H. J. Weis

725 total citations
21 papers, 585 citations indexed

About

H. J. Weis is a scholar working on Surgery, Oncology and Molecular Biology. According to data from OpenAlex, H. J. Weis has authored 21 papers receiving a total of 585 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Surgery, 6 papers in Oncology and 5 papers in Molecular Biology. Recurrent topics in H. J. Weis's work include Cholesterol and Lipid Metabolism (7 papers), Drug Transport and Resistance Mechanisms (6 papers) and Metabolism and Genetic Disorders (2 papers). H. J. Weis is often cited by papers focused on Cholesterol and Lipid Metabolism (7 papers), Drug Transport and Resistance Mechanisms (6 papers) and Metabolism and Genetic Disorders (2 papers). H. J. Weis collaborates with scholars based in Germany and United States. H. J. Weis's co-authors include John M. Dietschy, K. H. Holtermüller, B. Kommerell, P. Czygan, A. Stiehl, Flavio Nervi, Lee A. Bricker, Marvin D. Siperstein, Jan van der Linden and Irene L. Noronha and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Gastroenterology.

In The Last Decade

H. J. Weis

18 papers receiving 521 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
H. J. Weis Germany	10	314	228	132	118	95	21	585
N. Domingo France	18	388 1.2×	384 1.7×	305 2.3×	135 1.1×	96 1.0×	60	841
Rick Havinga Netherlands	10	238 0.8×	275 1.2×	161 1.2×	115 1.0×	79 0.8×	12	539
Reno Z. Vlahcevic United States	9	358 1.1×	429 1.9×	122 0.9×	189 1.6×	77 0.8×	9	671
Jan‐Erik Åkerlund Sweden	11	304 1.0×	176 0.8×	120 0.9×	76 0.6×	57 0.6×	12	432
Russell F. Hanson United States	14	237 0.8×	316 1.4×	189 1.4×	104 0.9×	45 0.5×	29	642
D Trülzsch United States	10	195 0.6×	344 1.5×	195 1.5×	231 2.0×	30 0.3×	16	709
E Kok United States	12	346 1.1×	285 1.3×	215 1.6×	68 0.6×	75 0.8×	20	568
Vlahcevic Zr United States	10	260 0.8×	296 1.3×	83 0.6×	78 0.7×	50 0.5×	13	437
H.-U. Schulz Germany	11	200 0.6×	99 0.4×	160 1.2×	53 0.4×	54 0.6×	18	489
Mora Kallner Sweden	11	179 0.6×	221 1.0×	45 0.3×	152 1.3×	52 0.5×	15	373

Countries citing papers authored by H. J. Weis

Since Specialization

Citations

This map shows the geographic impact of H. J. Weis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. J. Weis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. J. Weis more than expected).

Fields of papers citing papers by H. J. Weis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H. J. Weis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. J. Weis. The network helps show where H. J. Weis may publish in the future.

Co-authorship network of co-authors of H. J. Weis

This figure shows the co-authorship network connecting the top 25 collaborators of H. J. Weis. A scholar is included among the top collaborators of H. J. Weis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. J. Weis. H. J. Weis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Holtermüller, K. H., et al.. (2008). Behandlung von Ulcera peptica jejuni mit Cimetidin oder einem Antacidum? - Ergebnisse einer Langzeitstudie^*. DMW - Deutsche Medizinische Wochenschrift. 107(47). 1800–1804.

Noronha, Irene L., et al.. (1993). Expression of cytokines, growth factors, and their receptors in renal allograft biopsies.. PubMed. 25(1 Pt 2). 917–8. 9 indexed citations

Weis, H. J., et al.. (1984). [Intramural pseudodiverticulosis of the esophagus].. PubMed. 37(11). 276–80. 1 indexed citations

Brockerhoff, P., et al.. (1982). The influence of oral contraceptives on the composition of bile. Journal of Molecular Medicine. 60(3). 153–157. 3 indexed citations

Linden, Jan van der, et al.. (1980). Tupaias (tree shrews) — A new animal model for gallstone research. Research in Experimental Medicine. 178(1). 21–28. 2 indexed citations

Weis, H. J., et al.. (1980). Gallstone dissolution with chenodeoxycholic acid. Journal of Molecular Medicine. 58(6). 313–320. 14 indexed citations

Weis, H. J., et al.. (1979). Tupaias (tree shrews)—A new animal model for gallstone research. Research in Experimental Medicine. 176(2). 157–172. 8 indexed citations

Brockerhoff, P., et al.. (1979). [Hormonal contraception and lipid metabolism. Prospective and retrospective studies of lipid metabolic parameters during the use of contraceptives].. PubMed. 97(41). 1858–61. 1 indexed citations

Stiehl, A., P. Czygan, B. Kommerell, H. J. Weis, & K. H. Holtermüller. (1978). Ursodeoxycholic acid versus chenodeoxycholic acid. Comparison of their effects on bile acid and bile lipid composition in patients with cholesterol gallstones.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 75(6). 1016–20. 102 indexed citations

10.

Nervi, Flavio, H. J. Weis, & John M. Dietschy. (1975). The kinetic characteristics of inhibition of hepatic cholesterogenesis by lipoproteins of intestinal origin. Journal of Biological Chemistry. 250(11). 4145–4151. 75 indexed citations

11.

Weis, H. J. & John M. Dietschy. (1975). The interaction of various control mechanisms in determining the rate of hepatic cholesterogenesis in the rat. Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism. 398(2). 315–324. 38 indexed citations

12.

Weihrauch, T. R., Klaus Ewe, H. J. Weis, & G. W. Korting. (1975). Manometrische untersuchungen zur oesophagusbeteiligung bei progressiver sklerodermie. Archives of Dermatological Research. 254(1). 29–35. 3 indexed citations

13.

Weis, H. J. & John M. Dietschy. (1974). Adaptive Responses in Hepatic and Intestinal Cholesterogenesis Following Ileal Resection in the Rat*. European Journal of Clinical Investigation. 4(1). 33–41. 19 indexed citations

14.

Weis, H. J., et al.. (1974). Die Pathogenese der Hypercholesterol�mie nach Gallengangsverschlu�. Journal of Molecular Medicine. 52(23). 1122–1127. 1 indexed citations

15.

Bricker, Lee A., H. J. Weis, & Marvin D. Siperstein. (1972). In Vivo Demonstration of the Cholesterol Feedback System by Means of a Desmosterol Suppression Technique. Journal of Clinical Investigation. 51(2). 197–205. 36 indexed citations

16.

Weis, H. J. & John M. Dietschy. (1971). Presence of An Intact Cholesterol Feedback Mechanism in the Liver in Biliary Stasis. Gastroenterology. 61(1). 77–84. 30 indexed citations

17.

Weis, H. J., et al.. (1971). Die Herzpunktion bei der Ratte zur wiederholten Blutentnahme und Injektion. Research in Experimental Medicine. 156(4). 314–316. 1 indexed citations

18.

Weis, H. J., et al.. (1970). Protein-Losing Enteropathy Caused by a Retained Intestinal Tube. Gastroenterology. 58(2). 217–222. 1 indexed citations

19.

Weis, H. J.. (1970). Der Stoffwechsel des Cholesterols. Journal of Molecular Medicine. 48(20). 1203–1209. 3 indexed citations

20.

Weis, H. J. & John M. Dietschy. (1969). Failure of bile acids to control hepatic cholesterogenesis: evidence for endogenous cholesterol feedback. Journal of Clinical Investigation. 48(12). 2398–2408. 148 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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