Gwendolyn Spizz

2.4k citations
26 papers · 2.2k indexed · h-index 20
Topics
Muscle Physiology and Disorders (12 papers)Ubiquitin and proteasome pathways (6 papers)Muscle metabolism and nutrition (4 papers)
Partner nations
United StatesBulgaria

In The Last Decade

Gwendolyn Spizz

26 papers receiving 2.1k citations

Peers

Gwendolyn Spizz
Comparison fields: 5 of 100
  • Molecular Biology 1.6k
  • Genetics 291
  • Cell Biology 287
  • Oncology 240
  • Immunology 208
Replace Niels H. Gehring with:
Niels H. Gehring Germany
Douglas A. Holtzman United States
Gustav Hagen Germany
Daekwan Seo South Korea
Claudius Vincenz United States
Knut Martin Torgersen Norway
Ekkehart Lausch Germany
Menie Merika United States
Chiharu Uchida Japan
Gwendolyn Spizz relative to Niels H. Gehring Germany Niels H. Gehring's profile →
Citations per field
00.5×1.6×
Niels H. Gehring · 1×
Citations per year

Countries citing papers authored by Gwendolyn Spizz

Since Specialization
Citations

This map shows the geographic impact of Gwendolyn Spizz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gwendolyn Spizz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gwendolyn Spizz more than expected).

Fields of papers citing papers by Gwendolyn Spizz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gwendolyn Spizz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gwendolyn Spizz. The network helps show where Gwendolyn Spizz may publish in the future.

Co-authorship network of co-authors of Gwendolyn Spizz

This figure shows the co-authorship network connecting the top 25 collaborators of Gwendolyn Spizz. A scholar is included among the top collaborators of Gwendolyn Spizz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gwendolyn Spizz. Gwendolyn Spizz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 3
2 15
3 7
4 58
5 39
6 137
7 37
8 29
9 4
10 36
11 83
12 234
13 38
14 73
15 22
16 196
17 177
18 351
19 9
20 109

About Gwendolyn Spizz

Gwendolyn Spizz is a scholar working on Cell Biology, Virology and Molecular Biology, having authored 26 papers that have together received 2.2k indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (12 papers), Ubiquitin and proteasome pathways (6 papers) and Muscle metabolism and nutrition (4 papers). The work is most often cited by research in Virology (173 citations), Molecular Biology (1.6k citations) and Cell Biology (287 citations). Gwendolyn Spizz has collaborated with scholars based in United States and Bulgaria. Frequent co-authors include Eric N. Olson, E A Sternberg, Michael A. Tainsky, Celeste A. Wilcox, J S Hu, Richard Condit, Perry J. Blackshear, Douglas L. Vizard, D G Roman and Arnold W. Strauss. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Cell Biology and Molecular and Cellular Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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