Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
A mutation in the surfactant protein B gene responsible for fatal neonatal respiratory disease in multiple kindreds.
1994431 citationsLawrence M. Nogee, Gérard Garnier et al.Journal of Clinical Investigationprofile →
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late)
cites ·
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This map shows the geographic impact of Hal Dietz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hal Dietz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hal Dietz more than expected).
This network shows the impact of papers produced by Hal Dietz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hal Dietz. The network helps show where Hal Dietz may publish in the future.
Co-authorship network of co-authors of Hal Dietz
This figure shows the co-authorship network connecting the top 25 collaborators of Hal Dietz.
A scholar is included among the top collaborators of Hal Dietz based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Hal Dietz. Hal Dietz is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Holmes, Kathryn W., Sheila Markwardt, Kim A. Eagle, et al.. (2022). Cardiovascular Outcomes in Aortopathy. Journal of the American College of Cardiology. 79(21). 2069–2081.17 indexed citations
Lindsay, Mark E. & Hal Dietz. (2014). The Genetic Basis of Aortic Aneurysm. Cold Spring Harbor Perspectives in Medicine. 4(9). a015909–a015909.48 indexed citations
7.
Holmes, Kathryn W., G. Michael Silberbach, Cheryl L. Maslen, et al.. (2013). Abstract 13178: Bicuspid Aortic Valve and Marfan Syndrome: Two Strikes?. Circulation. 128.1 indexed citations
Habashi, Jennifer, Tammy M. Holm, Jefferson J. Doyle, et al.. (2009). Abstract 4521: AT2 Signaling is a Positive Prognostic and Therapeutic Modifier of Marfan Syndrome: Lessons on the Inequality of ACEi and ARBs. Circulation. 120.4 indexed citations
Dietz, Hal, et al.. (1994). MDE heteroduplex analysis of PCR products spanning each exon of the fibrillin (FBN1) gene greatly increases the efficiency of mutation detection in the Marfan syndrome. The American Journal of Human Genetics. 55.
18.
Nogee, Lawrence M., Gérard Garnier, Hal Dietz, et al.. (1994). A mutation in the surfactant protein B gene responsible for fatal neonatal respiratory disease in multiple kindreds.. Journal of Clinical Investigation. 93(4). 1860–1863.431 indexed citations breakdown →
Dietz, Hal, Emily P. Zeitler, & Ronald Wolf. (1966). [The scintigraphic demonstration of the cerebrospinal space with I-131-labeled human serum albumin (RIHSA). Methods, indications, results].. PubMed. 105(4). 537–55.9 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.