Hana Holubec

3.5k total citations
50 papers, 2.8k citations indexed

About

Hana Holubec is a scholar working on Surgery, Molecular Biology and Oncology. According to data from OpenAlex, Hana Holubec has authored 50 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Surgery, 18 papers in Molecular Biology and 13 papers in Oncology. Recurrent topics in Hana Holubec's work include Infant Nutrition and Health (11 papers), Helicobacter pylori-related gastroenterology studies (10 papers) and Drug Transport and Resistance Mechanisms (8 papers). Hana Holubec is often cited by papers focused on Infant Nutrition and Health (11 papers), Helicobacter pylori-related gastroenterology studies (10 papers) and Drug Transport and Resistance Mechanisms (8 papers). Hana Holubec collaborates with scholars based in United States, Italy and United Kingdom. Hana Holubec's co-authors include Claire M. Payne, Bohuslav Dvořák, Carol Bernstein, Harris Bernstein, Melissa D. Halpern, Katerina Dvorak, Catherine S. Williams, Jessica A. Dominguez, Jessica A. Clark and Robert S. McCuskey and has published in prestigious journals such as Gastroenterology, Cancer Research and Gut.

In The Last Decade

Hana Holubec

50 papers receiving 2.8k citations

Peers

Hana Holubec
Katerina Dvorak United States
Xiao‐Di Tan United States
Robert C. De Lisle United States
Birgitta Schmidt United States
Willem Renooij Netherlands
Hana Holubec
Citations per year, relative to Hana Holubec Hana Holubec (= 1×) peers Hasan Kulaksiz

Countries citing papers authored by Hana Holubec

Since Specialization
Citations

This map shows the geographic impact of Hana Holubec's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hana Holubec with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hana Holubec more than expected).

Fields of papers citing papers by Hana Holubec

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hana Holubec. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hana Holubec. The network helps show where Hana Holubec may publish in the future.

Co-authorship network of co-authors of Hana Holubec

This figure shows the co-authorship network connecting the top 25 collaborators of Hana Holubec. A scholar is included among the top collaborators of Hana Holubec based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hana Holubec. Hana Holubec is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bernstein, Carol, Hana Holubec, A. K. Bhattacharyya, et al.. (2011). Carcinogenicity of deoxycholate, a secondary bile acid. Archives of Toxicology. 85(8). 863–871. 276 indexed citations
2.
Dvorak, Katerina, Melissa Chavarria, Claire M. Payne, et al.. (2007). Ursodeoxycholic acid inhibits oxidative stress induced by bile acids and gastric acid in esophageal cell lines: Relevance to Barrett’s esophagus pathogenesis. Cancer Research. 67. 1405–1405. 1 indexed citations
3.
Chandramouli, Anupama, Jiaqi Shi, Yongmei Feng, et al.. (2007). Haploinsufficiency of the cdc2l gene contributes to skin cancer development in mice. Carcinogenesis. 28(9). 2028–2035. 16 indexed citations
4.
Bernstein, Harris, Hana Holubec, Carol Bernstein, et al.. (2007). Deoxycholate-Induced Colitis is Markedly Attenuated in Nos2 Knockout Mice in Association with Modulation of Gene Expression Profiles. Digestive Diseases and Sciences. 52(3). 628–642. 23 indexed citations
5.
Payne, C. M., Craig Weber, Katerina Dvorak, et al.. (2006). Deoxycholate induces mitochondrial oxidative stress and activates NF- B through multiple mechanisms in HCT-116 colon epithelial cells. Carcinogenesis. 28(1). 215–222. 136 indexed citations
6.
Bernstein, Harris, Hana Holubec, Carol Bernstein, et al.. (2006). Unique dietary-related mouse model of colitis. Inflammatory Bowel Diseases. 12(4). 278–293. 37 indexed citations
7.
Halpern, Melissa D., Hana Holubec, Tara A. Saunders, et al.. (2006). Bile Acids Induce Ileal Damage During Experimental Necrotizing Enterocolitis. Gastroenterology. 130(2). 359–372. 81 indexed citations
8.
Payne, C. M., Cara L Crowley-Weber, Katerina Dvorak, et al.. (2005). Mitochondrial perturbation attenuates bile acid-induced cytotoxicity. Cell Biology and Toxicology. 21(5-6). 215–231. 28 indexed citations
9.
Yerushalmi, Hagit, David G. Besselsen, Natalia A. Ignatenko, et al.. (2005). The role of NO synthases in arginine-dependent small intestinal and colonic carcinogenesis. Molecular Carcinogenesis. 45(2). 93–105. 33 indexed citations
10.
Bernstein, Harris, Claire M. Payne, Kathleen Kunke, et al.. (2004). A proteomic study of resistance to deoxycholate-induced apoptosis. Carcinogenesis. 25(5). 681–692. 25 indexed citations
11.
Dvořák, Bohuslav, Melissa D. Halpern, Hana Holubec, et al.. (2004). Rat Milk Decreases Necrotizing Enterocolitis in a Rat Model. Advances in experimental medicine and biology. 554. 471–473. 4 indexed citations
12.
Clark, Jessica A., Robert H. Lane, Nicole K. MacLennan, et al.. (2004). Epidermal growth factor reduces intestinal apoptosis in an experimental model of necrotizing enterocolitis. American Journal of Physiology-Gastrointestinal and Liver Physiology. 288(4). G755–G762. 115 indexed citations
13.
Halpern, Melissa D., Jessica A. Dominguez, Kateřina Dvořáková, et al.. (2003). Ileal Cytokine Dysregulation in Experimental Necrotizing Enterocolitis Is Reduced by Epidermal Growth Factor. Journal of Pediatric Gastroenterology and Nutrition. 36(1). 126–133. 9 indexed citations
14.
Payne, C. M., Cara L Crowley-Weber, Harris Bernstein, et al.. (2003). Caspase-6 mediated cleavage of guanylate cyclase alpha 1 during deoxycholate-induced apoptosis: Protective role of the nitric oxide signaling module. Cell Biology and Toxicology. 19(6). 373–392. 18 indexed citations
15.
Halpern, Melissa D., Jessica A. Dominguez, Kateřina Dvořáková, et al.. (2003). Ileal Cytokine Dysregulation in Experimental Necrotizing Enterocolitis Is Reduced by Epidermal Growth Factor. Journal of Pediatric Gastroenterology and Nutrition. 36(1). 126–133. 60 indexed citations
16.
Halpern, Melissa D., Hana Holubec, Jessica A. Dominguez, et al.. (2002). Up-Regulation of IL-18 and IL-12 in the Ileum of Neonatal Rats with Necrotizing Enterocolitis. Pediatric Research. 51(6). 733–739. 103 indexed citations
17.
Bernstein, Harris, Hana Holubec, James Warneke, et al.. (2002). Patchy field defects of apoptosis resistance and dedifferentiation in flat mucosa of colon resections from colon cancer patients. Annals of Surgical Oncology. 9(5). 505–517. 36 indexed citations
18.
Payne, Claire M., Margaret M. Briehl, Harris Bernstein, et al.. (1998). The stress-response proteins poly(ADP-ribose) polymerase and NF-κB protect against bile salt-induced apoptosis. Cell Death and Differentiation. 5(7). 623–636. 79 indexed citations
19.
Dvořák, Bohuslav, et al.. (1994). Epidermal growth factor and transforming growth factor‐α mRNA in rat small intestine: In situ hybridization study. FEBS Letters. 352(3). 291–295. 25 indexed citations
20.
Chvapil, Miloš, et al.. (1993). Collagen fibers as a temporary scaffold for replacement of ACL in goats. Journal of Biomedical Materials Research. 27(3). 313–325. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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