Hechmi Louzir

894 total citations
17 papers, 538 citations indexed

About

Hechmi Louzir is a scholar working on Immunology, Rheumatology and Ophthalmology. According to data from OpenAlex, Hechmi Louzir has authored 17 papers receiving a total of 538 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Immunology, 7 papers in Rheumatology and 4 papers in Ophthalmology. Recurrent topics in Hechmi Louzir's work include Ocular Diseases and Behçet’s Syndrome (4 papers), Toxin Mechanisms and Immunotoxins (3 papers) and T-cell and B-cell Immunology (3 papers). Hechmi Louzir is often cited by papers focused on Ocular Diseases and Behçet’s Syndrome (4 papers), Toxin Mechanisms and Immunotoxins (3 papers) and T-cell and B-cell Immunology (3 papers). Hechmi Louzir collaborates with scholars based in Tunisia, France and Netherlands. Hechmi Louzir's co-authors include Mèlika Ben Ahmed, Maha Abdeladhim, N. Hmida, M. Miled, Nadine Cerf–Bensussan, Koussay Dellagi, H. Houman, Nicolette Moes, Sophie Buyse and A. Ben Osman and has published in prestigious journals such as The Journal of Immunology, International Journal of Molecular Sciences and The Lancet Infectious Diseases.

In The Last Decade

Hechmi Louzir

15 papers receiving 528 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hechmi Louzir Tunisia 9 252 140 122 109 85 17 538
Ekkehard May Germany 11 416 1.7× 76 0.5× 9 0.1× 21 0.2× 309 3.6× 18 722
Tomonari Shigemura Japan 14 255 1.0× 142 1.0× 19 0.2× 3 0.0× 39 0.5× 61 596
Alessandra G. Commodaro Brazil 15 274 1.1× 208 1.5× 170 1.4× 2 0.0× 72 0.8× 32 690
C E Yockey United States 6 714 2.8× 110 0.8× 5 0.0× 26 0.2× 11 0.1× 8 883
A Salerno Italy 16 264 1.0× 80 0.6× 3 0.0× 10 0.1× 80 0.9× 41 575
Roel Heijmans Netherlands 8 172 0.7× 73 0.5× 4 0.0× 16 0.1× 27 0.3× 9 322
Stéphane Leclair Germany 2 334 1.3× 47 0.3× 6 0.0× 11 0.1× 18 0.2× 3 493
Beatrice Passarini Italy 13 55 0.2× 100 0.7× 6 0.0× 4 0.0× 70 0.8× 35 356
U F Haustein Germany 12 59 0.2× 102 0.7× 5 0.0× 8 0.1× 68 0.8× 40 393
T.O. Gabrielsen Norway 7 105 0.4× 59 0.4× 4 0.0× 11 0.1× 19 0.2× 7 376

Countries citing papers authored by Hechmi Louzir

Since Specialization
Citations

This map shows the geographic impact of Hechmi Louzir's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hechmi Louzir with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hechmi Louzir more than expected).

Fields of papers citing papers by Hechmi Louzir

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hechmi Louzir. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hechmi Louzir. The network helps show where Hechmi Louzir may publish in the future.

Co-authorship network of co-authors of Hechmi Louzir

This figure shows the co-authorship network connecting the top 25 collaborators of Hechmi Louzir. A scholar is included among the top collaborators of Hechmi Louzir based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hechmi Louzir. Hechmi Louzir is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Hannachi, Naïla, et al.. (2025). Comparative Analysis of Serum BAFF and IL-17 Levels Pre- and Post-Antipsychotic Treatment for Acute Schizophrenia. International Journal of Molecular Sciences. 26(1). 385–385. 1 indexed citations
2.
3.
Louzir, Hechmi, et al.. (2025). A deep dive into shrimp allergy: clinical spectrum of shrimp allergy in a Tunisian pilot study. Frontiers in Allergy. 6. 1568475–1568475.
4.
Kana, Bavesh D, Patrick Arbuthnot, Yahya E. Choonara, et al.. (2023). Opportunities and challenges of leveraging COVID-19 vaccine innovation and technologies for developing sustainable vaccine manufacturing capabilities in Africa. The Lancet Infectious Diseases. 23(8). e288–e300. 14 indexed citations
5.
Marzouki, Soumaya, I. Zarâa, Maha Abdeladhim, et al.. (2020). Implicating bites from a leishmaniasis sand fly vector in the loss of tolerance in pemphigus. JCI Insight. 5(23). 3 indexed citations
6.
Marzouki, Soumaya, Maha Abdeladhim, Fabiano Oliveira, et al.. (2017). Phlebotomus papatasi Yellow-Related and Apyrase Salivary Proteins Are Candidates for Vaccination against Human Cutaneous Leishmaniasis. Journal of Investigative Dermatology. 138(3). 598–606. 12 indexed citations
7.
Ahmed, Mèlika Ben, I. Zarâa, N. Hmida, et al.. (2011). Functional defects of peripheral regulatory T lymphocytes in patients with progressive vitiligo. Pigment Cell & Melanoma Research. 25(1). 99–109. 79 indexed citations
8.
Marzouki, Soumaya, Mèlika Ben Ahmed, Maha Abdeladhim, et al.. (2011). Characterization of the Antibody Response to the Saliva of Phlebotomus papatasi in People Living in Endemic Areas of Cutaneous Leishmaniasis. American Journal of Tropical Medicine and Hygiene. 84(5). 653–661. 52 indexed citations
9.
Hmida, N., Mèlika Ben Ahmed, Bertrand Meresse, et al.. (2011). Impaired Control of Effector T Cells by Regulatory T Cells: A Clue to Loss of Oral Tolerance and Autoimmunity in Celiac Disease?. The American Journal of Gastroenterology. 107(4). 604–611. 88 indexed citations
10.
Ahmed, Mèlika Ben, M.H. Houman, Maha Abdeladhim, et al.. (2011). Resistance to Exogenous TGF-β Effects in Patients with Systemic Lupus Erythematosus. Journal of Clinical Immunology. 31(4). 574–583. 12 indexed citations
11.
Ahmed, Mèlika Ben, N. Hmida, Nicolette Moes, et al.. (2009). IL-15 Renders Conventional Lymphocytes Resistant to Suppressive Functions of Regulatory T Cells through Activation of the Phosphatidylinositol 3-Kinase Pathway. The Journal of Immunology. 182(11). 6763–6770. 110 indexed citations
12.
Mokni, M., et al.. (2008). Involvement of granzyme B and granulysin in the cytotoxic response in lichen planus. Journal of Cutaneous Pathology. 35(7). 630–634. 29 indexed citations
13.
Louzir, Hechmi, Mohamed Elloumi, H. Abid, et al.. (2007). [Clinical spectrum of cobalamin deficiency in Tunisia].. PubMed. 65(2). 135–42. 4 indexed citations
14.
Ahmed, Mèlika Ben, H. Houman, I. Ben Ghorbel, et al.. (2006). Cytokine Expression within Mucocutaneous Lesions of Behçe‘s Disease: Involvement of Proinflammatory and Th1 Cytokines. Kluwer Academic Publishers eBooks. 528. 343–346. 1 indexed citations
15.
Ahmed, Mèlika Ben, H. Houman, Sonia Abdelhak, et al.. (2006). MICA Transmembrane Region Polymorphism and HLA B51 in Tunisian Behçeťs Disease Patients. Kluwer Academic Publishers eBooks. 528. 225–228. 7 indexed citations
16.
Ahmed, Mèlika Ben, H. Houman, M. Miled, Koussay Dellagi, & Hechmi Louzir. (2004). Involvement of chemokines and Th1 cytokines in the pathogenesis of mucocutaneous lesions of Behçet's disease. Arthritis & Rheumatism. 50(7). 2291–2295. 125 indexed citations
17.
Houman, M.H., Sonia Abdelhak, Mèlika Ben Ahmed, et al.. (2002). Étude du polymorphisme du microsatellite de la région transmembranaire de MICA et du HLA B51 chez les patients tunisiens atteints d'une maladie de Behçet. La Revue de Médecine Interne. 23. 577s–577s. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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