Gill Webster

1.3k total citations · 1 hit paper
24 papers, 1.1k citations indexed

About

Gill Webster is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Gill Webster has authored 24 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Immunology, 6 papers in Molecular Biology and 6 papers in Oncology. Recurrent topics in Gill Webster's work include Immunotherapy and Immune Responses (8 papers), Immune Response and Inflammation (5 papers) and Multiple Sclerosis Research Studies (4 papers). Gill Webster is often cited by papers focused on Immunotherapy and Immune Responses (8 papers), Immune Response and Inflammation (5 papers) and Multiple Sclerosis Research Studies (4 papers). Gill Webster collaborates with scholars based in New Zealand, United Kingdom and United States. Gill Webster's co-authors include Neil D. Perkins, Andrew Snowden, Lisa A. Anderson, Paul R. Harrison, Leonard L. Wu, Jerry Lanfear, Janis Fleming, Anne Camille La Flamme, Madeleine P. J. White and Kunle Odunsi and has published in prestigious journals such as Journal of Clinical Oncology, PLoS ONE and Molecular and Cellular Biology.

In The Last Decade

Gill Webster

24 papers receiving 1.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Transcriptional Cross Talk between NF-κB and p53

1999 518 citations Gill Webster, Neil D. Perkins Molecular and Cellular Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Gill Webster New Zealand	15	573	425	354	324	103	24	1.1k
Ye Jin Jang South Korea	13	617 1.1×	205 0.5×	237 0.7×	328 1.0×	37 0.4×	15	1.1k
Matthew R. Farren United States	19	396 0.7×	625 1.5×	482 1.4×	195 0.6×	37 0.4×	34	1.3k
Daniela S. Daniela Sanchez Bassères Brazil	18	925 1.6×	379 0.9×	306 0.9×	504 1.6×	42 0.4×	37	1.6k
Norma Llansa United States	13	534 0.9×	285 0.7×	265 0.7×	262 0.8×	32 0.3×	15	1.1k
James W. DeWille United States	23	738 1.3×	326 0.8×	167 0.5×	178 0.5×	124 1.2×	38	1.2k
Caroline H. Arnott United Kingdom	8	489 0.9×	478 1.1×	597 1.7×	394 1.2×	25 0.2×	11	1.3k
Hsu-Ping Kuo United States	12	892 1.6×	274 0.6×	167 0.5×	187 0.6×	25 0.2×	12	1.1k
Taylor B. Guo China	18	460 0.8×	230 0.5×	663 1.9×	146 0.5×	30 0.3×	35	1.5k
Claudia Fiorini Italy	19	587 1.0×	194 0.5×	254 0.7×	199 0.6×	30 0.3×	26	1.1k

Countries citing papers authored by Gill Webster

Since Specialization

Citations

This map shows the geographic impact of Gill Webster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gill Webster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gill Webster more than expected).

Fields of papers citing papers by Gill Webster

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gill Webster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gill Webster. The network helps show where Gill Webster may publish in the future.

Co-authorship network of co-authors of Gill Webster

This figure shows the co-authorship network connecting the top 25 collaborators of Gill Webster. A scholar is included among the top collaborators of Gill Webster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gill Webster. Gill Webster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Graner, Michael W., Cheryl L. Beseler, Gill Webster, et al.. (2023). Plasma IgG aggregates as biomarkers for multiple sclerosis. Clinical Immunology. 256. 109801–109801. 6 indexed citations

Marczynska, Joanna, et al.. (2021). Innate Signaling in the CNS Prevents Demyelination in a Focal EAE Model. Frontiers in Neuroscience. 15. 682451–682451. 6 indexed citations

McGray, AJ Robert, Cheryl Eppolito, Anthony Miliotto, et al.. (2021). A prime/boost vaccine platform efficiently identifies CD27 agonism and depletion of myeloid-derived suppressor cells as therapies that rationally combine with checkpoint blockade in ovarian cancer. Cancer Immunology Immunotherapy. 70(12). 3451–3460. 11 indexed citations

Ishihara, Mikiya, Yoshihiro Miyahara, Daisuke Muraoka, et al.. (2020). First-in-human phase I clinical trial of the NY-ESO-1 protein cancer vaccine with NOD2 and TLR9 stimulants in patients with NY-ESO-1-expressing refractory solid tumors. Cancer Immunology Immunotherapy. 69(4). 663–675. 21 indexed citations

Khorooshi, Reza, Joanna Marczynska, Christian Rønn Hansen, et al.. (2020). Innate signaling within the central nervous system recruits protective neutrophils. Acta Neuropathologica Communications. 8(1). 2–2. 14 indexed citations

McGray, AJ Robert, Ruea-Yea Huang, Sebastiano Battaglia, et al.. (2019). Oncolytic Maraba virus armed with tumor antigen boosts vaccine priming and reveals diverse therapeutic response patterns when combined with checkpoint blockade in ovarian cancer. Journal for ImmunoTherapy of Cancer. 7(1). 189–189. 45 indexed citations

Thio, Christina Li‐Ping, Alan Chuan‐Ying Lai, Po‐Yu Chi, Gill Webster, & Ya‐Jen Chang. (2019). Toll-like receptor 9–dependent interferon production prevents group 2 innate lymphoid cell–driven airway hyperreactivity. Journal of Allergy and Clinical Immunology. 144(3). 682–697.e9. 35 indexed citations

White, Madeleine P. J., et al.. (2018). Innate IFN-γ ameliorates experimental autoimmune encephalomyelitis and promotes myeloid expansion and PDL-1 expression. Scientific Reports. 8(1). 259–259. 24 indexed citations

Mainini, Francesco, David S. Larsen, Gill Webster, Sarah L. Young, & Michael R. Eccles. (2018). MIS416 as a siRNA Delivery System with the Ability to Target Antigen-Presenting Cells. Nucleic Acid Therapeutics. 28(4). 225–232. 2 indexed citations

10.

Ishihara, Mikiya, Yoshihiro Miyahara, Naozumi Harada, et al.. (2018). First-in-human phase I clinical trial of NY-ESO-1 protein cancer vaccine with a novel adjuvant MIS416, NOD2 and TLR9 stimulant, for patients with NY-ESO-1 expressing solid tumors.. Journal of Clinical Oncology. 36(15_suppl). e15176–e15176. 1 indexed citations

11.

Webster, Gill, Dalice Sim, Anne Camille La Flamme, & Nancy E. Mayo. (2017). Evaluation of neurological changes in secondary progressive multiple sclerosis patients treated with immune modulator MIS416: results from a feasibility study. Pilot and Feasibility Studies. 3(1). 60–60. 3 indexed citations

12.

Mainini, Francesco, David S. Larsen, Gill Webster, Sarah L. Young, & Michael R. Eccles. (2015). Bridging Small Molecules to Modified Bacterial Microparticles Using a Disulphide Linkage: MIS416 as a Cargo Delivery System. PLoS ONE. 10(12). e0145403–e0145403. 2 indexed citations

13.

Luckey, Alison, et al.. (2015). Safety, tolerability and pharmacodynamics of a novel immunomodulator, MIS416, in patients with chronic progressive multiple sclerosis. Multiple Sclerosis Journal - Experimental Translational and Clinical. 1. 2450003353–2450003353. 10 indexed citations

14.

White, Madeleine P. J., Gill Webster, David O’Sullivan, Sarrabeth Stone, & Anne Camille La Flamme. (2014). Targeting Innate Receptors with MIS416 Reshapes Th Responses and Suppresses CNS Disease in a Mouse Model of Multiple Sclerosis. PLoS ONE. 9(1). e87712–e87712. 28 indexed citations

15.

Webster, Gill, et al.. (2013). Development of a Combined Bovine Colostrum and Immune-Stimulatory Carbohydrate Nutraceutical for Enhancement of Endogenous Stem Cell Activity. 6(1). 2 indexed citations

16.

Knight, Deborah A., et al.. (2010). MIS416, a non-toxic microparticle adjuvant derived from Propionibacterium acnes comprising immunostimulatory muramyl dipeptide and bacterial DNA promotes cross-priming and Th1 immunity. Vaccine. 29(3). 545–557. 34 indexed citations

17.

Reid, Glen, Natacha Coppieters, Rachna Patel, et al.. (2009). Potent subunit-specific effects on cell growth and drug sensitivity from optimised siRNA-mediated silencing of ribonucleotide reductase.. PubMed. 5(1). 321–30. 30 indexed citations

18.

Snowden, Andrew, Lisa A. Anderson, Gill Webster, & Neil D. Perkins. (2000). A Novel Transcriptional Repression Domain Mediates p21^WAF1/CIP1 Induction of p300 Transactivation. Molecular and Cellular Biology. 20(8). 2676–2686. 114 indexed citations

19.

Webster, Gill & Neil D. Perkins. (1999). Transcriptional Cross Talk between NF-κB and p53. Molecular and Cellular Biology. 19(5). 3485–3495. 518 indexed citations breakdown →

20.

Lanfear, Jerry, Janis Fleming, Leonard L. Wu, Gill Webster, & Paul R. Harrison. (1994). The selenium metabolite selenodiglutathione induces p⁵³ and apoptosis: relevance to the chemopreventive effects of selenium?. Carcinogenesis. 15(7). 1387–1392. 108 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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