Graeme K. Carnegie

1.2k total citations
19 papers, 978 citations indexed

About

Graeme K. Carnegie is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cell Biology. According to data from OpenAlex, Graeme K. Carnegie has authored 19 papers receiving a total of 978 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 9 papers in Cardiology and Cardiovascular Medicine and 4 papers in Cell Biology. Recurrent topics in Graeme K. Carnegie's work include Signaling Pathways in Disease (7 papers), Protein Kinase Regulation and GTPase Signaling (5 papers) and Cellular transport and secretion (3 papers). Graeme K. Carnegie is often cited by papers focused on Signaling Pathways in Disease (7 papers), Protein Kinase Regulation and GTPase Signaling (5 papers) and Cellular transport and secretion (3 papers). Graeme K. Carnegie collaborates with scholars based in United States, United Kingdom and Japan. Graeme K. Carnegie's co-authors include John D. Scott, Lorene K. Langeberg, Christopher K. Means, F. Donelson Smith, Joseph S. Soughayer, George McConnachie, Naoto Hoshi, Dermot M.F. Cooper, Carmen Dessauer and Wei Wong and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Experimental Medicine and Genes & Development.

In The Last Decade

Graeme K. Carnegie

19 papers receiving 964 citations

Peers

Graeme K. Carnegie

MB

CCM

CB

CMN

ONCOL

Christopher K. Means United States

Kevin M. Kaltenbronn United States

Sandrine Evellin Germany

Keli Hu United States

Katalin Pászty Hungary

Dolores Moreno Spain

Ludovic Gillet France

Rebekka L. Nicol United States

Xianlong Gao United States

Michael R. Morissette United States

Christopher K. Means United States

Graeme K. Carnegie

869 ×1.1

MB

329 ×1.5

CCM

168 ×1.3

CB

83 ×0.8

CMN

47 ×0.7

ONCOL

Citations per year, relative to Graeme K. Carnegie Graeme K. Carnegie (= 1×) peers Christopher K. Means

Countries citing papers authored by Graeme K. Carnegie

Since Specialization

Citations

This map shows the geographic impact of Graeme K. Carnegie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Graeme K. Carnegie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Graeme K. Carnegie more than expected).

Fields of papers citing papers by Graeme K. Carnegie

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Graeme K. Carnegie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Graeme K. Carnegie. The network helps show where Graeme K. Carnegie may publish in the future.

Co-authorship network of co-authors of Graeme K. Carnegie

This figure shows the co-authorship network connecting the top 25 collaborators of Graeme K. Carnegie. A scholar is included among the top collaborators of Graeme K. Carnegie based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Graeme K. Carnegie. Graeme K. Carnegie is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

1.

Wang, Li, et al.. (2015). Protein Kinase A (PKA) Phosphorylation of Shp2 Protein Inhibits Its Phosphatase Activity and Modulates Ligand Specificity. Journal of Biological Chemistry. 290(19). 12058–12067. 15 indexed citations

2.

Nicodemus‐Johnson, Jessie, et al.. (2015). Genome-Wide Gene Expression Analysis Shows AKAP13-Mediated PKD1 Signaling Regulates the Transcriptional Response to Cardiac Hypertrophy. PLoS ONE. 10(7). e0132474–e0132474. 19 indexed citations

3.

Wang, Li, George S. Baillie, Andrei V. Karginov, et al.. (2015). UCR1C is a novel activator of phosphodiesterase 4 (PDE4) long isoforms and attenuates cardiomyocyte hypertrophy. Cellular Signalling. 27(5). 908–922. 28 indexed citations

4.

Liu, Xiaowen, Tao Yang, Koya Suzuki, et al.. (2015). Moesin and myosin phosphatase confine neutrophil orientation in a chemotactic gradient. The Journal of Cell Biology. 208(3). 2083OIA12–2083OIA12. 1 indexed citations

5.

Liu, Xiaowen, Tao Yang, Koya Suzuki, et al.. (2015). Moesin and myosin phosphatase confine neutrophil orientation in a chemotactic gradient. The Journal of Experimental Medicine. 212(2). 267–280. 40 indexed citations

6.

Piegeler, Tobias, E. Gina Votta‐Velis, Farnaz R. Bakhshi, et al.. (2014). Endothelial Barrier Protection by Local Anesthetics. Anesthesiology. 120(6). 1414–1428. 72 indexed citations

7.

Taglieri, Domenico M., Michelle M. Monasky, Matthew Spindler, et al.. (2013). The C-terminus of the long AKAP13 isoform (AKAP-Lbc) is critical for development of compensatory cardiac hypertrophy. Journal of Molecular and Cellular Cardiology. 66. 27–40. 32 indexed citations

8.

Wang, Li & Graeme K. Carnegie. (2013). Flow Cytometric Analysis of Bimolecular Fluorescence Complementation: A High Throughput Quantitative Method to Study Protein-protein Interaction. Journal of Visualized Experiments. 6 indexed citations

9.

Wang, Li & Graeme K. Carnegie. (2013). Flow Cytometric Analysis of Bimolecular Fluorescence Complementation: A High Throughput Quantitative Method to Study Protein-protein Interaction. Journal of Visualized Experiments. 3 indexed citations

10.

Wong, Katy A., Jessica Wilson, Angela Russo, et al.. (2012). Intersectin (ITSN) Family of Scaffolds Function as Molecular Hubs in Protein Interaction Networks. PLoS ONE. 7(4). e36023–e36023. 44 indexed citations

11.

Taglieri, Domenico M., et al.. (2012). Src Homology 2 Domain-containing Phosphatase 2 (Shp2) Is a Component of the A-kinase-anchoring Protein (AKAP)-Lbc Complex and Is Inhibited by Protein Kinase A (PKA) under Pathological Hypertrophic Conditions in the Heart. Journal of Biological Chemistry. 287(48). 40535–40546. 16 indexed citations

12.

Nicodemus‐Johnson, Jessie, et al.. (2012). Molecular evolution of a-kinase anchoring protein (AKAP)-7: implications in comparative PKA compartmentalization. BMC Evolutionary Biology. 12(1). 125–125. 19 indexed citations

13.

Carnegie, Graeme K., et al.. (2011). A-Kinase Anchoring Proteins That Regulate Cardiac Remodeling. Journal of Cardiovascular Pharmacology. 58(5). 451–458. 17 indexed citations

14.

Carnegie, Graeme K., Christopher K. Means, & John D. Scott. (2009). A‐kinase anchoring proteins: From protein complexes to physiology and disease. IUBMB Life. 61(4). 394–406. 144 indexed citations

15.

Carnegie, Graeme K., Joseph S. Soughayer, F. Donelson Smith, et al.. (2008). AKAP-Lbc Mobilizes a Cardiac Hypertrophy Signaling Pathway. Molecular Cell. 32(2). 169–179. 117 indexed citations

16.

Bauman, Andrea L., Joseph S. Soughayer, Bao Tran Nguyen, et al.. (2006). Dynamic Regulation of cAMP Synthesis through Anchored PKA-Adenylyl Cyclase V/VI Complexes. Molecular Cell. 23(6). 925–931. 177 indexed citations

17.

Carnegie, Graeme K., F. Donelson Smith, George McConnachie, Lorene K. Langeberg, & John D. Scott. (2004). AKAP-Lbc Nucleates a Protein Kinase D Activation Scaffold. Molecular Cell. 15(6). 889–899. 124 indexed citations

18.

Carnegie, Graeme K. & John D. Scott. (2003). A-kinase anchoring proteins and neuronal signaling mechanisms. Genes & Development. 17(13). 1557–1568. 53 indexed citations

19.

Carnegie, Graeme K., Judith Sleeman, Nick Morrice, et al.. (2003). Protein phosphatase 4 interacts with the Survival of Motor Neurons complex and enhances the temporal localisation of snRNPs. Journal of Cell Science. 116(10). 1905–1913. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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