Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
Lung Cancer—Epidemiology, Pathogenesis, Treatment and Molecular Aspect (Review of Literature)
202518 citationsBeata Smolarz, H Łukasiewicz et al.International Journal of Molecular Sciencesprofile →
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late)
cites ·
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This map shows the geographic impact of H Łukasiewicz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H Łukasiewicz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H Łukasiewicz more than expected).
This network shows the impact of papers produced by H Łukasiewicz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H Łukasiewicz. The network helps show where H Łukasiewicz may publish in the future.
Co-authorship network of co-authors of H Łukasiewicz
This figure shows the co-authorship network connecting the top 25 collaborators of H Łukasiewicz.
A scholar is included among the top collaborators of H Łukasiewicz based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with H Łukasiewicz. H Łukasiewicz is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Łukasiewicz, H, et al.. (1995). [Molecular markers of hemostasis activation in children with nephrotic syndrome].. PubMed. 70(12). 1029–35.3 indexed citations
Niewiarowski, Stefan, et al.. (1975). Inihibition of human platelet aggregation by dipyridamole and two related compounds and its modification by acid glycoproteins of human plasma.. PubMed. 86(1). 64–76.23 indexed citations
Łukasiewicz, H, et al.. (1973). Immunological phenomen in the porcine liver perfused with human blood.. PubMed. 14(2). 83–8.
17.
Łukasiewicz, H, et al.. (1972). Biosynthetic activities in normal and pathological platelet populations.. PubMed. 53. 105–12.2 indexed citations
18.
Rowińskí, W, et al.. (1972). Rosette-inhibiting activity of uraemic sera and the influence of dialysis.. PubMed. 9. 507–13.
19.
Jp, Caen, et al.. (1970). Constitutional and acquired abnormalities of platelet aggregation.. PubMed. 3(4). 83–99.5 indexed citations
20.
Łukasiewicz, H, et al.. (1966). [Studies on the pathogenesis of the acute pancreatic necrosis. II. Changes in the organism due to the presence of pancreatic juice in circulating blood].. PubMed. 38(7). 631–5 contd.1 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.