Hailun Gu

597 total citations
19 papers, 489 citations indexed

About

Hailun Gu is a scholar working on Epidemiology, Rheumatology and Cancer Research. According to data from OpenAlex, Hailun Gu has authored 19 papers receiving a total of 489 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Epidemiology, 7 papers in Rheumatology and 5 papers in Cancer Research. Recurrent topics in Hailun Gu's work include Osteoarthritis Treatment and Mechanisms (7 papers), Adipokines, Inflammation, and Metabolic Diseases (6 papers) and Cancer-related molecular mechanisms research (4 papers). Hailun Gu is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (7 papers), Adipokines, Inflammation, and Metabolic Diseases (6 papers) and Cancer-related molecular mechanisms research (4 papers). Hailun Gu collaborates with scholars based in China. Hailun Gu's co-authors include Li Liu, Xingyao Li, Xiaolu Yu, Xiaolei Xu, Xin Dong, Lifeng Ding, Xudan Liu, Jun Yang, Yue Zhao and Yue Huang and has published in prestigious journals such as International Journal of Molecular Sciences, Nutrients and Toxicology and Applied Pharmacology.

In The Last Decade

Hailun Gu

19 papers receiving 486 citations

Peers

Hailun Gu
Comparison fields: 5 of 75
  • Rheumatology 229
  • Molecular Biology 203
  • Pharmacology 104
  • Cancer Research 75
  • Epidemiology 71
Xiaojuan Chen China
Tianwen Ma China
Ju‐Ryoung Kim South Korea
Daniel Ferguson United States
Zijian Yan China
Mengqi Zeng China
Liangchang Li China
Qi Ding China
Michele Joana Alves Brazil
Didem Turgut Coşan Türkiye
Xiaojuan Chen China View profile →
Citations per field, relative to Hailun Gu
Hailun Gu · 1×
Citations per year, relative to Hailun Gu
Hailun Gu · 1×

Countries citing papers authored by Hailun Gu

Since Specialization
Citations

This map shows the geographic impact of Hailun Gu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hailun Gu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hailun Gu more than expected).

Fields of papers citing papers by Hailun Gu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hailun Gu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hailun Gu. The network helps show where Hailun Gu may publish in the future.

Co-authorship network of co-authors of Hailun Gu

This figure shows the co-authorship network connecting the top 25 collaborators of Hailun Gu. A scholar is included among the top collaborators of Hailun Gu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hailun Gu. Hailun Gu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
# Title Journal Authors Indexed citations
1 n-3 polyunsaturated fatty acids alleviate the progression of obesity-related osteoarthritis and protect cartilage through inhibiting the HMGB1-RAGE/TLR4 signaling pathway International Immunopharmacology Tao Xiong, Xinjuan Wang et al. 7
2 Dietary Fatty Acid Regulation of the NLRP3 Inflammasome via the TLR4/NF‐κB Signaling Pathway Affects Chondrocyte Pyroptosis Oxidative Medicine and Cellular Longevity Xin Dong, Li Liu et al. 43
3 PCB153 suppressed autophagy via PI3K/Akt/mTOR and RICTOR/Akt/mTOR signaling by the upregulation of microRNA-155 in rat primary chondrocytes Toxicology and Applied Pharmacology Ziyu Liu, Yue Huang et al. 9
4 Leptin Induced TLR4 Expression via the JAK2‐STAT3 Pathway in Obesity‐Related Osteoarthritis Oxidative Medicine and Cellular Longevity Xin Dong, Hailun Gu et al. 27
5 Curcumin-Alleviated Osteoarthritic Progression in Rats Fed a High-Fat Diet by Inhibiting Apoptosis and Activating Autophagy via Modulation of MicroRNA-34a Journal of Inflammation Research Xin Dong, Li Liu et al. 31
6 <p>Resveratrol Exerts Anti-Osteoarthritic Effect by Inhibiting TLR4/NF-κB Signaling Pathway via the TLR4/Akt/FoxO1 Axis in IL-1β-Stimulated SW1353 Cells</p> Drug Design Development and Therapy Xiaolei Xu, Xudan Liu et al. 31
7 Protective effect of resveratrol on obesity-related osteoarthritis via alleviating JAK2/STAT3 signaling pathway is independent of SOCS3 Toxicology and Applied Pharmacology Hailun Gu, Yue Huang et al. 25
8 Resveratrol inhibits the development of obesity-related osteoarthritis via the TLR4 and PI3K/Akt signaling pathways Connective Tissue Research Xiaolei Xu, Xudan Liu et al. 44
9 Oral Administration of Resveratrol Alleviates Osteoarthritis Pathology in C57BL/6J Mice Model Induced by a High-Fat Diet Mediators of Inflammation Xingyao Li, Xiaolu Yu et al. 54
10 Resveratrol inhibits the IL-1β-induced expression of MMP-13 and IL-6 in human articular chondrocytes via TLR4/MyD88-dependent and -independent signaling cascades International Journal of Molecular Medicine Hailun Gu, Xiaolu Yu et al. 44
11 In utero exposure to di-(2-ethylhexyl) phthalate induces metabolic disorder and increases fat accumulation in visceral depots of C57BL/6J mice offspring Experimental and Therapeutic Medicine Hailun Gu, Wei Wang et al. 16
12 Oral Resveratrol Prevents Osteoarthritis Progression in C57BL/6J Mice Fed a High-Fat Diet Nutrients Hailun Gu, Keyu Li et al. 47
13 Glypican 4 may be involved in the adipose tissue redistribution in high-fat feeding C57BL/6J mice with peroxisome proliferators-activated receptor γ agonist rosiglitazone treatment Experimental and Therapeutic Medicine Li Liu, Hailun Gu et al. 10
14 Protective Effect of Resveratrol against IL-1β-Induced Inflammatory Response on Human Osteoarthritic Chondrocytes Partly via the TLR4/MyD88/NF-κB Signaling Pathway: An “in Vitro Study” International Journal of Molecular Sciences Li Liu, Hailun Gu et al. 78
15 Inhibition of SOCS-3 in adipocytes of rats with diet-induced obesity increases leptin-mediated fatty acid oxidation Endocrine Hailun Gu, Li Liu et al. 12
16 Adipogenic differentiation is not influenced by lentivirus-mediated shRNA targeting the SOCS3 gene in adipose-derived stromal cells Molecular Biology Reports Li Liu, Hailun Gu et al. 6
17 [Effect of rat recombinant leptin on expression of SOCS-3 in mature adipocytes]. PubMed Li Liu, Hailun Gu et al. 1
18 [Study on SOCS-3, PPARgamma and ACO mRNA expression of adipose tissues in obese rats with leptin resistance]. PubMed Li Liu, Hailun Gu et al. 2
19 [Adenovirus-mediated human bone morphogenetic protein 2 gene transferred to rabbit intervertebral disc cells in vitro]. PubMed Hailun Gu, Gang Lv et al. 2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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