Giuseppe Celesti

742 total citations
16 papers, 551 citations indexed

About

Giuseppe Celesti is a scholar working on Oncology, Immunology and Pathology and Forensic Medicine. According to data from OpenAlex, Giuseppe Celesti has authored 16 papers receiving a total of 551 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Oncology, 7 papers in Immunology and 4 papers in Pathology and Forensic Medicine. Recurrent topics in Giuseppe Celesti's work include Chemokine receptors and signaling (6 papers), Genetic factors in colorectal cancer (4 papers) and Immune Cell Function and Interaction (4 papers). Giuseppe Celesti is often cited by papers focused on Chemokine receptors and signaling (6 papers), Genetic factors in colorectal cancer (4 papers) and Immune Cell Function and Interaction (4 papers). Giuseppe Celesti collaborates with scholars based in Italy, United States and Switzerland. Giuseppe Celesti's co-authors include Luigi Laghi, Paolo Bianchi, Fabio Grizzi, Alberto Malesci, Alberto Mantovani, Gianluca Basso, Massimo Roncalli, Cristian Gruppi, Alessandra Balduini and Alessandro Malara and has published in prestigious journals such as The Journal of Immunology, Gastroenterology and PLoS ONE.

In The Last Decade

Giuseppe Celesti

16 papers receiving 551 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Giuseppe Celesti Italy 12 300 199 122 120 85 16 551
B. Reufi Germany 14 190 0.6× 178 0.9× 165 1.4× 147 1.2× 47 0.6× 26 593
Antonia Reale Italy 13 198 0.7× 94 0.5× 217 1.8× 315 2.6× 49 0.6× 32 560
Kaniel Cassady United States 12 173 0.6× 271 1.4× 253 2.1× 198 1.6× 49 0.6× 26 624
Brigitte Wollert‐Wulf Germany 7 169 0.6× 149 0.7× 42 0.3× 209 1.7× 213 2.5× 9 538
Eva Maria Murga Penas Germany 14 247 0.8× 111 0.6× 141 1.2× 182 1.5× 239 2.8× 31 550
B N Franklin Germany 4 190 0.6× 184 0.9× 95 0.8× 224 1.9× 32 0.4× 5 711
Naoto Nakamichi Japan 12 97 0.3× 89 0.4× 94 0.8× 173 1.4× 50 0.6× 22 366
Eugenia Martella Italy 12 351 1.2× 54 0.3× 247 2.0× 204 1.7× 106 1.2× 34 605
Nadia Coltella Italy 16 194 0.6× 109 0.5× 108 0.9× 387 3.2× 50 0.6× 24 709
Inge Lodewijckx Belgium 5 425 1.4× 291 1.5× 33 0.3× 273 2.3× 53 0.6× 7 738

Countries citing papers authored by Giuseppe Celesti

Since Specialization
Citations

This map shows the geographic impact of Giuseppe Celesti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Giuseppe Celesti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Giuseppe Celesti more than expected).

Fields of papers citing papers by Giuseppe Celesti

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Giuseppe Celesti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Giuseppe Celesti. The network helps show where Giuseppe Celesti may publish in the future.

Co-authorship network of co-authors of Giuseppe Celesti

This figure shows the co-authorship network connecting the top 25 collaborators of Giuseppe Celesti. A scholar is included among the top collaborators of Giuseppe Celesti based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Giuseppe Celesti. Giuseppe Celesti is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Porta, Chiara, Francesca Maria Consonni, Giuseppe Celesti, et al.. (2018). Protumor Steering of Cancer Inflammation by p50 NF-κB Enhances Colorectal Cancer Progression. Cancer Immunology Research. 6(5). 578–593. 44 indexed citations
2.
Malesci, Alberto, Paolo Bianchi, Giuseppe Celesti, et al.. (2017). Tumor-associated macrophages and response to 5-fluorouracil adjuvant therapy in stage III colorectal cancer. OncoImmunology. 6(12). e1342918–e1342918. 100 indexed citations
3.
Faktor, Ouriel, Luigi Laghi, Giuseppe Celesti, et al.. (2016). Feasibility of Unbiased RNA Profiling of Colorectal Tumors: A Proof of Principle. PLoS ONE. 11(7). e0159522–e0159522. 3 indexed citations
4.
Malara, Alessandro, Cristian Gruppi, Giuseppe Celesti, et al.. (2016). Brief Report: Alternative Splicing of Extra Domain A (EIIIA) of Fibronectin Plays a Tissue-Specific Role in Hematopoietic Homeostasis. Stem Cells. 34(8). 2263–2268. 9 indexed citations
5.
Abbonante, Vittorio, Christian A. Di Buduo, Cristian Gruppi, et al.. (2016). Thrombopoietin/TGF-β1 Loop Regulates Megakaryocyte Extracellular Matrix Component Synthesis. Stem Cells. 34(4). 1123–1133. 46 indexed citations
6.
Erreni, Marco, Imran Siddiqui, Giulia Marelli, et al.. (2015). The Fractalkine-Receptor Axis Improves Human Colorectal Cancer Prognosis by Limiting Tumor Metastatic Dissemination. The Journal of Immunology. 196(2). 902–914. 33 indexed citations
7.
Savino, Benedetta, Nicoletta Caronni, Achille Anselmo, et al.. (2014). ERK-Dependent Downregulation of the Atypical Chemokine Receptor D6 Drives Tumor Aggressiveness in Kaposi Sarcoma. Cancer Immunology Research. 2(7). 679–689. 34 indexed citations
8.
Bonecchi, Raffaella, Benedetta Savino, Nicoletta Caronni, et al.. (2014). Atypical chemokine receptor 2: a brake against Kaposi's sarcoma aggressiveness. OncoImmunology. 3(12). e955337–e955337. 13 indexed citations
9.
Malesci, Alberto, Gianluca Basso, Paolo Bianchi, et al.. (2014). Molecular heterogeneity and prognostic implications of synchronous advanced colorectal neoplasia. British Journal of Cancer. 110(5). 1228–1235. 16 indexed citations
10.
Celesti, Giuseppe, G.Q. Di, Paolo Bianchi, et al.. (2013). Early expression of the fractalkine receptor CX3CR1 in pancreatic carcinogenesis. British Journal of Cancer. 109(9). 2424–2433. 23 indexed citations
11.
Celesti, Giuseppe, Giuseppe Di, Paolo Bianchi, et al.. (2013). Presence of Twist1-Positive Neoplastic Cells in the Stroma of Chromosome-Unstable Colorectal Tumors. Gastroenterology. 145(3). 647–657.e15. 48 indexed citations
12.
Malara, Alessandro, Manuela Currao, Cristian Gruppi, et al.. (2013). Megakaryocytes Contribute to the Bone Marrow-Matrix Environment by Expressing Fibronectin, Type IV Collagen, and Laminin. Stem Cells. 32(4). 926–937. 104 indexed citations
13.
Di, Giuseppe, Giuseppe Celesti, Fabio Grizzi, et al.. (2013). Combined low-FOXP3+ and -CD3+tumor infiltrating lymphocytes: a signature of stage II MSS colorectal cancer at high-risk of recurrence. Journal for ImmunoTherapy of Cancer. 1(S1). 1 indexed citations
14.
Laghi, Luigi, Paolo Bianchi, Gabriele Delconte, et al.. (2012). MSH3 Protein Expression and Nodal Status in MLH1-Deficient Colorectal Cancers. Clinical Cancer Research. 18(11). 3142–3153. 20 indexed citations
15.
Laghi, Luigi, Stefania Beghelli, Antonino Spinelli, et al.. (2012). Irrelevance of Microsatellite Instability in the Epidemiology of Sporadic Pancreatic Ductal Adenocarcinoma. PLoS ONE. 7(9). e46002–e46002. 56 indexed citations
16.
Erreni, Marco, Luigi Laghi, Giuseppe Celesti, et al.. (2010). W1767 Expression and Function of the Chemokine CX3CL1 and Its Receptor Cx3cr1 in Human Colorectal Cancer. Gastroenterology. 138(5). S–736. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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