Gilbert O’Young

2.0k total citations
22 papers, 1.4k citations indexed

About

Gilbert O’Young is a scholar working on Molecular Biology, Oncology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Gilbert O’Young has authored 22 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 12 papers in Oncology and 4 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Gilbert O’Young's work include Cancer Cells and Metastasis (4 papers), TGF-β signaling in diseases (3 papers) and Melanoma and MAPK Pathways (3 papers). Gilbert O’Young is often cited by papers focused on Cancer Cells and Metastasis (4 papers), TGF-β signaling in diseases (3 papers) and Melanoma and MAPK Pathways (3 papers). Gilbert O’Young collaborates with scholars based in United States, United Kingdom and New Zealand. Gilbert O’Young's co-authors include Ann M. Kapoun, Andrew A. Protter, Andrew Lam, Timothy Hoey, Marcus Fischer, Diana Quon, George F. Schreiner, Deborah Damm, Ying Jing and Belinda Cancilla and has published in prestigious journals such as Nature, Blood and Circulation Research.

In The Last Decade

Gilbert O’Young

22 papers receiving 1.4k citations

Peers

Gilbert O’Young
Joshua I. Greenberg United States
Bálint Nagy Hungary
Alejandro J. Garcia United States
Cecilia A. Fernández United States
Bradford A. Young United States
Joshua I. Greenberg United States
Gilbert O’Young
Citations per year, relative to Gilbert O’Young Gilbert O’Young (= 1×) peers Joshua I. Greenberg

Countries citing papers authored by Gilbert O’Young

Since Specialization
Citations

This map shows the geographic impact of Gilbert O’Young's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gilbert O’Young with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gilbert O’Young more than expected).

Fields of papers citing papers by Gilbert O’Young

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gilbert O’Young. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gilbert O’Young. The network helps show where Gilbert O’Young may publish in the future.

Co-authorship network of co-authors of Gilbert O’Young

This figure shows the co-authorship network connecting the top 25 collaborators of Gilbert O’Young. A scholar is included among the top collaborators of Gilbert O’Young based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gilbert O’Young. Gilbert O’Young is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Argast, Gretchen, Belinda Cancilla, Fiore Cattaruzza, et al.. (2018). Abstract 5627: Anti-TIGIT biomarker study: Inhibition of TIGIT induces loss of Tregs from tumors and requires effector function for tumor growth inhibition. Cancer Research. 78(13_Supplement). 5627–5627. 4 indexed citations
2.
Lim, Jing Shan, Alvaro Ibaseta, Marcus Fischer, et al.. (2017). Intratumoural heterogeneity generated by Notch signalling promotes small-cell lung cancer. Nature. 545(7654). 360–364. 311 indexed citations
3.
Fischer, Marcus, Vincent Yeung, Fiore Cattaruzza, et al.. (2017). RSPO3 antagonism inhibits growth and tumorigenicity in colorectal tumors harboring common Wnt pathway mutations. Scientific Reports. 7(1). 15270–15270. 37 indexed citations
4.
Fischer, Marcus, Belinda Cancilla, Vincent Yeung, et al.. (2017). WNT antagonists exhibit unique combinatorial antitumor activity with taxanes by potentiating mitotic cell death. Science Advances. 3(6). e1700090–e1700090. 122 indexed citations
5.
Cattaruzza, Fiore, Min Wang, Alayne Brunner, et al.. (2017). Abstract 599: Pharmacodynamic biomarkers for anti-TIGIT treatment and prevalence of TIGIT expression in multiple solid tumor types. Cancer Research. 77(13_Supplement). 599–599. 2 indexed citations
6.
Zhang, Chun, Yuwang Liu, Min Wang, et al.. (2016). Abstract 404: Development of a RSPO3 CLIA-validated assay as a predictive biomarker for response to anti-RSPO3 antibody treatment in patients with solid tumors. Cancer Research. 76(14_Supplement). 404–404. 1 indexed citations
7.
Brunner, Alayne, Fiore Cattaruzza, Wan-Ching Yen, et al.. (2016). Abstract 4652: Effects of anti-DLL4 treatment on non-small cell lung cancer (NSCLC) human xenograft tumors. Cancer Research. 76(14_Supplement). 4652–4652. 3 indexed citations
9.
Shah, Jalpa, Gilbert O’Young, Jie Wei, et al.. (2015). Abstract 2323: Tarextumab (Anti-NOTCH2/3) reverses NOTCH2 and NOTCH3-dependent tumorigenicity and metastases in small cell lung cancer. Cancer Research. 75(15_Supplement). 2323–2323. 1 indexed citations
10.
Srivastava, Minu K., Christopher L. Murriel, Erin Mayes, et al.. (2015). Co-targeting of delta-like ligand 4 (DLL4) and vascular endothelial growth factor a (VEGF) with programmed death 1 (PD1) blockade inhibits tumor growth and facilitates anti-tumor immune responses. Journal for ImmunoTherapy of Cancer. 3(S2). 2 indexed citations
11.
Fischer, Marcus, Wan-Ching Yen, Ann M. Kapoun, et al.. (2010). Anti-DLL4 Inhibits Growth and Reduces Tumor-Initiating Cell Frequency in Colorectal Tumors with Oncogenic KRAS Mutations. Cancer Research. 71(5). 1520–1525. 100 indexed citations
12.
Medicherla, Satyanarayana, Lingyun Li, Ying Jing, et al.. (2008). Antitumor activity of TGF-beta inhibitor is dependent on the microenvironment.. PubMed. 27(6B). 4149–57. 54 indexed citations
13.
Li, Lingyun, Ann M. Kapoun, Satyanarayana Medicherla, et al.. (2007). Inhibition of Transforming Growth Factor β Signaling Reduces Pancreatic Adenocarcinoma Growth and Invasiveness. Molecular Pharmacology. 72(1). 152–161. 103 indexed citations
14.
Li, Zhihe, Ying Zhang, Ying Jing, et al.. (2007). Recombinant Vascular Endothelial Growth Factor 121 Attenuates Hypertension and Improves Kidney Damage in a Rat Model of Preeclampsia. Hypertension. 50(4). 686–692. 201 indexed citations
15.
Medicherla, Satyanarayana, Ying Jing, Ruban Mangadu, et al.. (2006). A Selective p38α Mitogen-Activated Protein Kinase Inhibitor Reverses Cartilage and Bone Destruction in Mice with Collagen-Induced Arthritis. Journal of Pharmacology and Experimental Therapeutics. 318(1). 132–141. 65 indexed citations
16.
Nguyen, Aaron, Elizabeth G. Stebbins, Margaret Henson, et al.. (2006). Normalizing the bone marrow microenvironment with p38 inhibitor reduces multiple myeloma cell proliferation and adhesion and suppresses osteoclast formation. Experimental Cell Research. 312(10). 1909–1923. 49 indexed citations
18.
Li, Zhihe, Thomas-Toan Tran, Ying Jing, et al.. (2004). p38α Mitogen-Activated Protein Kinase Inhibition Improves Cardiac Function and Reduces Myocardial Damage in Isoproterenol-Induced Acute Myocardial Injury in Rats. Journal of Cardiovascular Pharmacology. 44(4). 486–492. 34 indexed citations
19.
Kapoun, Ann M., Faquan Liang, Gilbert O’Young, et al.. (2004). B-Type Natriuretic Peptide Exerts Broad Functional Opposition to Transforming Growth Factor-β in Primary Human Cardiac Fibroblasts. Circulation Research. 94(4). 453–461. 239 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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