Galit Shahaf

960 total citations
23 papers, 801 citations indexed

About

Galit Shahaf is a scholar working on Molecular Biology, Surgery and Immunology. According to data from OpenAlex, Galit Shahaf has authored 23 papers receiving a total of 801 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 7 papers in Surgery and 7 papers in Immunology. Recurrent topics in Galit Shahaf's work include Pancreatic function and diabetes (7 papers), Diabetes and associated disorders (5 papers) and Protease and Inhibitor Mechanisms (4 papers). Galit Shahaf is often cited by papers focused on Pancreatic function and diabetes (7 papers), Diabetes and associated disorders (5 papers) and Protease and Inhibitor Mechanisms (4 papers). Galit Shahaf collaborates with scholars based in Israel, United States and Australia. Galit Shahaf's co-authors include Eli C. Lewis, Eyal Ozeri, Mark Mizrahi, Etta Livneh, David E. Ochayon, Danny Jonigk, Kwang‐Won Hong, Martina Dorsch, Tobias Welte and Ravi Mahadeva and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Immunology and PLoS ONE.

In The Last Decade

Galit Shahaf

23 papers receiving 789 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Galit Shahaf Israel 16 304 244 156 136 131 23 801
Haifeng Zhao China 19 358 1.2× 253 1.0× 156 1.0× 172 1.3× 119 0.9× 83 1.0k
Vera Binder Germany 15 382 1.3× 246 1.0× 174 1.1× 112 0.8× 43 0.3× 35 795
Rachel A. Egler United States 14 397 1.3× 101 0.4× 103 0.7× 152 1.1× 211 1.6× 25 907
Sergey Tkachuk Germany 16 260 0.9× 218 0.9× 143 0.9× 93 0.7× 84 0.6× 23 647
Ce Shi China 14 361 1.2× 146 0.6× 160 1.0× 94 0.7× 197 1.5× 51 794
Benjamin Richard France 17 257 0.8× 279 1.1× 95 0.6× 96 0.7× 100 0.8× 26 873
Akihiro Katayama Japan 17 432 1.4× 368 1.5× 177 1.1× 159 1.2× 110 0.8× 19 1.0k
Alexandra Papoudou‐Bai Greece 19 330 1.1× 162 0.7× 168 1.1× 301 2.2× 89 0.7× 84 1.1k
Ingmar Meinecke Germany 9 311 1.0× 124 0.5× 110 0.7× 123 0.9× 130 1.0× 22 737

Countries citing papers authored by Galit Shahaf

Since Specialization
Citations

This map shows the geographic impact of Galit Shahaf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Galit Shahaf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Galit Shahaf more than expected).

Fields of papers citing papers by Galit Shahaf

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Galit Shahaf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Galit Shahaf. The network helps show where Galit Shahaf may publish in the future.

Co-authorship network of co-authors of Galit Shahaf

This figure shows the co-authorship network connecting the top 25 collaborators of Galit Shahaf. A scholar is included among the top collaborators of Galit Shahaf based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Galit Shahaf. Galit Shahaf is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ozeri, Eyal, Peleg Rider, Galit Shahaf, et al.. (2020). Differential signaling patterns of stimulated bone marrow-derived dendritic cells under α1-antitrypsin-enriched conditions. Cellular Immunology. 361. 104281–104281. 4 indexed citations
2.
Ozeri, Eyal, Galit Shahaf, David E. Ochayon, et al.. (2016). Exploration of α1-Antitrypsin Treatment Protocol for Islet Transplantation: Dosing Plan and Route of Administration. Journal of Pharmacology and Experimental Therapeutics. 359(3). 482–490. 11 indexed citations
3.
Salem, Shimrit Yaniv, et al.. (2014). Diminished activity of circulatingα1-antitrypsin is associated with pre-gestational isolated obesity. The Journal of Maternal-Fetal & Neonatal Medicine. 28(5). 500–503. 6 indexed citations
4.
Ochayon, David E., et al.. (2014). Acute Phase Protein α1-Antitrypsin Reduces the Bacterial Burden in Mice by Selective Modulation of Innate Cell Responses. The Journal of Infectious Diseases. 211(9). 1489–1498. 50 indexed citations
5.
Shahaf, Galit, Eyal Sheiner, Shimrit Yaniv Salem, et al.. (2013). Low levels of circulating alpha-1 antitrypsin are associated with spontaneous abortions. The Journal of Maternal-Fetal & Neonatal Medicine. 26(18). 1782–1787. 24 indexed citations
6.
Ochayon, David E., et al.. (2013). Human α1-Antitrypsin Binds to Heat-Shock Protein gp96 and Protects from Endogenous gp96-Mediated Injury In vivo. Frontiers in Immunology. 4. 320–320. 26 indexed citations
7.
8.
Shahaf, Galit, et al.. (2012). PKCη is a negative regulator of AKT inhibiting the IGF-I induced proliferation. Experimental Cell Research. 318(7). 789–799. 10 indexed citations
9.
Sheiner, Eyal, Galit Shahaf, Shimrit Yaniv Salem, et al.. (2012). Lower circulation levels and activity of α-1 Antitrypsin in pregnant women with severe preeclampsia. The Journal of Maternal-Fetal & Neonatal Medicine. 25(12). 2667–2670. 27 indexed citations
10.
Ozeri, Eyal, Mark Mizrahi, Galit Shahaf, & Eli C. Lewis. (2012). α-1 Antitrypsin Promotes Semimature, IL-10–Producing and Readily Migrating Tolerogenic Dendritic Cells. The Journal of Immunology. 189(1). 146–153. 64 indexed citations
11.
Ozeri, Eyal, et al.. (2012). Revascularization of Pancreatic Islet Allografts is Enhanced by α-1-Antitrypsin under Anti-Inflammatory Conditions. Cell Transplantation. 22(11). 2119–2133. 30 indexed citations
12.
13.
Shahaf, Galit, et al.. (2011). α-1-Antitrypsin Gene Delivery Reduces Inflammation, Increases T-Regulatory Cell Population Size and Prevents Islet Allograft Rejection. Molecular Medicine. 17(9-10). 1000–1011. 57 indexed citations
14.
Raveh-Amit, Hadas, et al.. (2011). Protein kinase Cη activates NF-κB in response to camptothecin-induced DNA damage. Biochemical and Biophysical Research Communications. 412(2). 313–317. 15 indexed citations
15.
Baron, Joel, Eyal Sheiner, Galit Shahaf, et al.. (2011). α1-Antitrypsin insufficiency is a possible contributor to preterm premature rupture of membranes. The Journal of Maternal-Fetal & Neonatal Medicine. 25(7). 934–937. 15 indexed citations
16.
Boura‐Halfon, Sigalit, Roi Isaac, Galit Shahaf, et al.. (2010). Elimination of Negative Feedback Control Mechanisms Along the Insulin Signaling Pathway Improves β-Cell Function Under Stress. Diabetes. 59(9). 2188–2197. 15 indexed citations
17.
Ochayon, David E., et al.. (2010). CS1-6 Protection of insulin producing beta cells by the anti-inflammatory acute phase protein alpha-1- antitrypsin. Cytokine. 52(1-2). 6–6. 1 indexed citations
18.
Shahaf, Galit, et al.. (2005). PKCη is localized in the Golgi, ER and nuclear envelope and translocates to the nuclear envelope upon PMA activation and serum-starvation:. Cellular Signalling. 18(8). 1127–1139. 46 indexed citations
19.
Fima, Eyal, et al.. (2001). PKCη enhances cell cycle progression, the expression of G1 cyclins and p21 in MCF-7 cells. Oncogene. 20(46). 6794–6804. 47 indexed citations
20.
Fima, Eyal, et al.. (1999). Expression of PKCeta in NIH-3T3 cells promotes production of the pro-inflammatory cytokine interleukin-6.. PubMed. 10(4). 491–500. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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