Heberty Tarso Facundo

1.8k total citations
30 papers, 1.5k citations indexed

About

Heberty Tarso Facundo is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Heberty Tarso Facundo has authored 30 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 16 papers in Pathology and Forensic Medicine and 6 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Heberty Tarso Facundo's work include Cardiac Ischemia and Reperfusion (15 papers), Mitochondrial Function and Pathology (11 papers) and Glycosylation and Glycoproteins Research (8 papers). Heberty Tarso Facundo is often cited by papers focused on Cardiac Ischemia and Reperfusion (15 papers), Mitochondrial Function and Pathology (11 papers) and Glycosylation and Glycoproteins Research (8 papers). Heberty Tarso Facundo collaborates with scholars based in Brazil, United States and Portugal. Heberty Tarso Facundo's co-authors include Steven P. Jones, Gladys A. Ngoh, Alicia J. Kowaltowski, Lewis J. Watson, Robert E. Brainard, Wolfgang Dillmann, Ayesha Zafir, Tariq Hamid, Sumanth D. Prabhu and Bethany W. Long and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation Research and Biochemical Journal.

In The Last Decade

Heberty Tarso Facundo

30 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Heberty Tarso Facundo Brazil 19 1.0k 325 319 252 211 30 1.5k
An‐Chi Wei Taiwan 15 800 0.8× 141 0.4× 122 0.4× 94 0.4× 188 0.9× 33 1.0k
Qida Ju United States 12 988 1.0× 174 0.5× 90 0.3× 96 0.4× 170 0.8× 16 2.0k
Jianling Xie Australia 25 1.0k 1.0× 175 0.5× 102 0.3× 65 0.3× 46 0.2× 55 1.8k
Qinglin Yang United States 23 1.3k 1.3× 252 0.8× 105 0.3× 42 0.2× 478 2.3× 59 2.0k
Daisuke Kamei Japan 20 720 0.7× 198 0.6× 59 0.2× 115 0.5× 58 0.3× 33 1.7k
Qiao Zheng China 23 809 0.8× 121 0.4× 149 0.5× 66 0.3× 49 0.2× 62 1.6k
Jean‐Claude Thiers France 20 796 0.8× 488 1.5× 69 0.2× 54 0.2× 118 0.6× 45 1.7k
Hong‐Ye Zhao China 21 878 0.9× 112 0.3× 57 0.2× 112 0.4× 77 0.4× 82 1.5k

Countries citing papers authored by Heberty Tarso Facundo

Since Specialization
Citations

This map shows the geographic impact of Heberty Tarso Facundo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heberty Tarso Facundo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heberty Tarso Facundo more than expected).

Fields of papers citing papers by Heberty Tarso Facundo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Heberty Tarso Facundo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Heberty Tarso Facundo. The network helps show where Heberty Tarso Facundo may publish in the future.

Co-authorship network of co-authors of Heberty Tarso Facundo

This figure shows the co-authorship network connecting the top 25 collaborators of Heberty Tarso Facundo. A scholar is included among the top collaborators of Heberty Tarso Facundo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Heberty Tarso Facundo. Heberty Tarso Facundo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Facundo, Heberty Tarso, et al.. (2025). Mitochondrial electron transport chain disruption and oxidative stress in lipopolysaccharide-induced cardiac dysfunction in rats and mice. Free Radical Research. 59(5). 377–391. 1 indexed citations
2.
Facundo, Heberty Tarso, et al.. (2023). Competitive interaction between ATP and GTP regulates mitochondrial ATP-sensitive potassium channels. Chemico-Biological Interactions. 381. 110560–110560. 5 indexed citations
3.
Neves, Kelly Rose Tavares, et al.. (2022). Effects of vitamin D (VD3) supplementation on the brain mitochondrial function of male rats, in the 6-OHDA-induced model of Parkinson's disease. Neurochemistry International. 154. 105280–105280. 17 indexed citations
4.
Yoshinaga, Marcos Y., et al.. (2022). Calorie restriction changes lipidomic profiles and maintains mitochondrial function and redox balance during isoproterenol-induced cardiac hypertrophy. Journal of Physiology and Biochemistry. 78(1). 283–294. 5 indexed citations
5.
6.
Brainard, Robert E. & Heberty Tarso Facundo. (2021). Cardiac hypertrophy drives PGC-1α suppression associated with enhanced O-glycosylation. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1867(5). 166080–166080. 27 indexed citations
7.
Kowaltowski, Alicia J., et al.. (2020). Quercetin treatment increases H2O2 removal by restoration of endogenous antioxidant activity and blocks isoproterenol-induced cardiac hypertrophy. Naunyn-Schmiedeberg s Archives of Pharmacology. 394(2). 217–226. 21 indexed citations
8.
Lima, Marcos Antônio Pereira de, et al.. (2019). Association between Epstein-Barr Virus and Oral Carcinoma: A Systematic Review with Meta-Analysis. Critical Reviews™ in Oncogenesis. 24(4). 349–368. 21 indexed citations
9.
Kowaltowski, Alicia J., et al.. (2018). Calorie restriction attenuates hypertrophy-induced redox imbalance and mitochondrial ATP-sensitive K+ channel repression. The Journal of Nutritional Biochemistry. 62. 87–94. 11 indexed citations
10.
Lima, Marcos Antônio Pereira de, et al.. (2017). Association between Epstein-Barr virus (EBV) and cervical carcinoma: A meta-analysis. Gynecologic Oncology. 148(2). 317–328. 53 indexed citations
11.
Facundo, Heberty Tarso, et al.. (2017). Mitochondria and Cardiac Hypertrophy. Advances in experimental medicine and biology. 982. 203–226. 52 indexed citations
12.
Kowaltowski, Alicia J., et al.. (2016). Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects. Chemico-Biological Interactions. 261. 50–55. 22 indexed citations
13.
Kowaltowski, Alicia J., et al.. (2015). Mitochondrial ATP-sensitive Potassium Channel Opening Inhibits Isoproterenol-induced Cardiac Hypertrophy by Preventing Oxidative Damage. Journal of Cardiovascular Pharmacology. 65(4). 393–397. 15 indexed citations
14.
Huang, Jing, Jian Guo, Farideh Beigi, et al.. (2013). HASF is a stem cell paracrine factor that activates PKC epsilon mediated cytoprotection. Journal of Molecular and Cellular Cardiology. 66. 157–164. 26 indexed citations
15.
Ngoh, Gladys A., Lewis J. Watson, Heberty Tarso Facundo, & Steven P. Jones. (2010). Augmented O-GlcNAc signaling attenuates oxidative stress and calcium overload in cardiomyocytes. Amino Acids. 40(3). 895–911. 136 indexed citations
16.
Ngoh, Gladys A., Lewis J. Watson, Heberty Tarso Facundo, Wolfgang Dillmann, & Steven P. Jones. (2008). Non-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transition. Journal of Molecular and Cellular Cardiology. 45(2). 313–325. 110 indexed citations
17.
Facundo, Heberty Tarso, et al.. (2007). Mitochondrial ATP-sensitive K+ channels are redox-sensitive pathways that control reactive oxygen species production. Free Radical Biology and Medicine. 42(7). 1039–1048. 98 indexed citations
18.
Facundo, Heberty Tarso, et al.. (2005). Mitochondrial ATP-Sensitive K+ Channels Prevent Oxidative Stress, Permeability Transition and Cell Death. Journal of Bioenergetics and Biomembranes. 37(2). 75–82. 80 indexed citations
19.
Facundo, Heberty Tarso, et al.. (2005). Tissue protection mediated by mitochondrial K+ channels. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1762(2). 202–212. 84 indexed citations
20.
Facundo, Heberty Tarso, et al.. (2005). Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial K+ channel activity. Free Radical Biology and Medicine. 40(3). 469–479. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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