Hans‐Jörg Weig

642 total citations
17 papers, 485 citations indexed

About

Hans‐Jörg Weig is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Hans‐Jörg Weig has authored 17 papers receiving a total of 485 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Cardiology and Cardiovascular Medicine, 8 papers in Molecular Biology and 4 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Hans‐Jörg Weig's work include Cardiac electrophysiology and arrhythmias (6 papers), Viral Infections and Immunology Research (3 papers) and Ion channel regulation and function (3 papers). Hans‐Jörg Weig is often cited by papers focused on Cardiac electrophysiology and arrhythmias (6 papers), Viral Infections and Immunology Research (3 papers) and Ion channel regulation and function (3 papers). Hans‐Jörg Weig collaborates with scholars based in Germany, Greece and United Kingdom. Hans‐Jörg Weig's co-authors include Martin Ungerer, Melchior Seyfarth, Albert Schömig, Alessandra Moretti, Karl‐Ludwig Laugwitz, Meinrad Gawaz, Thomas Ott, Angelika Gillitzer, Lorenz Bott‐Flügel and Peter Seizer and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation and Journal of the American College of Cardiology.

In The Last Decade

Hans‐Jörg Weig

17 papers receiving 471 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hans‐Jörg Weig Germany 12 312 217 54 50 50 17 485
Alexandre Hego Belgium 12 118 0.4× 163 0.8× 61 1.1× 15 0.3× 78 1.6× 25 465
Kazuwa Nakao Japan 8 167 0.5× 251 1.2× 41 0.8× 38 0.8× 66 1.3× 9 470
Mariaelvy Bianchini Germany 9 135 0.4× 98 0.5× 48 0.9× 20 0.4× 26 0.5× 13 385
Javad Jabbari Denmark 13 492 1.6× 464 2.1× 30 0.6× 14 0.3× 17 0.3× 19 856
Claudia Noack Germany 12 203 0.7× 395 1.8× 123 2.3× 16 0.3× 20 0.4× 21 548
E. Fleck Germany 9 221 0.7× 177 0.8× 89 1.6× 10 0.2× 31 0.6× 19 475
J Schaper Germany 13 120 0.4× 115 0.5× 72 1.3× 19 0.4× 88 1.8× 29 435
Jihei Sara Lee South Korea 14 222 0.7× 57 0.3× 31 0.6× 36 0.7× 65 1.3× 47 492
Olga Azevedo Portugal 13 185 0.6× 114 0.5× 52 1.0× 13 0.3× 120 2.4× 64 494
Arian Pano United States 9 173 0.6× 262 1.2× 47 0.9× 15 0.3× 185 3.7× 16 574

Countries citing papers authored by Hans‐Jörg Weig

Since Specialization
Citations

This map shows the geographic impact of Hans‐Jörg Weig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hans‐Jörg Weig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hans‐Jörg Weig more than expected).

Fields of papers citing papers by Hans‐Jörg Weig

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hans‐Jörg Weig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hans‐Jörg Weig. The network helps show where Hans‐Jörg Weig may publish in the future.

Co-authorship network of co-authors of Hans‐Jörg Weig

This figure shows the co-authorship network connecting the top 25 collaborators of Hans‐Jörg Weig. A scholar is included among the top collaborators of Hans‐Jörg Weig based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hans‐Jörg Weig. Hans‐Jörg Weig is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
2.
Mueller, Karin Anne Lydia, et al.. (2012). Thrombolysis is an appropriate treatment in lead-associated infective endocarditis with giant vegetations located on the right atrial lead: Figure 1. BMJ Case Reports. 2012. bcr0920114855–bcr0920114855. 4 indexed citations
3.
Kerst, Gunter, Hans‐Jörg Weig, Slawomir Weretka, et al.. (2012). Contact force–controlled zero-fluoroscopy catheter ablation of right-sided and left atrial arrhythmia substrates. Heart Rhythm. 9(5). 709–714. 52 indexed citations
4.
Stellos, Konstantinos, Kateryna Sopova, Κimon Stamatelopoulos, et al.. (2011). Expression of platelet‐bound stromal cell‐derived factor‐1 in patients with non‐valvular atrial fibrillation and ischemic heart disease. Journal of Thrombosis and Haemostasis. 10(1). 49–55. 21 indexed citations
5.
Liu, J., Yan Zeng, Shihua Chen, et al.. (2011). Causes of moderate to large pericardial effusion requiring pericardiocentesis in 140 Han Chinese patients. Herz. 37(2). 183–187. 48 indexed citations
6.
Mueller, Iris, Meinrad Gawaz, Christine S. Zuern, et al.. (2010). Restrictive cardiomyopathy in inherited ATTR amyloidosis (TTR-Ser23Asn) in a patient of German-Italian extraction. BMJ Case Reports. 2010. bcr0620092032–bcr0620092032. 3 indexed citations
7.
Bigalke, Boris, Konstantinos Stellos, Hans‐Jörg Weig, et al.. (2009). Regulation of platelet glycoprotein VI (GPVI) surface expression and of soluble GPVI in patients with atrial fibrillation (AF) and acute coronary syndrome (ACS). Basic Research in Cardiology. 104(3). 352–357. 38 indexed citations
8.
Bott‐Flügel, Lorenz, et al.. (2008). Quantitative Analysis of Apoptotic Markers in Human End-Stage Heart Failure. European Journal of Heart Failure. 10(2). 129–132. 13 indexed citations
9.
Bott‐Flügel, Lorenz, et al.. (2005). Gene transfer of the pancaspase inhibitor P35 reduces myocardial infarct size and improves cardiac function. Journal of Molecular Medicine. 83(7). 526–534. 11 indexed citations
10.
Bölck, Birgit, Götz Münch, Martin Hellmich, et al.. (2004). Na+/Ca2+exchanger overexpression impairs frequency- and ouabain-dependent cell shortening in adult rat cardiomyocytes. American Journal of Physiology-Heart and Circulatory Physiology. 287(4). H1435–H1445. 25 indexed citations
11.
Moretti, Alessandra, Hans‐Jörg Weig, Thomas Ott, et al.. (2002). Essential myosin light chain as a target for caspase-3 in failing myocardium. Proceedings of the National Academy of Sciences. 99(18). 11860–11865. 86 indexed citations
12.
Laugwitz, Karl‐Ludwig, Alessandra Moretti, Hans‐Jörg Weig, et al.. (2001). Blocking Caspase-Activated Apoptosis Improves Contractility in Failing Myocardium. Human Gene Therapy. 12(17). 2051–2063. 72 indexed citations
13.
Laugwitz, Karl‐Ludwig, Hans‐Jörg Weig, Alessandra Moretti, et al.. (2001). Gene Transfer of Heterologous G Protein–Coupled Receptors to Cardiomyocytes. Circulation Research. 88(7). 688–695. 6 indexed citations
14.
Ungerer, Martin, et al.. (2000). Regional Pre- and Postsynaptic Sympathetic System in the Failing Human Heart — Regulation of βARK-1. European Journal of Heart Failure. 2(1). 23–31. 19 indexed citations
15.
Weig, Hans‐Jörg, Karl‐Ludwig Laugwitz, Alessandra Moretti, et al.. (2000). Enhanced Cardiac Contractility After Gene Transfer of V2 Vasopressin Receptors In Vivo by Ultrasound-Guided Injection or Transcoronary Delivery. Circulation. 101(13). 1578–1585. 55 indexed citations
16.
Laugwitz, Karl‐Ludwig, Martin Ungerer, Torsten Schöneberg, et al.. (1999). Adenoviral Gene Transfer of the Human V2 Vasopressin Receptor Improves Contractile Force of Rat Cardiomyocytes. Circulation. 99(7). 925–933. 23 indexed citations
17.
Ungerer, Martin, et al.. (1998). A recombinant, CMV promotor-driven G protein-coupled receptor in cardiomyocytes is not downregulated by agonists. Journal of the American College of Cardiology. 31. 107–107. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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