George Bosworth Brown

7.2k total citations
170 papers, 4.1k citations indexed

About

George Bosworth Brown is a scholar working on Molecular Biology, Organic Chemistry and Cellular and Molecular Neuroscience. According to data from OpenAlex, George Bosworth Brown has authored 170 papers receiving a total of 4.1k indexed citations (citations by other indexed papers that have themselves been cited), including 73 papers in Molecular Biology, 64 papers in Organic Chemistry and 16 papers in Cellular and Molecular Neuroscience. Recurrent topics in George Bosworth Brown's work include Synthesis and Biological Evaluation (36 papers), Ion channel regulation and function (21 papers) and Chemical Reaction Mechanisms (17 papers). George Bosworth Brown is often cited by papers focused on Synthesis and Biological Evaluation (36 papers), Ion channel regulation and function (21 papers) and Chemical Reaction Mechanisms (17 papers). George Bosworth Brown collaborates with scholars based in United States, Japan and Canada. George Bosworth Brown's co-authors include John W. Daly, Wayne J. Brouillette, M. Earl Balis, John Davoll, William A. Catterall, Frederick A. Fuhrman, Harry S. Mosher, Yong H. Kim, Timothy M. DeLorey and Herman W. Smith and has published in prestigious journals such as Nature, Science and New England Journal of Medicine.

In The Last Decade

George Bosworth Brown

164 papers receiving 3.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
George Bosworth Brown United States 34 2.5k 1.1k 661 256 209 170 4.1k
Fritz Lipmann United States 61 8.2k 3.3× 752 0.7× 388 0.6× 102 0.4× 145 0.7× 172 11.0k
Wolfgang Pfleiderer Germany 39 4.3k 1.7× 3.4k 3.1× 252 0.4× 103 0.4× 841 4.0× 581 7.7k
John J. Baldwin Australia 35 2.3k 0.9× 2.2k 2.0× 297 0.4× 55 0.2× 187 0.9× 194 6.4k
Bernhard Witkop United States 56 6.0k 2.4× 3.5k 3.2× 1.1k 1.7× 400 1.6× 100 0.5× 296 11.9k
Tadeusz F. Molinski United States 46 3.3k 1.3× 3.8k 3.4× 510 0.8× 257 1.0× 78 0.4× 222 8.7k
Shirley Schreier Brazil 36 2.8k 1.1× 1.1k 1.0× 343 0.5× 130 0.5× 126 0.6× 135 4.8k
Berton C. Pressman United States 34 4.4k 1.7× 435 0.4× 691 1.0× 47 0.2× 46 0.2× 74 7.5k
Youssef Hatefi United States 57 9.3k 3.7× 563 0.5× 751 1.1× 47 0.2× 71 0.3× 190 11.6k
W. Leimgruber United States 17 2.2k 0.9× 871 0.8× 269 0.4× 79 0.3× 51 0.2× 28 4.2k
John Westley United States 39 2.2k 0.9× 1.1k 1.0× 494 0.7× 155 0.6× 50 0.2× 179 5.3k

Countries citing papers authored by George Bosworth Brown

Since Specialization
Citations

This map shows the geographic impact of George Bosworth Brown's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by George Bosworth Brown with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites George Bosworth Brown more than expected).

Fields of papers citing papers by George Bosworth Brown

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by George Bosworth Brown. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by George Bosworth Brown. The network helps show where George Bosworth Brown may publish in the future.

Co-authorship network of co-authors of George Bosworth Brown

This figure shows the co-authorship network connecting the top 25 collaborators of George Bosworth Brown. A scholar is included among the top collaborators of George Bosworth Brown based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with George Bosworth Brown. George Bosworth Brown is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Banerjee, Sami, Ernest V. Curto, Matthew L. Beckman, et al.. (2005). Expression of functional scorpion neurotoxin Lqq-V in E.coli. Peptides. 27(1). 49–54. 14 indexed citations
2.
Lu, Chiung‐Mei, et al.. (1998). Isolation of the 5'-Flanking Region for Human Brain Sodium Channel Subtype II α-Subunit. Journal of Molecular Neuroscience. 11(3). 179–182. 8 indexed citations
3.
Lu, Chiung‐Mei & George Bosworth Brown. (1998). Isolation of a human-brain sodium-channel gene encoding two isoforms of the subtype III α-subunit. Journal of Molecular Neuroscience. 10(1). 67–70. 16 indexed citations
4.
George, Alfred L., Harry A. Drabkin, Jianying Han, et al.. (1995). Assignment of the human heart tetrodotoxin-resistant voltage-gated Na<sup>+</sup> channel α-subunit gene (SCN5A) to band 3p21. Cytogenetic and Genome Research. 68(1-2). 67–70. 87 indexed citations
5.
Teisberg, Elizabeth, McDowell Porter, & George Bosworth Brown. (1994). Making competition in health care work.. PubMed. 72(4). 131–41. 52 indexed citations
6.
George, Alfred L., et al.. (1994). Assignment of a Human Voltage-Dependent Sodium Channel α-Subunit Gene (SCN6A) to 2q21-q23. Genomics. 19(2). 395–397. 9 indexed citations
7.
Brown, George Bosworth. (1988). Batrachotoxin: A Window on The Allosteric Nature of The Voltage-Sensitive Sodium Channel. International review of neurobiology. 29. 77–116. 36 indexed citations
8.
9.
Brown, George Bosworth & John W. Daly. (1981). Interaction of batrachotoxinin-A benzoate with voltage-sensitive sodium channels: The effects of pH. Cellular and Molecular Neurobiology. 1(4). 361–371. 15 indexed citations
10.
Brown, George Bosworth, et al.. (1981). Batrachotoxinin-A 20-?-benzoate: A new radioactive ligand for voltage sensitive sodium channels. Cellular and Molecular Neurobiology. 1(1). 19–40. 50 indexed citations
11.
Teller, Morris N., et al.. (1978). Comparison of the oncogenicities of four structurally isomeric N-hydroxyxanthines.. PubMed. 38(7). 2038–42. 6 indexed citations
12.
Giner‐Sorolla, Alfredo, et al.. (1973). Nitrosaminopurines and nucleosides, synthesis and biological activity. Journal of Medicinal Chemistry. 16(4). 365–369. 6 indexed citations
13.
Corbin, E, et al.. (1972). Enzymatic activation of the oncogen 3-hydroxyxanthine.. PubMed. 32(3). 637–42. 19 indexed citations
14.
Birdsall, N.J.M., et al.. (1971). Purine N-oxides—XL. Tetrahedron. 27(24). 5969–5978. 15 indexed citations
15.
Brown, George Bosworth, et al.. (1970). Oncogenic Purine Derivatives: Evidence for a Possible Proximate Oncogen. Science. 167(3925). 1622–1624. 23 indexed citations
16.
Brown, George Bosworth, et al.. (1969). Purine N-oxides. XXII. Structures of 3-hydroxyxanthine and guanine 3-oxide. The Journal of Organic Chemistry. 34(4). 978–981. 21 indexed citations
17.
Smith, Herman W., et al.. (1960). Purine N-Oxides. VII. Reaction of Aminopurine 1-N-Oxides with Acetic Anhydride1. Journal of the American Chemical Society. 82(5). 1148–1152. 6 indexed citations
18.
Brown, George Bosworth. (1958). The Use of Soviet Medical Research Information by American Medical Scientists. Europe PMC (PubMed Central). 46(4). 643–643. 1 indexed citations
19.
Roll, Paul M., et al.. (1956). THE UTILIZATION OF NUCLEOTIDES BY THE MAMMAL. Journal of Biological Chemistry. 220(1). 439–454. 89 indexed citations
20.
Brown, George Bosworth, et al.. (1955). Repellents to Protect Trees and Shrubs From Damage by Rabbits. Technical Bulletins. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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