Henriette Brésil

855 total citations
21 papers, 737 citations indexed

About

Henriette Brésil is a scholar working on Molecular Biology, Cancer Research and Pharmacology. According to data from OpenAlex, Henriette Brésil has authored 21 papers receiving a total of 737 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 8 papers in Cancer Research and 2 papers in Pharmacology. Recurrent topics in Henriette Brésil's work include Carcinogens and Genotoxicity Assessment (8 papers), DNA Repair Mechanisms (7 papers) and DNA and Nucleic Acid Chemistry (4 papers). Henriette Brésil is often cited by papers focused on Carcinogens and Genotoxicity Assessment (8 papers), DNA Repair Mechanisms (7 papers) and DNA and Nucleic Acid Chemistry (4 papers). Henriette Brésil collaborates with scholars based in France, United States and Italy. Henriette Brésil's co-authors include Ruggero Montesano, Geoffrey P. Margison, Anthony E. Pegg, Helmut Bartsch, C. Malaveille, A. Barbin, Alain Croisy, J. Hall, P. Jacquignon and M Vuillaume and has published in prestigious journals such as Proceedings of the National Academy of Sciences, JNCI Journal of the National Cancer Institute and Biochemical and Biophysical Research Communications.

In The Last Decade

Henriette Brésil

20 papers receiving 668 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Henriette Brésil France 13 512 337 131 89 54 21 737
Alexei J. Likhachev Russia 14 372 0.7× 298 0.9× 82 0.6× 105 1.2× 34 0.6× 29 631
Regine Goth Germany 6 491 1.0× 316 0.9× 81 0.6× 59 0.7× 53 1.0× 8 684
Valda M. Craddock United Kingdom 17 497 1.0× 184 0.5× 93 0.7× 57 0.6× 63 1.2× 32 821
Sipra Banerjee United States 19 596 1.2× 291 0.9× 169 1.3× 75 0.8× 93 1.7× 55 948
A. Gentil France 17 768 1.5× 426 1.3× 144 1.1× 62 0.7× 80 1.5× 40 970
Miriam C. Poirier United States 10 352 0.7× 370 1.1× 93 0.7× 166 1.9× 65 1.2× 11 687
Miroslava Protić‐Sabljić United States 12 481 0.9× 183 0.5× 58 0.4× 48 0.5× 84 1.6× 17 620
M. Rojas France 14 462 0.9× 337 1.0× 124 0.9× 159 1.8× 28 0.5× 24 811
Marina Bonfanti Italy 14 331 0.6× 130 0.4× 99 0.8× 53 0.6× 33 0.6× 38 584
Daniel L. Stout United States 12 308 0.6× 132 0.4× 74 0.6× 33 0.4× 39 0.7× 21 455

Countries citing papers authored by Henriette Brésil

Since Specialization
Citations

This map shows the geographic impact of Henriette Brésil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Henriette Brésil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Henriette Brésil more than expected).

Fields of papers citing papers by Henriette Brésil

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Henriette Brésil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Henriette Brésil. The network helps show where Henriette Brésil may publish in the future.

Co-authorship network of co-authors of Henriette Brésil

This figure shows the co-authorship network connecting the top 25 collaborators of Henriette Brésil. A scholar is included among the top collaborators of Henriette Brésil based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Henriette Brésil. Henriette Brésil is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Jongmans, Wim, et al.. (1996). The role of Ataxia telangiectasia and the DNA-dependent protein kinase in the p53-mediated cellular response to ionising radiation.. PubMed. 13(6). 1133–8. 39 indexed citations
2.
Esteve, Asunción, et al.. (1995). The role of the Ataxia telangiectasia gene in the p53, WAF1/CIP1(p21)- and GADD45-mediated response to DNA damage produced by ionising radiation.. PubMed. 11(8). 1427–35. 71 indexed citations
3.
Hall, Janet, Henriette Brésil, Christopher P. Wild, et al.. (1993). Alkylation and oxidative‐DNA damage repair activity in blood leukocytes of smokers and non‐smokers. International Journal of Cancer. 54(5). 728–733. 33 indexed citations
4.
Hall, Jennifer, et al.. (1990). Modulation of O6-methylguanine-DNA methyltransferase in rat and hamster liver after treatment with dimethylnitrosamine.. PubMed. 50(17). 5426–30. 24 indexed citations
5.
Montesano, Ruggero, et al.. (1988). Detection in human cells of alkylated macromolecules attributable to exposure to nitrosamines.. PubMed. 75–82. 7 indexed citations
6.
Montesano, Ruggero, et al.. (1987). [Effect of molybdenum Mo on the alkylation of DNA in the liver of rats treated with 14C-diethylnitrosamine].. PubMed. 9(1). 21–4. 1 indexed citations
7.
Hall, Janet, Henriette Brésil, & Ruggero Montesano. (1985). O6-Alkylguanine DNA alkyltransferase activity in monkey, human and rat liver. Carcinogenesis. 6(2). 209–211. 41 indexed citations
8.
Becker, Richard A., et al.. (1985). DNA ethylation in target and non-target organs of hamsters and rats treated with diethylnitrosamine. Cancer Letters. 26(1). 17–24. 8 indexed citations
9.
Margison, Geoffrey P., Donald P. Cooper, Roberts A. Smith, et al.. (1984). Enhanced repair of O6-alkylguanine in mammalian tissues.. PubMed. 30 Spec No. 132–44. 2 indexed citations
10.
Likhachev, Alexei J., et al.. (1983). Carcinogenicity of single doses of N-nitroso-N-methylurea and N-nitroso-N-ethylurea in Syrian golden hamsters and the persistence of alkylated purines in the DNA of various tissues.. PubMed. 43(2). 829–33. 21 indexed citations
11.
Montesano, Ruggero, et al.. (1983). Modification of DNA Repair Processes Induced by Nitrosamines. PubMed. 24. 531–543. 2 indexed citations
12.
Montesano, Ruggero, Henriette Brésil, C. Drevon, & C. Piccoli. (1982). DNA repair in mammalian cells exposed to multiple doses of alkylating agents. Biochimie. 64(8-9). 591–594. 3 indexed citations
13.
Pegg, Anthony E., Marcel Roberfroid, C von Bahr, et al.. (1982). Removal of O6-methylguanine from DNA by human liver fractions.. Proceedings of the National Academy of Sciences. 79(17). 5162–5165. 83 indexed citations
14.
Montesano, Ruggero, et al.. (1980). Effect of chronic treatment of rats with dimethylnitrosamine on the removal of O6-methylguanine from DNA.. PubMed. 40(2). 452–8. 86 indexed citations
15.
Montesano, Ruggero, et al.. (1980). Modulation of removal of 06 methyl guanine from liver dna of rats treated chronically with di methyl nitrosamine. 21. 2. 1 indexed citations
16.
Montesano, Ruggero, Henriette Brésil, & Geoffrey P. Margison. (1979). Increased excision of O6-methylguanine from rat liver DNA after chronic administration of dimethylnitrosamine.. PubMed. 39(5). 1798–802. 87 indexed citations
17.
Bartsch, Helmut, C. Malaveille, A. Barbin, et al.. (1976). Mutagenicity and metabolism of vinyl chloride and related compounds.. Environmental Health Perspectives. 17. 193–198. 37 indexed citations
18.
Margison, Geoffrey P., et al.. (1976). Effect of chronic administration of dimethylnitrosamine on the excision of 06-methylguanine from rat liver DNA. Cancer Letters. 2(2). 79–85. 13 indexed citations
19.
Barbin, A., Henriette Brésil, Alain Croisy, et al.. (1975). Liver-microsome-mediated formation of alkylating agents from vinyl bromide and vinyl chloride. Biochemical and Biophysical Research Communications. 67(2). 596–603. 150 indexed citations
20.
Montesano, Ruggero, Helmut Bartsch, & Henriette Brésil. (1974). Nitrosation of d-N,N'-Bis(1-hydroxymethylpropyl)ethylenediamine, an Antitubercular Drug. JNCI Journal of the National Cancer Institute. 52(3). 907–910. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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