Gale A. Granger

6.9k total citations · 1 hit paper
156 papers, 5.4k citations indexed

About

Gale A. Granger is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Gale A. Granger has authored 156 papers receiving a total of 5.4k indexed citations (citations by other indexed papers that have themselves been cited), including 118 papers in Immunology, 38 papers in Molecular Biology and 19 papers in Oncology. Recurrent topics in Gale A. Granger's work include Immune Response and Inflammation (38 papers), Immunotherapy and Immune Responses (32 papers) and Toxin Mechanisms and Immunotoxins (27 papers). Gale A. Granger is often cited by papers focused on Immune Response and Inflammation (38 papers), Immunotherapy and Immune Responses (32 papers) and Toxin Mechanisms and Immunotoxins (27 papers). Gale A. Granger collaborates with scholars based in United States, Italy and France. Gale A. Granger's co-authors include Terry W. Williams, William P. Kolb, Tetsuya Gatanaga, John C. Hiserodt, Robert S. Yamamoto, Russell S. Weiser, William J. Kohr, Rigdon Lentz, Edward W. B. Jeffes and Glenn C. Rice and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Gale A. Granger

153 papers receiving 4.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Molecular cloning and expression of a receptor for human tumor necrosis factor

1990 852 citations Thomas J. Schall, Kerry J. Koller et al. Cell profile →

Peers

Gale A. Granger

IMMUN

ONCOL

RNMI

EPIDE

P Ralph United States

Irene S. Figari United States

J J Oppenheim United States

D J McKean United States

P C Familletti United States

Glenn C. Rice United States

Donald A. Rowley United States

R B Herberman United States

DW Golde United States

Robert Kassel United States

P Ralph United States

Gale A. Granger

2.9k ×0.9

IMMUN

2.0k ×1.4

1.2k ×1.2

ONCOL

603 ×1.1

RNMI

382 ×0.8

EPIDE

Citations per year, relative to Gale A. Granger Gale A. Granger (= 1×) peers P Ralph

Countries citing papers authored by Gale A. Granger

Since Specialization

Citations

This map shows the geographic impact of Gale A. Granger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gale A. Granger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gale A. Granger more than expected).

Fields of papers citing papers by Gale A. Granger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gale A. Granger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gale A. Granger. The network helps show where Gale A. Granger may publish in the future.

Co-authorship network of co-authors of Gale A. Granger

This figure shows the co-authorship network connecting the top 25 collaborators of Gale A. Granger. A scholar is included among the top collaborators of Gale A. Granger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gale A. Granger. Gale A. Granger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Burger, Robert A., Kathleen M. Darcy, Philip J. DiSaia, et al.. (2004). Association between serum levels of soluble tumor necrosis factor receptors/CA 125 and disease progression in patients with epithelial ovarian malignancy. Cancer. 101(1). 106–115. 16 indexed citations

Badgwell, Brian D., Daniel J. Valentino, Edward W. B. Jeffes, et al.. (2003). Intra-arterial administration of TNF-α followed by arterial ablation is an effective therapy for a regionally confined TNF-resistant rat mammary adenocarcinoma. Cancer Immunology Immunotherapy. 52(1). 10–16. 1 indexed citations

Santin, Alessandro D., John C. Hiserodt, Philip J. DiSaia, Sërgio Pecorelli, & Gale A. Granger. (1996). Differential Effects of High-Dose Gamma Irradiation on the Production of Transforming Growth Factor-Beta in Fresh and Established Human Ovarian Cancer. Gynecologic Oncology. 61(3). 403–408. 13 indexed citations

Santin, Alessandro D., John C. Hiserodt, John P. Fruehauf, et al.. (1996). Effects of Irradiation on the Expression of Surface Antigens in Human Ovarian Cancer. Gynecologic Oncology. 60(3). 468–474. 33 indexed citations

Burger, Robert A., Elizabeth A. Grosen, Marc E. Van Eden, et al.. (1994). Host-Tumor Interaction in Ovarian Cancer. Gynecologic Oncology. 55(2). 294–303. 11 indexed citations

Lucci, Joseph A., Alberto Manetta, Federica Cappuccini, et al.. (1993). Immunotherapy of ovarian cancer. II. In vitro generation and characterization of lymphokine-activated killer T cells from the peripheral blood of recurrent ovarian cancer patients. International Journal of Gynecology & Obstetrics. 40(2). 179–180. 1 indexed citations

Jeffes, Edward W. B., Skip Jacques, Bharathi Vayuvegula, et al.. (1993). Therapy of recurrent high grade gliomas with surgery, and autologous mitogen activated IL-2 stimulated killer (MAK) Lymphocytes: I. Enhancement of MAK lytic activity and cytokine production by PHA and clinical use of PHA. Journal of Neuro-Oncology. 15(2). 141–155. 32 indexed citations

Hosszúfalusi, Nóra, et al.. (1993). Quantitative phenotypic and functional analyses of islet immune cells before and after diabetes onset in the BB rat. Diabetologia. 36(11). 1146–1154. 8 indexed citations

Hosszúfalusi, Nóra, et al.. (1992). Quantitative analyses comparing all major spleen cell phenotypes in BB and normal rats: Autoimmune imbalance and double negative T cells associated with resistant, prone and diabetic animals. Journal of Autoimmunity. 5(3). 305–318. 10 indexed citations

10.

Cappuccini, Fabio, et al.. (1991). Enhancement of lymphokine-activated T killer cell tumor necrosis factor receptor mRNA transcription, tumor necrosis factor receptor membrane expression, and tumor necrosis factor/lymphotoxin release by IL-1 beta, IL-4, and IL-6 in vitro. The Journal of Immunology. 146(5). 1522–1526. 17 indexed citations

11.

Yamamoto, Robert S., Bharathi Vayuvegula, Sudhir Gupta, et al.. (1991). Generation of stimulated, lymphokine activated T killer (T-LAK) cells from the peripheral blood of normal donors and adult patients with recurrent glioblastoma. Journal of Immunological Methods. 137(2). 225–235. 12 indexed citations

12.

Gatanaga, Tetsuya, et al.. (1991). The regulation of TNF receptor mRNA synthesis, membrane expression, and release by PMA- and LPS-stimulated human monocytic THP-1 cells in vitro. Cellular Immunology. 138(1). 1–10. 29 indexed citations

13.

Bonavida, Benjamin, et al.. (1990). Tumor necrosis factor : structure, mechanism of action, role in disease and therapy. KARGER eBooks. 26 indexed citations

14.

Schall, Thomas J., Kerry J. Koller, Angela Lee, et al.. (1990). Molecular cloning and expression of a receptor for human tumor necrosis factor. Cell. 61(2). 361–370. 852 indexed citations breakdown →

15.

Knauer, Mary F., et al.. (1990). Mechanism of human lymphotoxin and tumor necrosis factor induced destruction of cells in vitro: Phospholipase activation and deacylation of specific‐membrane phospholipids. Journal of Cellular Physiology. 142(3). 469–479. 34 indexed citations

16.

Sung, S S, Lawrence Jung, Jay A. Walters, et al.. (1989). Production of lymphotoxin by isolated human tonsillar B lymphocytes and B lymphocyte cell lines.. Journal of Clinical Investigation. 84(1). 236–243. 25 indexed citations

17.

Jeffes, Edward W. B., et al.. (1989). The presence of antibodies to lymphotoxin and tumor necrosis factor in normal serum. Arthritis & Rheumatism. 32(9). 1148–1152. 15 indexed citations

18.

Oppenheim, Joost J., R. Michael Blaese, John E. Horton, Daniel E. Thor, & Gale A. Granger. (1973). Production of macrophage migration inhibitory factor and lymphotoxin by leukocytes from normal and Wiskott-Aldrich syndrome patients. Cellular Immunology. 8(1). 62–70. 9 indexed citations

19.

Granger, Gale A. & William P. Kolb. (1968). Ylmphocyte in Vitro Cytotoxicity: Mechanisms of Immune and Non-Immune Small Lymphocyte Mediated Target L Cell Destruction. The Journal of Immunology. 101(1). 111–120. 161 indexed citations

20.

Granger, Gale A., et al.. (1966). The role of cytophilic antibody in immune macrophage-target-cell interaction. Abstr.. The Mouseion at the JAXlibrary (Jackson Laboratory). 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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