Gerard Schwartz

745 total citations
18 papers, 488 citations indexed

About

Gerard Schwartz is a scholar working on Clinical Biochemistry, Molecular Biology and Rheumatology. According to data from OpenAlex, Gerard Schwartz has authored 18 papers receiving a total of 488 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Clinical Biochemistry, 8 papers in Molecular Biology and 4 papers in Rheumatology. Recurrent topics in Gerard Schwartz's work include Metabolism and Genetic Disorders (12 papers), Folate and B Vitamins Research (4 papers) and Mitochondrial Function and Pathology (3 papers). Gerard Schwartz is often cited by papers focused on Metabolism and Genetic Disorders (12 papers), Folate and B Vitamins Research (4 papers) and Mitochondrial Function and Pathology (3 papers). Gerard Schwartz collaborates with scholars based in Israel, United States and United Kingdom. Gerard Schwartz's co-authors include A. C. Tarn, Yair Anikster, Yosef Shiloh, Savio L.C. Woo, B.M. Dean, Elaine Gale, Frank Becker, J. M. McNally, Smadar Avigad and G. F. Bottazzo and has published in prestigious journals such as Nature, Journal of Clinical Oncology and Human Molecular Genetics.

In The Last Decade

Gerard Schwartz

18 papers receiving 470 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Gerard Schwartz Israel 12 262 195 178 118 105 18 488
Sylvia Philipps Canada 11 50 0.2× 146 0.7× 63 0.4× 80 0.7× 32 0.3× 32 413
Mercedes Martínez‐Pardo Spain 16 589 2.2× 559 2.9× 94 0.5× 165 1.4× 38 0.4× 35 782
Kenpei Matsuoka Japan 6 207 0.8× 378 1.9× 79 0.4× 7 0.1× 94 0.9× 8 566
Mark M. Danney United States 7 93 0.4× 137 0.7× 58 0.3× 20 0.2× 59 0.6× 8 376
Darius J. Adams United States 10 328 1.3× 328 1.7× 71 0.4× 64 0.5× 13 0.1× 17 464
Inga Knudsen Denmark 11 463 1.8× 448 2.3× 61 0.3× 94 0.8× 9 0.1× 13 588
Beverley A. Millward United Kingdom 10 90 0.3× 98 0.5× 45 0.3× 8 0.1× 42 0.4× 14 340
Alice Basinger United States 8 47 0.2× 151 0.8× 56 0.3× 16 0.1× 39 0.4× 11 275
Y Okano Japan 13 440 1.7× 315 1.6× 67 0.4× 176 1.5× 23 0.2× 17 507
N Bruni France 6 20 0.1× 199 1.0× 72 0.4× 188 1.6× 142 1.4× 6 422

Countries citing papers authored by Gerard Schwartz

Since Specialization
Citations

This map shows the geographic impact of Gerard Schwartz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gerard Schwartz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gerard Schwartz more than expected).

Fields of papers citing papers by Gerard Schwartz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gerard Schwartz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gerard Schwartz. The network helps show where Gerard Schwartz may publish in the future.

Co-authorship network of co-authors of Gerard Schwartz

This figure shows the co-authorship network connecting the top 25 collaborators of Gerard Schwartz. A scholar is included among the top collaborators of Gerard Schwartz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gerard Schwartz. Gerard Schwartz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Pode‐Shakked, Ben, Alon Harmelin, Noa Stettner, et al.. (2013). Man made disease: Clinical manifestations of low phenylalanine levels in an inadequately treated phenylketonuria patient and mouse study. Molecular Genetics and Metabolism. 110. S66–S70. 19 indexed citations
2.
Irvin, William, Peter Rubin, Daniel R. Carrizosa, et al.. (2011). Comprehensive CYP2D6 genotyping in a multiracial population shows differences in allele frequencies between races.. Journal of Clinical Oncology. 29(15_suppl). e11095–e11095. 1 indexed citations
3.
Bercovich, Dani, Tal Yardeni, Joël Zlotogora, et al.. (2008). A Mutation Analysis of the Phenylalanine Hydroxylase (PAH) Gene in the Israeli Population. Annals of Human Genetics. 72(3). 305–309. 23 indexed citations
4.
Bercovich, Dani, Joël Zlotogora, Tal Yardeni, et al.. (2008). Genotype–phenotype correlations analysis of mutations in the phenylalanine hydroxylase (PAH) gene. Journal of Human Genetics. 53(5). 407–418. 36 indexed citations
5.
Fiocco, Alexandra, N.P.V. Nair, Gerard Schwartz, et al.. (2007). Influence of genetic polymorphisms in the apolipoprotein (APOE) and the butyrylcholinesterase (BCHE) gene on stress markers in older adults: A 3-year study. Neurobiology of Aging. 30(6). 1001–1005. 15 indexed citations
6.
Modan‐Moses, Dalit, et al.. (2007). Peak bone mass in patients with phenylketonuria. Journal of Inherited Metabolic Disease. 30(2). 202–208. 81 indexed citations
7.
Kleiman, Sandra E., Smadar Avigad, Lina Vanagaite, et al.. (1994). Origins of Hyperphenylalaninemiain Israel. European Journal of Human Genetics. 2(1). 24–34. 11 indexed citations
8.
Avigad, Smadar, et al.. (1993). A missense mutation, S349P, completely inactivates phenylalanine hydroxylase in North African Jews with phenylketonuria. Human Genetics. 90(6). 645–9. 22 indexed citations
9.
Kleiman, Sandra E., et al.. (1993). Inactivation of phenylalanine hydroxylase by a missense mutation, R270S, in a Palestinian kinship with phenylketonuria. Human Molecular Genetics. 2(5). 605–606. 6 indexed citations
10.
Kleiman, Sandra E., Lina Vanagaite, Gerard Schwartz, et al.. (1993). Phenylketonuria: variable phenotypic outcomes of the R261Q mutation and maternal PKU in the offspring of a healthy homozygote.. Journal of Medical Genetics. 30(4). 284–288. 11 indexed citations
11.
Kleiman, Sandra E., Gerard Schwartz, Savio L.C. Woo, & Yosef Shiloh. (1992). A 22-bp deletion in the phenylalanine hydroxylase gene causing phenylketonuria in an Arab family. Human Mutation. 1(4). 344–346. 5 indexed citations
12.
13.
Avigad, Smadar, Sandra E. Kleiman, B. E. Cohen, et al.. (1991). Compound heterozygosity in nonphenylketonuria hyperphenylalanemia: the contribution of mutations for classical phenylketonuria.. PubMed. 49(2). 393–9. 26 indexed citations
14.
Avigad, Smadar, B. E. Cohen, Sofía Bauer, et al.. (1990). A single origin of phenylketonuria in Yemenite Jews. Nature. 344(6262). 168–170. 64 indexed citations
15.
Hitman, G. A., J. A. Sachs, Paul G. Cassell, et al.. (1986). A DR3-related DX? gene polymorphism strongly associates with insulin-dependent diabetes mellitus. Immunogenetics. 23(1). 47–51. 48 indexed citations
16.
Dean, B.M., Frank Becker, J. M. McNally, et al.. (1986). Insulin autoantibodies in the pre-diabetic period: Correlation with islet cell antibodies and development of diabetes. Diabetologia. 29(5). 339–342. 102 indexed citations
17.
Bujanover, Yoram, et al.. (1985). Lactose Malabsorption Is Not a Cause of Diarrhea During Phototherapy. Journal of Pediatric Gastroenterology and Nutrition. 4(2). 196–198. 6 indexed citations
18.
Schwartz, Gerard, et al.. (1981). Identification of 2-Ethyl-Hexanoic (2-Ethyl-Caproic) Acid in Beer. Journal of the American Society of Brewing Chemists. 39(2). 44–46. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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