H.A. Molina

782 total citations
21 papers, 623 citations indexed

About

H.A. Molina is a scholar working on Epidemiology, Cardiology and Cardiovascular Medicine and Rheumatology. According to data from OpenAlex, H.A. Molina has authored 21 papers receiving a total of 623 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Epidemiology, 11 papers in Cardiology and Cardiovascular Medicine and 6 papers in Rheumatology. Recurrent topics in H.A. Molina's work include Trypanosoma species research and implications (10 papers), Eosinophilic Disorders and Syndromes (5 papers) and Synthesis and Biological Evaluation (5 papers). H.A. Molina is often cited by papers focused on Trypanosoma species research and implications (10 papers), Eosinophilic Disorders and Syndromes (5 papers) and Synthesis and Biological Evaluation (5 papers). H.A. Molina collaborates with scholars based in Argentina, United States and Germany. H.A. Molina's co-authors include F Kierszenbaum, José Milei, Susana Llesuy, Alberto Boveris, Rubén Storino, Kimm J. Hamann, J Monges, A.L. Taratuto, M. T. Rimoldi and Marcos Tatagiba and has published in prestigious journals such as Cancer, American Heart Journal and Acta Neuropathologica.

In The Last Decade

H.A. Molina

21 papers receiving 605 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
H.A. Molina Argentina 15 228 148 107 105 82 21 623
Katsuro Yagawa Japan 17 153 0.7× 45 0.3× 189 1.8× 182 1.7× 48 0.6× 56 752
Jerome F. Strauss United States 14 153 0.7× 58 0.4× 239 2.2× 86 0.8× 36 0.4× 16 909
Monique Agrapart France 15 68 0.3× 155 1.0× 368 3.4× 72 0.7× 110 1.3× 26 1.0k
Naomi Watanabe Japan 17 92 0.4× 47 0.3× 319 3.0× 96 0.9× 76 0.9× 63 736
Tamyra Downs United States 14 204 0.9× 64 0.4× 179 1.7× 95 0.9× 57 0.7× 19 830
V. Bonnevie‐Nielsen Denmark 17 94 0.4× 124 0.8× 309 2.9× 46 0.4× 87 1.1× 39 1.1k
Mei Xue China 15 131 0.6× 70 0.5× 226 2.1× 35 0.3× 51 0.6× 24 651
Pravin Patel United States 20 294 1.3× 36 0.2× 154 1.4× 32 0.3× 298 3.6× 40 886
T Szabó Hungary 11 159 0.7× 39 0.3× 159 1.5× 52 0.5× 125 1.5× 26 895
Wolfgang Northemann Germany 18 101 0.4× 36 0.2× 384 3.6× 40 0.4× 49 0.6× 39 936

Countries citing papers authored by H.A. Molina

Since Specialization
Citations

This map shows the geographic impact of H.A. Molina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H.A. Molina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H.A. Molina more than expected).

Fields of papers citing papers by H.A. Molina

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H.A. Molina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H.A. Molina. The network helps show where H.A. Molina may publish in the future.

Co-authorship network of co-authors of H.A. Molina

This figure shows the co-authorship network connecting the top 25 collaborators of H.A. Molina. A scholar is included among the top collaborators of H.A. Molina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H.A. Molina. H.A. Molina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rades, Dirk, Steven E. Schild, Marcos Tatagiba, H.A. Molina, & Winfried Alberti. (2004). Therapy of meningeal melanocytomas. Cancer. 100(11). 2442–2447. 61 indexed citations
2.
Stalldecker, Graciela, et al.. (1994). [Hypopituitarism caused by colonic carcinoma metastasis associated with hypophysial aspergillosis].. PubMed. 54(3). 248–52. 13 indexed citations
3.
Garretto, Nélida, et al.. (1992). Schwannomatosis: report of a new case. Arquivos de Neuro-Psiquiatria. 50(4). 539–542. 1 indexed citations
4.
Molina, H.A. & F Kierszenbaum. (1989). Eosinophil Activation in Acute and Chronic Chagasic Myocardial Lesions and Deposition of Toxic Eosinophil Granule Proteins on Heart Myofibers. Journal of Parasitology. 75(1). 129–129. 16 indexed citations
5.
Molina, H.A. & F Kierszenbaum. (1989). Interaction of human eosinophils or neutrophils with Trypanosoma cruzi in vitro causes bystander cardiac cell damage.. PubMed. 66(2). 289–95. 32 indexed citations
6.
Ameriso, Sebastián F., et al.. (1989). Type 2 neurofibromatosis with Lisch nodules.. PubMed. 2(5-6). 315–21. 11 indexed citations
7.
Molina, H.A., José Milei, M. T. Rimoldi, S. M. González Cappa, & Rubén Storino. (1988). Histopathology of the heart conducting system in experimental Chagas disease in mice. Transactions of the Royal Society of Tropical Medicine and Hygiene. 82(2). 241–246. 9 indexed citations
8.
Molina, H.A. & F Kierszenbaum. (1988). Kinetics of Development of Inflammatory Lesions in Myocardial and Skeletal Muscle in Experimental Trypanosoma cruzi Infection. Journal of Parasitology. 74(3). 370–370. 16 indexed citations
9.
Molina, H.A., et al.. (1988). Toxic Effects Produced or Mediated by Human Eosinophil Granule Components on Trypanosoma cruzi. American Journal of Tropical Medicine and Hygiene. 38(2). 327–334. 68 indexed citations
10.
Molina, H.A. & F Kierszenbaum. (1988). Immunohistochemical detection of deposits of eosinophil-derived neurotoxin and eosinophil peroxidase in the myocardium of patients with Chagas' disease.. PubMed. 64(4). 725–31. 20 indexed citations
11.
Milei, José, A. Vázquez, Alberto Boveris, et al.. (1988). The Role of Prenylamine in the Prevention of Adriamycin-Induced Cardiotoxicity. A Review of Experimental and Clinical Findings. Journal of International Medical Research. 16(1). 19–30. 15 indexed citations
12.
Molina, H.A., Rita L. Cardoni, & M. T. Rimoldi. (1987). The neuromuscular pathology of experimental Chagas' disease. Journal of the Neurological Sciences. 81(2-3). 287–300. 26 indexed citations
13.
Molina, H.A. & F Kierszenbaum. (1987). A study of human myocardial tissue in chagas' disease: distribution and frequency of inflammatory cell types. International Journal for Parasitology. 17(7). 1297–1305. 27 indexed citations
14.
Sanz, Olga P., et al.. (1987). Peripheral Nervous System Damage in Experimental Chronic Chagas' Disease. American Journal of Tropical Medicine and Hygiene. 36(1). 41–45. 18 indexed citations
15.
Milei, José, et al.. (1986). Amelioration of adriamycin-induced cardiotoxicity in rabbits by prenylamine and vitamins A and E. American Heart Journal. 111(1). 95–102. 65 indexed citations
16.
Taratuto, A.L., et al.. (1985). Primary rhabdomyosarcoma of brain and cerebellum. Acta Neuropathologica. 66(2). 98–104. 16 indexed citations
17.
Llesuy, Susana, et al.. (1985). Comparison of Lipid Peroxidation and Myocardial Damage Induced by Adriamycin and 4′-Epiadriamycin in Mice. Tumori Journal. 71(3). 241–249. 152 indexed citations
18.
Milei, José, et al.. (1985). Prenylamine inhibition of adriamycin-induced myocardiopathy.. PubMed. 40(4). 383–96. 4 indexed citations
19.
Molina, H.A., José Milei, & Rubén Storino. (1984). Chronic Chagas’ Myocardiopathy. Cardiology. 71(6). 297–306. 16 indexed citations
20.
Taratuto, A.L., H.A. Molina, Graciela Zúccaro, & J Monges. (1983). 27 PRIMARY RHABDOMYOSARCOMA OF BRAIN AND CEREBELLUM-Report of four cases in infants. An immunohistochemical and electron microscopic study. Journal of Neuropathology & Experimental Neurology. 42(3). 314–314. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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