Helena Ågerstam

1.1k total citations
20 papers, 574 citations indexed

About

Helena Ågerstam is a scholar working on Hematology, Genetics and Immunology. According to data from OpenAlex, Helena Ågerstam has authored 20 papers receiving a total of 574 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Hematology, 8 papers in Genetics and 7 papers in Immunology. Recurrent topics in Helena Ågerstam's work include Chronic Myeloid Leukemia Treatments (13 papers), Acute Myeloid Leukemia Research (7 papers) and Eosinophilic Disorders and Syndromes (5 papers). Helena Ågerstam is often cited by papers focused on Chronic Myeloid Leukemia Treatments (13 papers), Acute Myeloid Leukemia Research (7 papers) and Eosinophilic Disorders and Syndromes (5 papers). Helena Ågerstam collaborates with scholars based in Sweden, Denmark and Finland. Helena Ågerstam's co-authors include Thoas Fioretos, Marianne Rissler, Johan Richter, Marcus Järås, Nils Hansen, Maria Askmyr, Carin Lassen, Henrik Lilljebjörn, Carl Sandén and Sofia von Palffy and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Blood.

In The Last Decade

Helena Ågerstam

18 papers receiving 569 citations

Peers

Helena Ågerstam
Koorosh Korfi United Kingdom
Hardik Modi United States
Danielle Cain United States
F-X Mahon France
Samuel B. Luty United States
Courtney Prince United States
Kim-Hien T. Dao United States
Gurveen Saberwal United States
Helena Ågerstam
Citations per year, relative to Helena Ågerstam Helena Ågerstam (= 1×) peers Nils Hansen

Countries citing papers authored by Helena Ågerstam

Since Specialization
Citations

This map shows the geographic impact of Helena Ågerstam's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Helena Ågerstam with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Helena Ågerstam more than expected).

Fields of papers citing papers by Helena Ågerstam

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Helena Ågerstam. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Helena Ågerstam. The network helps show where Helena Ågerstam may publish in the future.

Co-authorship network of co-authors of Helena Ågerstam

This figure shows the co-authorship network connecting the top 25 collaborators of Helena Ågerstam. A scholar is included among the top collaborators of Helena Ågerstam based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Helena Ågerstam. Helena Ågerstam is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lilljebjörn, Henrik, Rasmus Henningsson, Marianne Rissler, et al.. (2025). The AML cellular state space unveils NPM1 immune evasion subtypes with distinct clinical outcomes. Nature Communications. 16(1). 10592–10592.
2.
Sandén, Carl, Niklas Landberg, Shruti Daga, et al.. (2025). Aberrant expression of SLAMF6 constitutes a targetable immune escape mechanism in acute myeloid leukemia. Nature Cancer. 6(11). 1821–1838.
3.
Henningsson, Rasmus, Helena Ågerstam, Carl Sandén, et al.. (2024). Single-cell genomics details the maturation block in BCP-ALL and identifies therapeutic vulnerabilities in DUX4-r cases. Blood. 144(13). 1399–1411. 3 indexed citations
4.
Baliakas, Panagiotis, Bianca Tesi, Jörg Cammenga, et al.. (2024). How to manage patients with germline DDX41 variants: Recommendations from the Nordic working group on germline predisposition for myeloid neoplasms. HemaSphere. 8(8). e145–e145. 3 indexed citations
5.
Palffy, Sofia von, Carl Sandén, Anna M. Blom, et al.. (2022). The complement receptor C3AR constitutes a novel therapeutic target in NPM1-mutated AML. Blood Advances. 7(7). 1204–1218. 6 indexed citations
6.
Sandén, Carl, Henrik Lilljebjörn, Christina Orsmark‐Pietras, et al.. (2020). Clonal competition within complex evolutionary hierarchies shapes AML over time. Nature Communications. 11(1). 579–579. 21 indexed citations
7.
Landberg, Niklas, Sofia von Palffy, Maria Askmyr, et al.. (2017). CD36 defines primitive chronic myeloid leukemia cells less responsive to imatinib but vulnerable to antibody-based therapeutic targeting. Haematologica. 103(3). 447–455. 51 indexed citations
8.
Askmyr, Maria, Sofia von Palffy, Nils Hansen, et al.. (2017). Transgenic expression of human cytokines in immunodeficient mice does not facilitate myeloid expansion of BCR-ABL1 transduced human cord blood cells. PLoS ONE. 12(10). e0186035–e0186035. 4 indexed citations
9.
Landberg, Niklas, Arta Dreimane, Marianne Rissler, Rolf Billström, & Helena Ågerstam. (2017). Primary cells in BCR/FGFR1‐positive 8p11 myeloproliferative syndrome are sensitive to dovitinib, ponatinib, and dasatinib. European Journal Of Haematology. 99(5). 442–448. 15 indexed citations
10.
Ågerstam, Helena, Nils Hansen, Sofia von Palffy, et al.. (2016). IL1RAP antibodies block IL-1–induced expansion of candidate CML stem cells and mediate cell killing in xenograft models. Blood. 128(23). 2683–2693. 70 indexed citations
11.
Ågerstam, Helena, Christine Karlsson, Nils Hansen, et al.. (2015). Antibodies targeting human IL1RAP (IL1R3) show therapeutic effects in xenograft models of acute myeloid leukemia. Proceedings of the National Academy of Sciences. 112(34). 10786–10791. 86 indexed citations
12.
Askmyr, Maria, Helena Ågerstam, Henrik Lilljebjörn, et al.. (2014). Modeling chronic myeloid leukemia in immunodeficient mice reveals expansion of aberrant mast cells and accumulation of pre-B cells. Blood Cancer Journal. 4(12). e269–e269. 11 indexed citations
13.
Askmyr, Maria, Helena Ågerstam, Henrik Lilljebjörn, et al.. (2014). Modeling Chronic Myeloid Leukemia in Immunodeficient Mice Reveals an Inflammatory State with Expansion of Aberrant Mast Cells and Accumulation of Pre B Cells. Blood. 124(21). 3132–3132. 1 indexed citations
14.
Askmyr, Maria, Helena Ågerstam, Nils Hansen, et al.. (2013). Selective killing of candidate AML stem cells by antibody targeting of IL1RAP. Blood. 121(18). 3709–3713. 91 indexed citations
15.
Hansen, Nils, Kristian Reckzeh, Helena Ågerstam, et al.. (2013). Upregulation Of IL1RAP On Human Progenitor/Stem Cells Induces Features Of a Myeloproliferative Disorder In Mice. Blood. 122(21). 1650–1650. 1 indexed citations
16.
Hansen, Nils, Helena Ågerstam, Martin Wahlestedt, et al.. (2012). SOCS2 is dispensable for BCR/ABL1-induced chronic myeloid leukemia-like disease and for normal hematopoietic stem cell function. Leukemia. 27(1). 130–135. 10 indexed citations
17.
Järås, Marcus, Nils Hansen, Helena Ågerstam, et al.. (2010). Isolation and killing of candidate chronic myeloid leukemia stem cells by antibody targeting of IL-1 receptor accessory protein. Proceedings of the National Academy of Sciences. 107(37). 16280–16285. 133 indexed citations
18.
Ågerstam, Helena, Marcus Järås, Anna Andersson, et al.. (2010). Modeling the human 8p11-myeloproliferative syndrome in immunodeficient mice. Blood. 116(12). 2103–2111. 16 indexed citations
19.
Järås, Marcus, Helena Ågerstam, Carin Lassen, et al.. (2009). Expression of P190 and P210 BCR/ABL1 in normal human CD34+ cells induces similar gene expression profiles and results in a STAT5-dependent expansion of the erythroid lineage. Experimental Hematology. 37(3). 367–375. 11 indexed citations
20.
Ågerstam, Helena, Henrik Lilljebjörn, Carin Lassen, et al.. (2007). Fusion gene‐mediated truncation of RUNX1 as a potential mechanism underlying disease progression in the 8p11 myeloproliferative syndrome. Genes Chromosomes and Cancer. 46(7). 635–643. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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