Hai‐Ming Wu

3.5k total citations · 3 hit papers
28 papers, 2.8k citations indexed

About

Hai‐Ming Wu is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Immunology. According to data from OpenAlex, Hai‐Ming Wu has authored 28 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 17 papers in Cardiology and Cardiovascular Medicine and 7 papers in Immunology. Recurrent topics in Hai‐Ming Wu's work include Cardiac Fibrosis and Remodeling (11 papers), Signaling Pathways in Disease (5 papers) and Chemotherapy-induced cardiotoxicity and mitigation (5 papers). Hai‐Ming Wu is often cited by papers focused on Cardiac Fibrosis and Remodeling (11 papers), Signaling Pathways in Disease (5 papers) and Chemotherapy-induced cardiotoxicity and mitigation (5 papers). Hai‐Ming Wu collaborates with scholars based in China, Pakistan and Thailand. Hai‐Ming Wu's co-authors include Wei Deng, Qizhu Tang, Zhen‐Guo Ma, Xin Zhang, Yu‐Pei Yuan, Qingqing Wu, Mingxia Duan, Heng Zhou, Qizhu Tang and Can Hu and has published in prestigious journals such as Free Radical Biology and Medicine, Cell Death and Differentiation and Cell Death and Disease.

In The Last Decade

Hai‐Ming Wu

28 papers receiving 2.8k citations

Hit Papers

Ferritinophagy-mediated ferroptosis is involved in sepsis... 2019 2026 2021 2023 2020 2019 2019 100 200 300 400

Peers

Hai‐Ming Wu
Jian Wu China
Aijun Sun China
Danielle Kamato Australia
Amy Simon United States
Hai‐Ming Wu
Citations per year, relative to Hai‐Ming Wu Hai‐Ming Wu (= 1×) peers Qingqing Wu

Countries citing papers authored by Hai‐Ming Wu

Since Specialization
Citations

This map shows the geographic impact of Hai‐Ming Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hai‐Ming Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hai‐Ming Wu more than expected).

Fields of papers citing papers by Hai‐Ming Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hai‐Ming Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hai‐Ming Wu. The network helps show where Hai‐Ming Wu may publish in the future.

Co-authorship network of co-authors of Hai‐Ming Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Hai‐Ming Wu. A scholar is included among the top collaborators of Hai‐Ming Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hai‐Ming Wu. Hai‐Ming Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wu, Guanghao, Zheng Zhou, Hai‐Ming Wu, et al.. (2024). The role of FERMT2 in the tumor microenvironment and immunotherapy in pan-cancer using comprehensive single-cell and bulk sequencing. Heliyon. 10(9). e30505–e30505. 1 indexed citations
2.
Ma, Shuqing, Zhen Guo, Fang-Yuan Liu, et al.. (2021). 6-Gingerol protects against cardiac remodeling by inhibiting the p38 mitogen-activated protein kinase pathway. Acta Pharmacologica Sinica. 42(10). 1575–1586. 36 indexed citations
3.
Che, Yan, Zhaopeng Wang, Yuan Yuan, et al.. (2021). By restoring autophagic flux and improving mitochondrial function, corosolic acid protects against Dox-induced cardiotoxicity. Cell Biology and Toxicology. 38(3). 451–467. 35 indexed citations
4.
Zhang, Xin, Can Hu, Yu‐Pei Yuan, et al.. (2021). Endothelial ERG alleviates cardiac fibrosis via blocking endothelin-1-dependent paracrine mechanism. Cell Biology and Toxicology. 37(6). 873–890. 69 indexed citations
5.
Yang, Jingjing, Nan Zhang, Jian Ni, et al.. (2021). Cardiomyocyte-Specific RIP2 Overexpression Exacerbated Pathologic Remodeling and Contributed to Spontaneous Cardiac Hypertrophy. Frontiers in Cell and Developmental Biology. 9. 688238–688238. 4 indexed citations
6.
Liu, Libo, Sihui Huang, Man Xu, et al.. (2021). Isoquercitrin protects HUVECs against high glucose‑induced apoptosis through regulating p53 proteasomal degradation. International Journal of Molecular Medicine. 48(1). 22 indexed citations
7.
Li, Wenjing, Hai‐Han Liao, Hong Feng, et al.. (2020). Combination treatment of perifosine and valsartan showed more efficiency in protecting against pressure overload induced mouse heart failure. Journal of Pharmacological Sciences. 143(3). 199–208. 3 indexed citations
8.
Zhang, Xin, Can Hu, Ning Zhang, et al.. (2020). Matrine attenuates pathological cardiac fibrosis via RPS5/p38 in mice. Acta Pharmacologica Sinica. 42(4). 573–584. 98 indexed citations
9.
Hu, Can, Xin Zhang, Peng Song, et al.. (2020). Meteorin-like protein attenuates doxorubicin-induced cardiotoxicity via activating cAMP/PKA/SIRT1 pathway. Redox Biology. 37. 101747–101747. 181 indexed citations
10.
Ma, Zhen‐Guo, Chun‐Yan Kong, Hai‐Ming Wu, et al.. (2020). Toll-like receptor 5 deficiency diminishes doxorubicin-induced acute cardiotoxicity in mice. Theranostics. 10(24). 11013–11025. 42 indexed citations
11.
Zhang, Xin, Can Hu, Hai‐Ming Wu, Zhen‐Guo Ma, & Qi‐Zhu Tang. (2020). Fibronectin type III domain-containing 5 in cardiovascular and metabolic diseases: a promising biomarker and therapeutic target. Acta Pharmacologica Sinica. 42(9). 1390–1400. 23 indexed citations
12.
Yang, Dan, Hanqing Liu, Fang-Yuan Liu, et al.. (2020). Critical roles of macrophages in pressure overload-induced cardiac remodeling. Journal of Molecular Medicine. 99(1). 33–46. 13 indexed citations
13.
Wu, Qingqing, Yang Xiao, Chen Liu, et al.. (2019). The protective effect of high mobility group protein HMGA2 in pressure overload-induced cardiac remodeling. Journal of Molecular and Cellular Cardiology. 128. 160–178. 21 indexed citations
14.
Li, Ning, Heng Zhou, Hai‐Ming Wu, et al.. (2019). STING-IRF3 contributes to lipopolysaccharide-induced cardiac dysfunction, inflammation, apoptosis and pyroptosis by activating NLRP3. Redox Biology. 24. 101215–101215. 456 indexed citations breakdown →
15.
Zhang, Xin, Jinxiu Zhu, Zhen‐Guo Ma, et al.. (2019). Rosmarinic acid alleviates cardiomyocyte apoptosis via cardiac fibroblast in doxorubicin-induced cardiotoxicity. International Journal of Biological Sciences. 15(3). 556–567. 114 indexed citations
16.
Xu, Man, Chun‐Xia Wan, Sihui Huang, et al.. (2019). Oridonin protects against cardiac hypertrophy by promoting P21-related autophagy. Cell Death and Disease. 10(6). 403–403. 67 indexed citations
17.
Zhang, Xin, Can Hu, Chun‐Yan Kong, et al.. (2019). FNDC5 alleviates oxidative stress and cardiomyocyte apoptosis in doxorubicin-induced cardiotoxicity via activating AKT. Cell Death and Differentiation. 27(2). 540–555. 359 indexed citations breakdown →
18.
Hu, Can, Xin Zhang, Wenying Wei, et al.. (2019). Matrine attenuates oxidative stress and cardiomyocyte apoptosis in doxorubicin-induced cardiotoxicity via maintaining AMPKα/UCP2 pathway. Acta Pharmaceutica Sinica B. 9(4). 690–701. 217 indexed citations
19.
Liu, Fang-Yuan, Di Fan, Zheng Yang, et al.. (2019). TLR9 is essential for HMGB1-mediated post-myocardial infarction tissue repair through affecting apoptosis, cardiac healing, and angiogenesis. Cell Death and Disease. 10(7). 480–480. 60 indexed citations
20.
Ma, Zhen‐Guo, Yu‐Pei Yuan, Hai‐Ming Wu, Xin Zhang, & Qizhu Tang. (2018). Cardiac fibrosis: new insights into the pathogenesis. International Journal of Biological Sciences. 14(12). 1645–1657. 252 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026