Guilian Niu
- Oncology top 0.5%
- Cytokine Signaling Pathways and Interactions 10
- HER2/EGFR in Cancer Research 2
- Immunology top 1%
- Immunotherapy and Immune Responses 3
- Cancer Research top 1%
- Cancer, Hypoxia, and Metabolism 2
- Toxicology top 1%
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- Cancer Mechanisms and Therapy 3
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- PI3K/AKT/mTOR signaling in cancer 4
- Melanoma and MAPK Pathways 3
- Angiogenesis and VEGF in Cancer 3
- Co-authors
- Hua YuRichard JoveDrew M. PardollShumin ZhangTianhong WangMarcin KortylewskiMaciej KujawskiRichard Heller
- Cited by
- OncologyImmunologyCancer Research
- Partner nations
- United StatesCanadaIsrael
In The Last Decade
Guilian Niu
19 papers receiving 5.6k citations
Hit Papers
Peers
Comparison fields: 5 of 105
- Oncology 3.6k
- Immunology 2.0k
- Cancer Research 1.1k
- Toxicology 173
- Pathology and Forensic Medicine 878
Countries citing papers authored by Guilian Niu
This map shows the geographic impact of Guilian Niu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guilian Niu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guilian Niu more than expected).
Fields of papers citing papers by Guilian Niu
This network shows the impact of papers produced by Guilian Niu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guilian Niu. The network helps show where Guilian Niu may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Guilian Niu, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Persistently Activated Stat3 Maintains Constitutive NF-κB Activity in Tumorsbreakdown → | 2009 | 558 |
| 2 | 2008 | 151 | |
| 3 | 2008 | 15 | |
| 4 | 2008 | 58 | |
| 5 | 2007 | 7 | |
| 6 | 2007 | 39 | |
| 7 | 2005 | 114 | |
| 8 | Targeting Stat3 blocks both HIF-1 and VEGF expression induced by multiple oncogenic growth signaling pathwaysbreakdown → | 2005 | 489 |
| 9 | 2005 | 45 | |
| 10 | Inhibiting Stat3 signaling in the hematopoietic system elicits multicomponent antitumor immunitybreakdown → | 2005 | 813 |
| 11 | 2005 | 332 | |
| 12 | Regulation of the innate and adaptive immune responses by Stat-3 signaling in tumor cellsbreakdown → | 2003 | 923 |
| 13 | [Role of matrix metalloproteinases (MMPs) in tumor invasion and metastasis: serial studies on MMPs and TIMPs]. | 2003 | 12 |
| 14 | Constitutive Stat3 activity up-regulates VEGF expression and tumor angiogenesisbreakdown → | 2002 | 1019 |
| 15 | 2002 | 355 | |
| 16 | 2002 | 102 | |
| 17 | Constitutive activation of Stat3 by the Src and JAK tyrosine kinases participates in growth regulation of human breast carcinoma cellsbreakdown → | 2001 | 654 |
| 18 | 2000 | 5 | |
| 19 | 1999 | 28 |
About Guilian Niu
Guilian Niu is a scholar working on Oncology, Cancer Research and Immunology, having authored 19 papers that have together received 5.7k indexed citations. Recurring topics across this work include Cytokine Signaling Pathways and Interactions (10 papers), PI3K/AKT/mTOR signaling in cancer (4 papers), Cancer Mechanisms and Therapy (3 papers), Melanoma and MAPK Pathways (3 papers), Immunotherapy and Immune Responses (3 papers), Angiogenesis and VEGF in Cancer (3 papers), Cancer, Hypoxia, and Metabolism (2 papers) and HER2/EGFR in Cancer Research (2 papers). The work is most often cited by research in Oncology (3.6k citations), Immunology (2.0k citations) and Cancer Research (1.1k citations). Guilian Niu has collaborated with scholars based in United States, Canada and Israel. Frequent co-authors include Hua Yu, Richard Jove, Drew M. Pardoll, Shumin Zhang, Tianhong Wang, Marcin Kortylewski, Maciej Kujawski, Richard Heller, Domenico Coppola and Mei Huang.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.