Giusy Battilana

3.5k total citations · 2 hit papers
16 papers, 2.5k citations indexed

About

Giusy Battilana is a scholar working on Cell Biology, Molecular Biology and Oncology. According to data from OpenAlex, Giusy Battilana has authored 16 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Cell Biology, 9 papers in Molecular Biology and 2 papers in Oncology. Recurrent topics in Giusy Battilana's work include Hippo pathway signaling and YAP/TAZ (11 papers), Cellular Mechanics and Interactions (3 papers) and Epigenetics and DNA Methylation (2 papers). Giusy Battilana is often cited by papers focused on Hippo pathway signaling and YAP/TAZ (11 papers), Cellular Mechanics and Interactions (3 papers) and Epigenetics and DNA Methylation (2 papers). Giusy Battilana collaborates with scholars based in Italy, Germany and United States. Giusy Battilana's co-authors include Stefano Piccolo, Michelangelo Cordenonsi, Francesca Zanconato, Luca Azzolin, Silvio Bicciato, Mattia Forcato, Antonio Rosato, Erika Quaranta, Beatrice Bodega and Matteo Fassan and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Medicine.

In The Last Decade

Giusy Battilana

16 papers receiving 2.4k citations

Hit Papers

Genome-wide association between YAP/TAZ/TEAD and AP-1 at ... 2015 2026 2018 2022 2015 2022 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Giusy Battilana Italy 14 1.6k 1.4k 382 205 177 16 2.5k
Silvia Bresolin Italy 27 1.3k 0.8× 2.1k 1.5× 454 1.2× 500 2.4× 240 1.4× 76 3.2k
Sandra Soligo Italy 10 855 0.5× 1.8k 1.3× 487 1.3× 309 1.5× 156 0.9× 11 2.4k
Karin Schlegelmilch United States 7 1.5k 0.9× 1.4k 1.0× 183 0.5× 266 1.3× 67 0.4× 7 2.1k
Todd R. Heallen United States 16 1.4k 0.9× 2.3k 1.6× 152 0.4× 207 1.0× 57 0.3× 17 3.1k
Laurent Fattet United States 18 699 0.4× 1.1k 0.8× 673 1.8× 344 1.7× 138 0.8× 23 2.0k
John M. Lamar United States 21 1.1k 0.7× 1.2k 0.8× 649 1.7× 384 1.9× 206 1.2× 38 2.3k
Emanuel Rognoni United Kingdom 21 740 0.5× 777 0.6× 245 0.6× 104 0.5× 218 1.2× 32 1.8k
Jonathan M. Cooper United States 16 597 0.4× 774 0.6× 383 1.0× 193 0.9× 177 1.0× 18 1.7k
Nil Ege United Kingdom 4 724 0.4× 593 0.4× 441 1.2× 168 0.8× 140 0.8× 4 1.3k
Kurt A. Engleka United States 22 690 0.4× 2.1k 1.5× 234 0.6× 250 1.2× 193 1.1× 27 2.7k

Countries citing papers authored by Giusy Battilana

Since Specialization
Citations

This map shows the geographic impact of Giusy Battilana's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Giusy Battilana with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Giusy Battilana more than expected).

Fields of papers citing papers by Giusy Battilana

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Giusy Battilana. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Giusy Battilana. The network helps show where Giusy Battilana may publish in the future.

Co-authorship network of co-authors of Giusy Battilana

This figure shows the co-authorship network connecting the top 25 collaborators of Giusy Battilana. A scholar is included among the top collaborators of Giusy Battilana based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Giusy Battilana. Giusy Battilana is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Zanconato, Francesca, Giusy Battilana, Tito Panciera, et al.. (2022). YAP/TAZ activity in stromal cells prevents ageing by controlling cGAS–STING. Nature. 607(7920). 790–798. 184 indexed citations breakdown →
2.
Gandin, Alessandro, Anna Citron, Paolo Contessotto, et al.. (2021). Simple yet effective methods to probe hydrogel stiffness for mechanobiology. Scientific Reports. 11(1). 22668–22668. 25 indexed citations
3.
Battilana, Giusy, Francesca Zanconato, & Stefano Piccolo. (2021). Mechanisms of YAP/TAZ transcriptional control. SHILAP Revista de lepidopterología. 5(11). 167–172. 35 indexed citations
4.
Panciera, Tito, Anna Citron, Daniele Di Biagio, et al.. (2020). Publisher Correction: Reprogramming normal cells into tumour precursors requires ECM stiffness and oncogene-mediated changes of cell mechanical properties. Nature Materials. 19(4). 475–475. 3 indexed citations
5.
Castellan, Martina, Atsushi Fujimura, Francesca Zanconato, et al.. (2020). Single-cell analyses reveal YAP/TAZ as regulators of stemness and cell plasticity in glioblastoma. Nature Cancer. 2(2). 174–188. 88 indexed citations
6.
Panciera, Tito, Anna Citron, Daniele Di Biagio, et al.. (2020). Reprogramming normal cells into tumour precursors requires ECM stiffness and oncogene-mediated changes of cell mechanical properties. Nature Materials. 19(7). 797–806. 166 indexed citations
7.
Cordenonsi, Michelangelo, Lei Chang, Luca Azzolin, et al.. (2020). Abstract B39: The SWI/SNF complex is a mechanoregulated inhibitor of YAP and TAZ. Molecular Cancer Research. 18(8_Supplement). B39–B39. 1 indexed citations
8.
Totaro, Antonio, Qiuyu Zhuang, Tito Panciera, et al.. (2019). Cell phenotypic plasticity requires autophagic flux driven by YAP/TAZ mechanotransduction. Proceedings of the National Academy of Sciences. 116(36). 17848–17857. 102 indexed citations
9.
Chang, Lei, Luca Azzolin, Daniele Di Biagio, et al.. (2018). The SWI/SNF complex is a mechanoregulated inhibitor of YAP and TAZ. Nature. 563(7730). 265–269. 239 indexed citations
10.
Zanconato, Francesca, Giusy Battilana, Mattia Forcato, et al.. (2018). Transcriptional addiction in cancer cells is mediated by YAP/TAZ through BRD4. Nature Medicine. 24(10). 1599–1610. 239 indexed citations
11.
Totaro, Antonio, Martina Castellan, Giusy Battilana, et al.. (2017). YAP/TAZ link cell mechanics to Notch signalling to control epidermal stem cell fate. Nature Communications. 8(1). 15206–15206. 223 indexed citations
12.
Zanconato, Francesca, Giusy Battilana, Michelangelo Cordenonsi, & Stefano Piccolo. (2016). YAP/TAZ as therapeutic targets in cancer. Current Opinion in Pharmacology. 29. 26–33. 170 indexed citations
13.
Zanconato, Francesca, Mattia Forcato, Giusy Battilana, et al.. (2015). Genome-wide association between YAP/TAZ/TEAD and AP-1 at enhancers drives oncogenic growth. Nature Cell Biology. 17(9). 1218–1227. 840 indexed citations breakdown →
14.
Rampazzo, Elena, Alessandro Della Puppa, Chiara Frasson, et al.. (2014). Phenotypic and functional characterization of Glioblastoma cancer stem cells identified trough 5-aminolevulinic acid-assisted surgery. Journal of Neuro-Oncology. 116(3). 505–513. 23 indexed citations
15.
Schiavone, Marco, Elena Rampazzo, Giusy Battilana, et al.. (2014). Zebrafish reporter lines reveal in vivo signaling pathway activities involved in pancreatic cancer. Disease Models & Mechanisms. 7(7). 883–94. 36 indexed citations
16.
Rampazzo, Elena, Luca Persano, Francesca Pistollato, et al.. (2013). Wnt activation promotes neuronal differentiation of Glioblastoma. Cell Death and Disease. 4(2). e500–e500. 97 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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