G J van Ommen
About
G J van Ommen is a scholar working on Molecular Biology, Genetics and Genetics.
According to data from OpenAlex, G J van Ommen has authored 38 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 16 papers in Genetics and 11 papers in Genetics. Recurrent topics in G J van Ommen's work include Muscle Physiology and Disorders (15 papers), Neurogenetic and Muscular Disorders Research (12 papers) and RNA modifications and cancer (7 papers). G J van Ommen is often cited by papers focused on Muscle Physiology and Disorders (15 papers), Neurogenetic and Muscular Disorders Research (12 papers) and RNA modifications and cancer (7 papers). G J van Ommen collaborates with scholars based in Netherlands, United Kingdom and United States. G J van Ommen's co-authors include Johan T. den Dunnen, Egbert Bakker, P.M. Grootscholten, Christine Van Broeckhoven, P. Pearson, Martin C. Wapenaar, H.B. Ginjaar, Lau A.J. Blonden, Daphne de Jong and Silvère M. van der Maarel and has published in prestigious journals such as Science, Nucleic Acids Research and Journal of Clinical Investigation.
In The Last Decade
G J van Ommen
38 papers receiving 2.4k citations
Hit Papers
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| G J van Ommen Netherlands | 20 | 1.8k | 632 | 436 | 361 | 352 | 38 | 2.4k | ||
| Monique Losekoot Netherlands | 34 | 1.4k 0.8× | 483 0.8× | 1.3k 2.9× | 96 0.3× | 283 0.8× | 125 | 3.1k | ||
| Ivo F.A.C. Fokkema Netherlands | 12 | 1.6k 0.9× | 300 0.5× | 719 1.6× | 307 0.9× | 141 0.4× | 18 | 2.1k | ||
| Anna Sárközy United Kingdom | 30 | 2.4k 1.3× | 192 0.3× | 449 1.0× | 503 1.4× | 165 0.5× | 91 | 2.9k | ||
| Gwenaëlle Collod‐Béroud France | 21 | 1.7k 1.0× | 195 0.3× | 1.6k 3.7× | 487 1.3× | 220 0.6× | 38 | 3.6k | ||
| Geneviève Piétu France | 26 | 839 0.5× | 219 0.3× | 323 0.7× | 146 0.4× | 147 0.4× | 69 | 2.0k | ||
| Jacopo Celli Netherlands | 13 | 1.6k 0.9× | 177 0.3× | 816 1.9× | 123 0.3× | 155 0.4× | 16 | 2.5k | ||
| Tetsuya Niihori Japan | 27 | 1.8k 1.0× | 199 0.3× | 495 1.1× | 164 0.5× | 126 0.4× | 78 | 2.9k | ||
| Yakov Fellig Israel | 23 | 934 0.5× | 315 0.5× | 155 0.4× | 115 0.3× | 105 0.3× | 78 | 1.8k | ||
| Ralf Herrmann Germany | 23 | 1.6k 0.9× | 170 0.3× | 171 0.4× | 435 1.2× | 304 0.9× | 48 | 2.0k | ||
| Mary Shago Canada | 21 | 1.4k 0.8× | 224 0.4× | 613 1.4× | 73 0.2× | 132 0.4× | 57 | 2.3k |
Countries citing papers authored by G J van Ommen
Since
Specialization
Citations
This map shows the geographic impact of G J van Ommen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G J van Ommen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G J van Ommen more than expected).
Fields of papers citing papers by G J van Ommen
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by G J van Ommen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G J van Ommen. The network helps show where G J van Ommen may publish in the future.
Co-authorship network of co-authors of G J van Ommen
This figure shows the co-authorship network connecting the top 25 collaborators of G J van Ommen. A scholar is included among the top collaborators of G J van Ommen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G J van Ommen. G J van Ommen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Hansson, Mats, G J van Ommen, Ruth Chadwick, & Joakim Dillner. (2013). Patients would benefit from simplified ethical review and consent procedure. The Lancet Oncology. 14(6). 451–453.
2.
Lemmers, Richard J.L.F., Patrick J. van der Vliet, Rinse Klooster, et al.. (2010). A Unifying Genetic Model for Facioscapulohumeral Muscular Dystrophy. Science. 329(5999). 1650–1653.
3.
Hoen, Peter A.C. ’t, Michael Hirsch, Emile J. de Meijer, et al.. (2010). mRNA degradation controls differentiation state-dependent differences in transcript and splice variant abundance. Nucleic Acids Research. 39(2). 556–566.
4.
Mercuri, Eugenio, Anna Mayhew, Francesco Muntoni, et al.. (2008). Towards harmonisation of outcome measures for DMD and SMA within TREAT-NMD; Report of three expert workshops: TREAT-NMD/ENMC Workshop on outcome measures, 12th–13th May 2007, Naarden, The Netherlands; TREAT-NMD Workshop on outcome measures in experimental trials for DMD, 30th June–1st July 2007, Naarden, The Netherlands; Conjoint Institute of Myology TREAT-NMD Meeting on physical activity monitoring in neuromuscular disorders, 11th July 2007, Paris, France. Neuromuscular Disorders. 18(11). 894–903.
5.
Heemskerk, Hans, Begoña Aguilera, Annika Janson, et al.. (2008). Muscle binding peptides found by phage display as delivery agent for antisense oligonucleotides. Journal of Controlled Release. 132(3). e3–e5.
6.
Yuille, Martin, G J van Ommen, Christian Bréchot, et al.. (2007). Biobanking for Europe. Briefings in Bioinformatics. 9(1). 14–24.
7.
Roest, Pauline A.M., Ieke B. Ginjaar, Egbert Bakker, et al.. (1996). Splicing mutations in DMD/BMD detected by RT-PCR/PTT: detection of a 19AA insertion in the cysteine rich domain of dystrophin compatible with BMD.. Journal of Medical Genetics. 33(11). 935–939.
8.
Grootscholten, P.M., J. M. Cobben, Stefania Zappata, et al.. (1996). Apparent gene conversions involving the SMN gene in the region of the spinal muscular atrophy locus on chromosome 5.. PubMed. 59(4). 834–8.
9.
Grootscholten, P.M., et al.. (1995). A Provisional Transcript Map ofthe Spinal Muscular Atrophy(SMA) Critical Region. European Journal of Human Genetics. 3(2). 87–95.
10.
Maarel, Silvère M. van der, J A Maat-Kievit, I Huber, et al.. (1995). Yeast Artificial Chromosome Cloningof the Xq13.3-q21.31 Region and FineMapping of a Deletion Associated withChoroideremia and NonspecificMental Retardation. European Journal of Human Genetics. 3(4). 207–218.
11.
Vernole, Patrizia, Daniela Caporossi, Bruna Tedeschi, B. Nicoletti, & G J van Ommen. (1995). Subregional localization of 14 yeast artificial chromosomes to human chromosome region 1p by fluorescence in situ hybridization. Cytogenetic and Genome Research. 70(1-2). 23–25.
12.
Heutink, Peter, Evert M. van Schothorst, A. Bardoel, et al.. (1994). Further Localization of the Genefor Hereditary Paragangliomasand Evidence for Linkage inUnrelated Families. European Journal of Human Genetics. 2(3). 148–158.
13.
Nicholson, L. V. B., Kate Bushby, Mark A. Johnson, et al.. (1992). Predicted and observed sizes of dystrophin in some patients with gene deletions that disrupt the open reading frame.. Journal of Medical Genetics. 29(12). 892–896.
14.
Wapenaar, Martin C., et al.. (1991). Deletions within the pseudoautosomal region help map three new markers and indicate a possible role of this region in linear growth.. PubMed. 49(4). 811–9.
15.
Visser, Marjolein, Egbert Bakker, Joep C. Defesche, P. A. Bolhuis, & G J van Ommen. (1990). An unusual variant of Becker muscular dystrophy. Annals of Neurology. 27(5). 578–581.
16.
McCabe, Edward R.B., Jeffrey A. Towbin, Jeffrey S. Chamberlain, et al.. (1989). Complementary DNA probes for the Duchenne muscular dystrophy locus demonstrate a previously undetectable deletion in a patient with dystrophic myopathy, glycerol kinase deficiency, and congenital adrenal hypoplasia.. Journal of Clinical Investigation. 83(1). 95–99.
17.
Jong, Daphne de, et al.. (1989). Translocation t(14;18) in B cell lymphomas as a cause for defective immunoglobulin production.. The Journal of Experimental Medicine. 169(3). 613–624.
18.
Dunnen, Johan T. den, P.M. Grootscholten, Egbert Bakker, et al.. (1989). Topography of the Duchenne muscular dystrophy (DMD) gene: FIGE and cDNA analysis of 194 cases reveals 115 deletions and 13 duplications.. PubMed. 45(6). 835–47.
19.
Chelly, Jamel, F. Marlhens, Bernard Dutrillaux, et al.. (1988). Deletion proximal to DXS68 locus (L1 probe site) in a boy with Duchenne muscular dystrophy, glycerol kinase deficiency, and adrenal hypoplasia. Human Genetics. 78(3). 222–227.
20.
Boyd, Yvonne, D.J. Cockburn, Scott M. Holt, et al.. (1988). Mapping of 12 translocation breakpoints in the Xp21 region with respect to the locus for Duchenne muscular dystrophy. Cytogenetic and Genome Research. 48(1). 28–34.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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