Guili Huang

402 total citations
17 papers, 306 citations indexed

About

Guili Huang is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Guili Huang has authored 17 papers receiving a total of 306 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 6 papers in Oncology and 3 papers in Cancer Research. Recurrent topics in Guili Huang's work include Retinoids in leukemia and cellular processes (6 papers), NF-κB Signaling Pathways (3 papers) and Drug Transport and Resistance Mechanisms (2 papers). Guili Huang is often cited by papers focused on Retinoids in leukemia and cellular processes (6 papers), NF-κB Signaling Pathways (3 papers) and Drug Transport and Resistance Mechanisms (2 papers). Guili Huang collaborates with scholars based in China. Guili Huang's co-authors include Dong‐Yan Shen, Wei Zhang, Songqing Liu, Xin Xi, Xiaoli Li, Jia Yang, Xiangru Zheng, Qian Du, Wenjun Li and Qing‐Xi Chen and has published in prestigious journals such as Molecular and Cellular Biology, Analytical Biochemistry and Cancer Letters.

In The Last Decade

Guili Huang

16 papers receiving 305 citations

Peers

Guili Huang
Jinyoung Suh South Korea
Qian Zuo China
Guili Huang
Citations per year, relative to Guili Huang Guili Huang (= 1×) peers Wence Zhou

Countries citing papers authored by Guili Huang

Since Specialization
Citations

This map shows the geographic impact of Guili Huang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guili Huang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guili Huang more than expected).

Fields of papers citing papers by Guili Huang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Guili Huang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guili Huang. The network helps show where Guili Huang may publish in the future.

Co-authorship network of co-authors of Guili Huang

This figure shows the co-authorship network connecting the top 25 collaborators of Guili Huang. A scholar is included among the top collaborators of Guili Huang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Guili Huang. Guili Huang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Zheng, Xiaoying, Lingxi Kong, Ying Shan, et al.. (2025). Cost-benefit analysis of pharmacist early active consultation in patients with multidrug-resistant bacteria in China. International Journal of Clinical Pharmacy. 47(3). 863–872. 1 indexed citations
2.
Chen, Ying, et al.. (2024). A real-world pharmacovigilance study of drug-induced QT interval prolongation: analysis of spontaneous reports submitted to FAERS. Frontiers in Cardiovascular Medicine. 11. 1363382–1363382. 3 indexed citations
4.
Du, Qian, Xin Xi, Jie Dong, et al.. (2023). The impact of pharmacist early active consultation (PEAC) on multidrug resistance organism treatment outcomes: A prospective historically controlled study. Frontiers in Pharmacology. 14. 1128219–1128219. 2 indexed citations
5.
Huang, Guili, Songqin Liu, Jie Dong, et al.. (2022). PD-1 inhibitor-based adverse events in solid tumors: A retrospective real-world study. Frontiers in Pharmacology. 13. 974376–974376. 7 indexed citations
7.
Li, Wenjun, Qian Du, Xiaoli Li, et al.. (2020). Eriodictyol Inhibits Proliferation, Metastasis and Induces Apoptosis of Glioma Cells via PI3K/Akt/NF-κB Signaling Pathway. Frontiers in Pharmacology. 11. 114–114. 77 indexed citations
8.
Huang, Guili, et al.. (2019). <p>Retinoic acid receptor &alpha; facilitates human colorectal cancer progression via Akt and MMP2 signaling</p>. OncoTargets and Therapy. Volume 12. 3087–3098. 6 indexed citations
10.
Chen, Bo, et al.. (2016). Upregulation of retinoic acid receptor-β reverses drug resistance in cholangiocarcinoma cells by enhancing susceptibility to apoptosis. Molecular Medicine Reports. 14(4). 3602–3608. 12 indexed citations
12.
Huang, Guili, et al.. (2016). A spectrophotometric assay for monoamine oxidase activity with 2, 4-dinitrophenylhydrazine as a derivatized reagent. Analytical Biochemistry. 512. 18–25. 23 indexed citations
13.
Huang, Guili, Wenming Liu, Wei Zhang, et al.. (2016). IL-1β induced RXRα overexpression through activation of NF-κB signaling in gastric carcinoma. Biomedicine & Pharmacotherapy. 78. 329–334. 14 indexed citations
14.
Liu, Fan, Guili Huang, Yu Liu, et al.. (2016). Positive feedback loop of IL-1β/Akt/RARα/Akt signaling mediates oncogenic property of RARα in gastric carcinoma. Oncotarget. 8(4). 6718–6729. 6 indexed citations
15.
Huang, Guili. (2015). β-escin reverses multidrug resistance through inhibition of the GSK3β/β-catenin pathway in cholangiocarcinoma. World Journal of Gastroenterology. 21(4). 1148–1148. 42 indexed citations
17.
Huang, Guili, Qi Luo, Gang Rui, et al.. (2013). Oncogenic Activity of Retinoic Acid Receptor γ Is Exhibited through Activation of the Akt/NF-κB and Wnt/β-Catenin Pathways in Cholangiocarcinoma. Molecular and Cellular Biology. 33(17). 3416–3425. 50 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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