed citations
7.
8.
Hase, Hiroaki, Kentaro Matsumoto, Toshiyuki Nagata, et al.. (2020). Cancer type‑SLCO1B3 promotes epithelial‑mesenchymal transition resulting in the tumour progression of non‑small cell lung cancer. Oncology Reports. 45(1). 309–316. 8 indexed citations
9.
Jingushi, Kentaro, Toshiyuki Nagata, Masami Sato, et al.. (2019). MicroRNA-130b functions as an oncomiRNA in non-small cell lung cancer by targeting tissue inhibitor of metalloproteinase-2. Scientific Reports. 9(1). 6956–6956. 32 indexed citations
10.
Ueda, Masahiro, Tadashi Shimizu, Miyuki Mabuchi, et al.. (2018). Novel Metabolically Stable PCA-1/ALKBH3 Inhibitor Has Potent Antiproliferative Effects on DU145 Cells In Vivo. Anticancer Research. 38(1). 211–218. 16 indexed citations
11.
Kitae, Kaori, et al.. (2017). TP53 gene status is a critical determinant of phenotypes induced by ALKBH3 knockdown in non-small cell lung cancers. Biochemical and Biophysical Research Communications. 488(2). 285–290. 11 indexed citations
12.
Jingushi, Kentaro, Yuko Ueda, Kaori Kitae, et al.. (2017). High miR-122 expression promotes malignant phenotypes in ccRCC by targeting occludin. International Journal of Oncology. 51(1). 289–297. 30 indexed citations
13.
Ueda, Yuko, Kaori Kitae, Kentaro Jingushi, et al.. (2017). AlkB homolog 3-mediated tRNA demethylation promotes protein synthesis in cancer cells. Scientific Reports. 7(1). 42271–42271. 247 indexed citations
14.
Kawakami, Ryoji, Kazuhiro Nakajima, Kaori Kitae, et al.. (2016). ALKBH8 promotes bladder cancer growth and progression through regulating the expression of survivin. Biochemical and Biophysical Research Communications. 477(3). 413–418. 31 indexed citations
15.
16.
Jingushi, Kentaro, Yuko Ueda, Kaori Kitae, et al.. (2014). miR-629 Targets TRIM33 to Promote TGFβ/Smad Signaling and Metastatic Phenotypes in ccRCC. Molecular Cancer Research. 13(3). 565–574. 67 indexed citations
17.
Hase, Hiroaki, Kentaro Jingushi, Yuko Ueda, et al.. (2014). LOXL2 Status Correlates with Tumor Stage and Regulates Integrin Levels to Promote Tumor Progression in ccRCC. Molecular Cancer Research. 12(12). 1807–1817. 38 indexed citations
18.
Hase, Hiroaki, et al.. (2013). Further studies on the adsorption of plant phenols by synthetic polymers. Acta Societatis Botanicorum Poloniae. 58(4). 583–592. 1 indexed citations
19.
Koike, Kazuo, Yuko Ueda, Hiroaki Hase, et al.. (2012). Anti-Tumor Effect of AlkB Homolog 3 Knockdown in Hormone- Independent Prostate Cancer Cells. Current Cancer Drug Targets. 12(7). 847–856. 36 indexed citations
20.
Kodaira, Toshiyuki, Makoto Taniguchi, Hiroaki Hase, & Tomochika Matsuyama. (1982). The Formation of Anion Radicals in Glassy N-Propyl and N-Benzyldimethacryloylamines γ-Irradiated at 77 K. Bulletin of the Chemical Society of Japan. 55(7). 2277–2278. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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