Hatsune Chiba
- Molecular Biology top 5%
- Genetics top 2%
- Pediatrics, Perinatology and Child Health top 1%
- Cancer Research top 5%
- Public Health, Environmental and Occupational Health top 5%
- Co-authors
- Masahiro KanedaHiroyuki SasakiTomohiro KonoRyutaro HirasawaYayoi ObataToru NakanoYasushi TotokiM. Azim Surani
- Topics
- Genetic Syndromes and Imprinting (13 papers)Epigenetics and DNA Methylation (12 papers)Prenatal Screening and Diagnostics (10 papers)
- Partner nations
- JapanUnited KingdomUnited States
In The Last Decade
Hatsune Chiba
17 papers receiving 2.6k citations
Hit Papers
Peers
Comparison fields: 5 of 91
- Molecular Biology 2.3k
- Genetics 910
- Pediatrics, Perinatology and Child Health 756
- Cancer Research 421
- Public Health, Environmental and Occupational Health 352
Countries citing papers authored by Hatsune Chiba
This map shows the geographic impact of Hatsune Chiba's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hatsune Chiba with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hatsune Chiba more than expected).
Fields of papers citing papers by Hatsune Chiba
This network shows the impact of papers produced by Hatsune Chiba. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hatsune Chiba. The network helps show where Hatsune Chiba may publish in the future.
Co-authorship network of co-authors of Hatsune Chiba
This figure shows the co-authorship network connecting the top 25 collaborators of Hatsune Chiba. A scholar is included among the top collaborators of Hatsune Chiba based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hatsune Chiba. Hatsune Chiba is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 18 | |
| 3 | 56 | |
| 4 | 11 | |
| 5 | 84 | |
| 6 | 45 | |
| 7 | 183 | |
| 8 | 65 | |
| 9 | 236 | |
| 10 | 56 | |
| 11 | 27 | |
| 12 | 91 | |
| 13 | 456 | |
| 14 | 79 | |
| 15 | 359 | |
| 16 | 34 | |
| 17 | Endogenous siRNAs from naturally formed dsRNAs regulate transcripts in mouse oocytesbreakdown → | 861 |
About Hatsune Chiba
Hatsune Chiba is a scholar working on Pediatrics, Perinatology and Child Health, Genetics and Molecular Biology, having authored 17 papers that have together received 2.7k indexed citations. Recurring topics across this work include Genetic Syndromes and Imprinting (13 papers), Epigenetics and DNA Methylation (12 papers) and Prenatal Screening and Diagnostics (10 papers). The work is most often cited by research in Pediatrics, Perinatology and Child Health (756 citations), Molecular Biology (2.3k citations) and Genetics (910 citations). Hatsune Chiba has collaborated with scholars based in Japan, United Kingdom and United States. Frequent co-authors include Masahiro Kaneda, Hiroyuki Sasaki, Tomohiro Kono, Ryutaro Hirasawa, Yayoi Obata, Toru Nakano, Yasushi Totoki, M. Azim Surani, Satomi Kuramochi‐Miyagawa and Toshiaki Watanabe. Their work appears in journals such as Nature, Nature Genetics and Genes & Development.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.