Hideki Igata

640 total citations
15 papers, 511 citations indexed

About

Hideki Igata is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Oncology. According to data from OpenAlex, Hideki Igata has authored 15 papers receiving a total of 511 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Cardiology and Cardiovascular Medicine, 6 papers in Molecular Biology and 4 papers in Oncology. Recurrent topics in Hideki Igata's work include Cardiac Fibrosis and Remodeling (3 papers), Heart Failure Treatment and Management (3 papers) and Renin-Angiotensin System Studies (3 papers). Hideki Igata is often cited by papers focused on Cardiac Fibrosis and Remodeling (3 papers), Heart Failure Treatment and Management (3 papers) and Renin-Angiotensin System Studies (3 papers). Hideki Igata collaborates with scholars based in Japan. Hideki Igata's co-authors include Akira Matsumori, Keiji Kusumoto, Tetsuo Shioi, Mami Ojima, Hideaki Nagaya, Hiroki Sakamoto, Takanobu Kuroita, Ryosuke Nishio, Shigetake Sasayama and Atsushi Iwasaki and has published in prestigious journals such as Circulation, Journal of Pharmacology and Experimental Therapeutics and British Journal of Pharmacology.

In The Last Decade

Hideki Igata

14 papers receiving 490 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hideki Igata Japan 8 235 196 112 90 55 15 511
Hossain Monshizadegan United States 12 156 0.7× 283 1.4× 70 0.6× 80 0.9× 43 0.8× 17 580
Holger Heitsch Germany 13 436 1.9× 277 1.4× 106 0.9× 229 2.5× 30 0.5× 20 756
Tetsuhiro Kubota Japan 12 99 0.4× 198 1.0× 135 1.2× 105 1.2× 17 0.3× 37 572
John T. Liles United States 14 108 0.5× 206 1.1× 45 0.4× 57 0.6× 23 0.4× 25 560
Kiyoshi Oizumi Japan 16 183 0.8× 228 1.2× 141 1.3× 63 0.7× 10 0.2× 28 588
Hiroshi Akutsu Japan 12 141 0.6× 129 0.7× 160 1.4× 70 0.8× 15 0.3× 39 511
Catherine Cazaubon France 15 466 2.0× 389 2.0× 105 0.9× 219 2.4× 49 0.9× 35 869
Viviane Caceres United States 9 213 0.9× 285 1.5× 55 0.5× 26 0.3× 44 0.8× 18 521
Nicholas S. Aberle United States 19 153 0.7× 306 1.6× 149 1.3× 27 0.3× 16 0.3× 29 776
Sung Eun Yoo South Korea 11 91 0.4× 258 1.3× 142 1.3× 105 1.2× 11 0.2× 28 562

Countries citing papers authored by Hideki Igata

Since Specialization
Citations

This map shows the geographic impact of Hideki Igata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideki Igata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideki Igata more than expected).

Fields of papers citing papers by Hideki Igata

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hideki Igata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideki Igata. The network helps show where Hideki Igata may publish in the future.

Co-authorship network of co-authors of Hideki Igata

This figure shows the co-authorship network connecting the top 25 collaborators of Hideki Igata. A scholar is included among the top collaborators of Hideki Igata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hideki Igata. Hideki Igata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Kusumoto, Keiji, Hideki Igata, Mami Ojima, et al.. (2011). Antihypertensive, insulin-sensitising and renoprotective effects of a novel, potent and long-acting angiotensin II type 1 receptor blocker, azilsartan medoxomil, in rat and dog models. European Journal of Pharmacology. 669(1-3). 84–93. 45 indexed citations
2.
Ojima, Mami, Hideki Igata, Masayuki Tanaka, et al.. (2010). In Vitro Antagonistic Properties of a New Angiotensin Type 1 Receptor Blocker, Azilsartan, in Receptor Binding and Function Studies. Journal of Pharmacology and Experimental Therapeutics. 336(3). 801–808. 122 indexed citations
3.
Asano, Yasutomi, Shuji Kitamura, Kazuyoshi Aso, et al.. (2008). Discovery, synthesis and biological evaluation of isoquinolones as novel and highly selective JNK inhibitors (1). Bioorganic & Medicinal Chemistry. 16(8). 4715–4732. 24 indexed citations
4.
Asano, Yasutomi, Shuji Kitamura, Fumio Itoh, et al.. (2008). Discovery, synthesis and biological evaluation of isoquinolones as novel and highly selective JNK inhibitors (2). Bioorganic & Medicinal Chemistry. 16(8). 4699–4714. 75 indexed citations
5.
Kusumoto, Keiji, Hideki Igata, Shota Ikeda, et al.. (2002). In vitro and in vivo pharmacology of a structurally novel Na+‐H+ exchange inhibitor, T‐162559. British Journal of Pharmacology. 135(8). 1995–2003. 21 indexed citations
6.
K, Ono, Akira Matsumori, Yoichi Furukawa, et al.. (1999). Prevention of myocardial reperfusion injury in rats by an antibody against monocyte chemotactic and activating factor/monocyte chemoattractant protein-1.. PubMed. 79(2). 195–203. 82 indexed citations
7.
Matsumori, Akira, Hideki Igata, Koh Ono, et al.. (1999). High Doses of Digitalis Increase the Myocardial Production of Proinflammatory Cytokines and Worsen Myocardial Injury in Viral Myocarditis. Japanese Circulation Journal. 63(12). 934–940. 47 indexed citations
8.
Matsumori, Akira, Koh Ono, Ryosuke Nishio, et al.. (1997). Modulation of Cytokine Production and Protection Against Lethal Endotoxemia by the Cardiac Glycoside Ouabain. Circulation. 96(5). 1501–1506. 74 indexed citations
9.
Ito, Katsuhiko, et al.. (1994). Effect of TCV-116, a novel non-peptide angiotensin II receptor antagonist, on chronic congestive heart failure (II).. The Japanese Journal of Pharmacology. 64. 103–103. 2 indexed citations
10.
Igata, Hideki, Katsuhiko Ito, & Go Kito. (1993). Antihypertensive and renal hemodynamic effects of a novel non-peptide angiotensin II receptor antagonist, TCV-116 in conscious renal hypertensive dogs.. The Japanese Journal of Pharmacology. 61. 169–169.
11.
Ito, Katsuhiko, et al.. (1993). Effect of TCV-116, a novel non-peptide angiotensin II receptor antagonist, on chronic congestive heart failure (CHF).. The Japanese Journal of Pharmacology. 61. 169–169. 2 indexed citations
12.
Takagi, Keiko, et al.. (1992). Effects of a New Histamine H2-Receptor Antagonist, Z-300, on Gastric Secretion and Gastro-Duodenal Lesions in Rats: Comparison with Roxatidine.. The Japanese Journal of Pharmacology. 59(3). 275–289. 6 indexed citations
13.
Okabe, Susumu, et al.. (1992). Effects of a New Histamine H2-Receptor Antagonist, Z-300, on Gastric Secretion and Gastro-Duodenal Lesions in Rats: Comparison with Roxatidine. The Japanese Journal of Pharmacology. 59(3). 275–289. 1 indexed citations
14.
Yamakawa, T., et al.. (1991). Synthesis and Structure-Activity Relationships of N-Substituted 2-((2-Imidazolylsulfinyl)methyl)anilines as a New Class of Gastric H+/K+-ATPase Inhibitors.. Chemical and Pharmaceutical Bulletin. 39(7). 1746–1752. 6 indexed citations
15.
Okabe, Susumu, et al.. (1990). Effects of IT-066, a new histamine H2-receptor antagonist, on gastric acid secretion and experimental gastric ulcers in rats and dogs.. Folia Pharmacologica Japonica. 95(5). 247–256. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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