This map shows the geographic impact of H. Sobol's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. Sobol with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. Sobol more than expected).
This network shows the impact of papers produced by H. Sobol. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. Sobol. The network helps show where H. Sobol may publish in the future.
Co-authorship network of co-authors of H. Sobol
This figure shows the co-authorship network connecting the top 25 collaborators of H. Sobol.
A scholar is included among the top collaborators of H. Sobol based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with H. Sobol. H. Sobol is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Tournier, Isabelle, Brigitte Bressac–de Paillerets, H. Sobol, et al.. (2004). Significant contribution of germline BRCA2 rearrangements in male breast cancer families. HAL (Le Centre pour la Communication Scientifique Directe).7 indexed citations
3.
Eisinger, François, et al.. (2001). PS2 expression in BRCA1-associated breast cancers.. PubMed. 21(4B). 2877–81.2 indexed citations
4.
Eisinger, François, Alain Brémond, J Dauplat, et al.. (1999). [INSERM-FNCLCC collective expertise. Recommendations for medical management of women with genetic risk of developing breast and/or ovarian cancer].. PubMed. 42(1). 51–64.5 indexed citations
Sobol, H., François Eisinger, R Sauvan, et al.. (1998). [Impact of recent oncogenetic progress on the management of high risk breast cancer patients: the example of BRCA1 and BRCA2 genes].. PubMed. 59(6). 459–64.1 indexed citations
7.
Eisinger, François, J. Jacquemier, Colette Charpin, et al.. (1998). Mutations at BRCA1: the medullary breast carcinoma revisited.. PubMed. 58(8). 1588–92.108 indexed citations
8.
Eisinger, François, Alain Brémond, J Dauplat, et al.. (1998). [INSERM-FNCLCC collective expert's report. Recommendations for management of women having a genetic risk of developing breast and/or ovarian cancer. National Federation of Centers of the Fight Against Cancer].. PubMed. 59(6). 470–84.2 indexed citations
Sobol, H., Brigitte Bressac–de Paillerets, J.P. Peyrat, et al.. (1996). Truncation at conserved terminal regions of BRCA1 protein is associated with highly proliferating hereditary breast cancers.. PubMed. 56(14). 3216–9.49 indexed citations
11.
Vennin, Philippe, S. Giard, Claire Julian‐Reynier, et al.. (1996). [Attitudes towards screening and prevention of breast and ovarian cancers with hereditary predisposition. Survey by female gynecologists in the north of France].. PubMed. 83(9). 697–702.4 indexed citations
Chabre, Olivier, F Labat-Moleur, François Berthod, et al.. (1992). [Cutaneous lesion associated with multiple endocrine neoplasms type 2A (Sipple's syndrome). An early clinical marker].. PubMed. 21(7). 299–303.11 indexed citations
14.
Sobol, H., et al.. (1989). Hereditary medullary thyroid carcinoma: genetic annalysis of three related syndromes. Groupe d'Etude des Tumeurs a Calcitonine.. PubMed. 37(3-4). 109–11.2 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.