G. G. Brownlee

716 total citations
14 papers, 585 citations indexed

About

G. G. Brownlee is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, G. G. Brownlee has authored 14 papers receiving a total of 585 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 9 papers in Hematology and 5 papers in Genetics. Recurrent topics in G. G. Brownlee's work include Hemophilia Treatment and Research (9 papers), Virus-based gene therapy research (5 papers) and CRISPR and Genetic Engineering (3 papers). G. G. Brownlee is often cited by papers focused on Hemophilia Treatment and Research (9 papers), Virus-based gene therapy research (5 papers) and CRISPR and Genetic Engineering (3 papers). G. G. Brownlee collaborates with scholars based in United Kingdom, United States and Japan. G. G. Brownlee's co-authors include Peter R. Winship, Iain D. Campbell, Penny A. Handford, Mark Mayhew, Maayan Baron, Ervin Fodor, Baik Lin Seong, F. Giannelli, D. E. G. Austen and D Lillicrap and has published in prestigious journals such as Nature, Nucleic Acids Research and Journal of Cell Science.

In The Last Decade

G. G. Brownlee

14 papers receiving 574 citations

Peers

G. G. Brownlee
C. Onnekink Netherlands
Dennis K. Watson United States
Randall K. Walker United States
Uwe Schlokat Austria
Jutta Friel Germany
Jan-Olov Kvassman United States
Diane Giannola United States
Denise Petersen United States
Nancy E. Seidel United States
Nabeel R. Yaseen United States
C. Onnekink Netherlands
G. G. Brownlee
Citations per year, relative to G. G. Brownlee G. G. Brownlee (= 1×) peers C. Onnekink

Countries citing papers authored by G. G. Brownlee

Since Specialization
Citations

This map shows the geographic impact of G. G. Brownlee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. G. Brownlee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. G. Brownlee more than expected).

Fields of papers citing papers by G. G. Brownlee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. G. Brownlee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. G. Brownlee. The network helps show where G. G. Brownlee may publish in the future.

Co-authorship network of co-authors of G. G. Brownlee

This figure shows the co-authorship network connecting the top 25 collaborators of G. G. Brownlee. A scholar is included among the top collaborators of G. G. Brownlee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. G. Brownlee. G. G. Brownlee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Brownlee, G. G., et al.. (1998). Differentiation-specific enhancer activity in transduced keratinocytes: a model for epidermal gene therapy. Gene Therapy. 5(3). 394–402. 21 indexed citations
2.
Giannelli, F., S. S. Sommer, Man‐Chiu Poon, et al.. (1997). Haemophilia B: database of point mutations and short additions and deletions, 7th edition. Nucleic Acids Research. 25(1). 133–135. 23 indexed citations
3.
Davies, Neil H., D. E. G. Austen, Margaret Wilde, G.J. Darlington, & G. G. Brownlee. (1997). Clotting factor IX levels in C/EBPα knockout mice. British Journal of Haematology. 99(3). 578–579. 9 indexed citations
4.
Austen, D. E. G., et al.. (1996). Recombinant factor IX secreted by transduced human keratinocytes is biologically active. British Journal of Haematology. 95(3). 561–563. 9 indexed citations
5.
Winship, Peter R., et al.. (1995). The high frequency of the — 6G → A factor IX promoter mutation is the result both of a founder effect and recurrent mutation at a CpG dinucleotide. British Journal of Haematology. 89(3). 672–674. 13 indexed citations
6.
Brownlee, G. G.. (1995). Prospects for gene therapy of haemophilia A and B. British Medical Bulletin. 51(1). 91–105. 13 indexed citations
7.
Giannelli, F., et al.. (1994). Haemophilia B: database of point mutations and short additions and deletions, fifth edition, 1994. Nucleic Acids Research. 22(17). 3534–3546. 38 indexed citations
8.
Giannelli, F., Katherine A. High, M.‐C. Poon, et al.. (1993). Haemophilia B: database of point mutations and short additions and deletions—fourth edition, 1993. Nucleic Acids Research. 21(13). 3075–3087. 35 indexed citations
9.
Fodor, Ervin, Baik Lin Seong, & G. G. Brownlee. (1993). Photochemical cross-linking of influenza A polymerase to its virion RNA promoter defines a polymerase binding site at residues 9 to 12 of the promoter. Journal of General Virology. 74(7). 1327–1333. 81 indexed citations
10.
Giannelli, F., Katherine A. High, D Lillicrap, et al.. (1992). Haemophilia B: database of point mutations and short additions and deletions--third edition, 1992. Nucleic Acids Research. 20(suppl). 2027–2063. 33 indexed citations
11.
Handford, Penny A., Mark Mayhew, Maayan Baron, et al.. (1991). Key residues involved in calcium-binding motifs in EGF-like domains. Nature. 351(6322). 164–167. 244 indexed citations
12.
Koch, Katherine S., et al.. (1991). Retroviral vector infection and transplantation in rats of primary fetal rat hepatocytes. Journal of Cell Science. 99(1). 121–130. 10 indexed citations
13.
Brownlee, G. G.. (1987). The molecular pathology of haemophilia B. Biochemical Society Transactions. 15(1). 1–8. 32 indexed citations
14.
Anson, D S, Randy A. Hock, D. E. G. Austen, et al.. (1987). Towards gene therapy for hemophilia B.. PubMed. 4(1). 11–20. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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