G. E. Milo

1.2k total citations
35 papers, 956 citations indexed

About

G. E. Milo is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, G. E. Milo has authored 35 papers receiving a total of 956 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 12 papers in Oncology and 9 papers in Cancer Research. Recurrent topics in G. E. Milo's work include Cancer-related Molecular Pathways (7 papers), DNA Repair Mechanisms (4 papers) and Carcinogens and Genotoxicity Assessment (4 papers). G. E. Milo is often cited by papers focused on Cancer-related Molecular Pathways (7 papers), DNA Repair Mechanisms (4 papers) and Carcinogens and Genotoxicity Assessment (4 papers). G. E. Milo collaborates with scholars based in United States, United Kingdom and Russia. G. E. Milo's co-authors include David G. Cornwell, Victor C. Gavino, James S. Miller, B. I. Sahai Srivastava, Naho Morisaki, Rao V. Panganamala, Howard Sprecher, David L. Crowe, William B. Malarkey and C. C. Capen and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

G. E. Milo

34 papers receiving 890 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
G. E. Milo United States	16	402	193	173	141	127	35	956
Yan Gu United States	17	521 1.3×	157 0.8×	90 0.5×	226 1.6×	107 0.8×	23	1.2k
Rudolf I. Salganik Russia	15	612 1.5×	133 0.7×	43 0.2×	75 0.5×	115 0.9×	24	1.1k
Per Ålin Sweden	15	1.8k 4.4×	158 0.8×	154 0.9×	149 1.1×	87 0.7×	23	2.1k
M. Waheed Roomi Canada	22	636 1.6×	145 0.8×	102 0.6×	55 0.4×	277 2.2×	83	1.5k
Manjit K. Saini United States	13	818 2.0×	106 0.5×	122 0.7×	72 0.5×	85 0.7×	16	1.1k
Marie A. Amoruso United States	15	334 0.8×	125 0.6×	41 0.2×	83 0.6×	175 1.4×	26	858
Yasuo Natori Japan	18	516 1.3×	116 0.6×	91 0.5×	73 0.5×	36 0.3×	77	1.1k
Violet Daniel Israel	18	1.2k 2.9×	78 0.4×	111 0.6×	97 0.7×	126 1.0×	38	1.6k
Joan A. Higgins United Kingdom	25	721 1.8×	147 0.8×	307 1.8×	40 0.3×	87 0.7×	67	1.4k
Lorenzo Ferri Italy	14	718 1.8×	247 1.3×	100 0.6×	198 1.4×	183 1.4×	36	1.3k

Countries citing papers authored by G. E. Milo

Since Specialization

Citations

This map shows the geographic impact of G. E. Milo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. E. Milo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. E. Milo more than expected).

Fields of papers citing papers by G. E. Milo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. E. Milo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. E. Milo. The network helps show where G. E. Milo may publish in the future.

Co-authorship network of co-authors of G. E. Milo

This figure shows the co-authorship network connecting the top 25 collaborators of G. E. Milo. A scholar is included among the top collaborators of G. E. Milo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. E. Milo. G. E. Milo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Lee, Hakho, T. Prior, B. C. Casto, et al.. (1997). Ineffectiveness of the presence of H-ras/p53 combination of mutations in squamous cell carcinoma cells to induce a conversion of a nontumorigenic to a tumorigenic phenotype. Cell Biology and Toxicology. 13(6). 419–434. 9 indexed citations

Wani, Gulzar, et al.. (1997). Expression of Molecular Biomarkers in Primary Breast Tumors Implanted into a Surrogate Host: Increased Levels of Cyclins Correlate with Tumor Progression. Molecular Medicine. 3(4). 273–283. 1 indexed citations

Milo, G. E., Duo Li, B. C. Casto, et al.. (1996). Malignant conversion of chemically transformed normal human cells.. Proceedings of the National Academy of Sciences. 93(11). 5229–5234. 5 indexed citations

Milo, G. E., et al.. (1995). A conundrum in molecular toxicology: Molecular and biological changes during neoplastic transformation of human cells. Cell Biology and Toxicology. 11(6). 329–345. 5 indexed citations

Li, Duo, et al.. (1995). Cloning and sequencing of CATR1.3, a human gene associated with tumorigenic conversion.. Proceedings of the National Academy of Sciences. 92(14). 6409–6413. 7 indexed citations

Nesnow, Stephen, et al.. (1990). Quantitative evaluation of the effects of human carcinogens and related chemicals on human foreskin fibroblasts. Cell Biology and Toxicology. 6(2). 171–84. 7 indexed citations

Milo, G. E., et al.. (1990). Nontumorigenic squamous cell carcinoma line converted to tumorigenicity with methyl methanesulfonate without activation of HRAS or MYC.. Proceedings of the National Academy of Sciences. 87(4). 1268–1272. 19 indexed citations

Milo, G. E., et al.. (1988). Cocarcinogenicity of saccharin andn‐alkylnitrosoureas in cultured human diploid fibroblasts. Journal of Toxicology and Environmental Health. 24(3). 413–421. 6 indexed citations

Jeffrey, A.M., et al.. (1985). Preferential binding of benzo[a]pyrene diol epoxide to the linker DNA of human foreskin fibroblasts in S phase in the presence of benzamide.. Proceedings of the National Academy of Sciences. 82(9). 2769–2773. 10 indexed citations

10.

Milo, G. E., et al.. (1982). Multiparametric evaluation of the toxic responses of normal human cells treatedin vitrowith different classes of environmental toxicants. Journal of Toxicology and Environmental Health. 10(1). 143–156. 3 indexed citations

11.

Morisaki, Naho, et al.. (1982). Dipyridamole: an antioxidant that promotes the proliferation of aorta smooth muscle cells.. PubMed. 11(2). 88–107. 23 indexed citations

12.

Gavino, Victor C., et al.. (1981). Effect of polyunsaturated fatty acids and antioxidants on lipid peroxidation in tissue cultures.. Journal of Lipid Research. 22(5). 763–769. 198 indexed citations

13.

Milo, G. E., et al.. (1981). Ultraviolet radiation-induced neoplastic transformation of normal human cells, in vitro. Chemico-Biological Interactions. 36(1). 45–59. 10 indexed citations

14.

Milo, G. E., et al.. (1980). The toxicological evaluation of the mycotoxins T-2 and T-2 tetraol using normal human fibroblasts in vitro. Toxicology and Applied Pharmacology. 52(1). 159–168. 58 indexed citations

15.

Matthews, Richard H., et al.. (1977). Effects of cysteine upon tumor cells.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 2(2). 65–9. 1 indexed citations

16.

Malarkey, William B., et al.. (1977). Defective dopaminergic regulation of prolactin secretion in a rat pituitary tumour cell line. Nature. 266(5603). 640–641. 35 indexed citations

17.

Milo, G. E. & B. I. Sahai Srivastava. (1969). Effect of cytokinins on tobacco mosaic virus production in local-lesion and systemic hosts. Virology. 38(1). 26–31. 23 indexed citations

18.

Milo, G. E. & B. I. Sahai Srivastava. (1969). RNA-DNA hybridization studies with the crown gall bacteria and the tobacco tumor tissue. Biochemical and Biophysical Research Communications. 34(2). 196–199. 34 indexed citations

19.

Milo, G. E. & B. I. Sahai Srivastava. (1969). Effect of cytokinins on tobacco mosaic virus production in tobacco pith tissue cultures. Virology. 39(3). 621–623. 10 indexed citations

20.

Milo, G. E., et al.. (1967). Changes in the ascorbate concentration of pinto bean leaves accompanying the formation of TMV-induced local lesions. Virology. 31(2). 197–206. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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