Fumitaka Katoh

842 total citations
19 papers, 713 citations indexed

About

Fumitaka Katoh is a scholar working on Molecular Biology, Oncology and Organic Chemistry. According to data from OpenAlex, Fumitaka Katoh has authored 19 papers receiving a total of 713 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 4 papers in Oncology and 2 papers in Organic Chemistry. Recurrent topics in Fumitaka Katoh's work include Connexins and lens biology (6 papers), Pluripotent Stem Cells Research (2 papers) and Cancer Cells and Metastasis (2 papers). Fumitaka Katoh is often cited by papers focused on Connexins and lens biology (6 papers), Pluripotent Stem Cells Research (2 papers) and Cancer Cells and Metastasis (2 papers). Fumitaka Katoh collaborates with scholars based in France, Japan and Italy. Fumitaka Katoh's co-authors include Hiroshi Yamasaki, Hiroshi Nojima, Hideo Asada, Hiroyoshi Hidaka, Kunihiko Yoshikawa, Morihito Okada, Sakan Maeda, Tatsuki R. Kataoka, Noriaki Tsubota and Hiroshi Yamasaki and has published in prestigious journals such as Journal of Clinical Investigation, Cancer and Journal of Clinical Microbiology.

In The Last Decade

Fumitaka Katoh

19 papers receiving 702 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Fumitaka Katoh France 14 524 96 83 77 76 19 713
Silke Erdmann Germany 14 384 0.7× 112 1.2× 57 0.7× 45 0.6× 39 0.5× 15 609
Maı̈na Lepourcelet United States 8 762 1.5× 204 2.1× 25 0.3× 52 0.7× 66 0.9× 8 910
Chiara Soncini Italy 8 352 0.7× 139 1.4× 45 0.5× 160 2.1× 39 0.5× 9 580
Priyam Banerjee United States 14 358 0.7× 121 1.3× 31 0.4× 113 1.5× 137 1.8× 35 616
Scott W. Blume United States 13 617 1.2× 116 1.2× 28 0.3× 44 0.6× 125 1.6× 22 761
Kyoichiro Higashi Japan 11 399 0.8× 110 1.1× 27 0.3× 64 0.8× 63 0.8× 15 628
Beatrice N. Engelsberg United States 10 323 0.6× 143 1.5× 26 0.3× 29 0.4× 81 1.1× 12 490
Hiroyuki Kouji Japan 12 331 0.6× 95 1.0× 30 0.4× 30 0.4× 47 0.6× 29 461
N. Martel France 15 505 1.0× 227 2.4× 16 0.2× 53 0.7× 144 1.9× 30 838
Gaoyang Zhu China 9 323 0.6× 115 1.2× 40 0.5× 31 0.4× 95 1.3× 14 486

Countries citing papers authored by Fumitaka Katoh

Since Specialization
Citations

This map shows the geographic impact of Fumitaka Katoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fumitaka Katoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fumitaka Katoh more than expected).

Fields of papers citing papers by Fumitaka Katoh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fumitaka Katoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fumitaka Katoh. The network helps show where Fumitaka Katoh may publish in the future.

Co-authorship network of co-authors of Fumitaka Katoh

This figure shows the co-authorship network connecting the top 25 collaborators of Fumitaka Katoh. A scholar is included among the top collaborators of Fumitaka Katoh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fumitaka Katoh. Fumitaka Katoh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Morimoto, Tatsuya, et al.. (2009). Synthesis and biological evaluation of novel phthalazinone derivatives as topically active phosphodiesterase 4 inhibitors. Bioorganic & Medicinal Chemistry. 17(19). 6959–6970. 33 indexed citations
2.
Katoh, Fumitaka, et al.. (2007). Inhibition of Balb/c 3T3 cell transformation by synthetic acyclic retinoid NIK-333; possible involvement of enhanced gap junctional intercellular communication. Cancer Detection and Prevention. 31(4). 332–338. 8 indexed citations
3.
Asada, Hideo, Hironori Niizeki, Fumitaka Katoh, et al.. (2007). Role for connexin 26 in metastasis of human malignant melanoma. Cancer. 110(5). 1162–1172. 44 indexed citations
4.
Katoh, Fumitaka, et al.. (2004). Dependence of chemotherapy response on p53 mutation status in a panel of human cancer lines maintained in nude mice. Cancer Science. 95(6). 541–546. 17 indexed citations
5.
6.
Tanaka, Hideki, et al.. (2003). HMN-176, an active metabolite of the synthetic antitumor agent HMN-214, restores chemosensitivity to multidrug-resistant cells by targeting the transcription factor NF-Y.. PubMed. 63(20). 6942–7. 59 indexed citations
7.
Iwai, Masaki, Yoshinori Harada, Akira Muramatsu, et al.. (2000). Development of gap junctional channels and intercellular communication in rat liver during ontogenesis. Journal of Hepatology. 32(1). 11–18. 76 indexed citations
8.
Katoh, Fumitaka, Tatsuki R. Kataoka, Morihito Okada, et al.. (2000). A role for heterologous gap junctions between melanoma and endothelial cells in metastasis. Journal of Clinical Investigation. 105(9). 1189–1197. 157 indexed citations
9.
Tanahashi, Toshihito, Masakazu Kita, Tadashi Kodama, et al.. (2000). Comparison of PCR-Restriction Fragment Length Polymorphism Analysis and PCR-Direct Sequencing Methods for Differentiating Helicobacter pylori ureB Gene Variants. Journal of Clinical Microbiology. 38(1). 165–169. 21 indexed citations
10.
Tsuchiya, Toshie, Kiyoshi Fukuhara, Yoshiaki Ikarashi, et al.. (1995). Studies on the tumor‐promoting activity of additives in biomaterials: Inhibition of metabolic cooperation by phenolic antioxidants involved in rubber materials. Journal of Biomedical Materials Research. 29(1). 121–126. 10 indexed citations
11.
12.
Katoh, Fumitaka & Hiroshi Yamasaki. (1991). Regulation of gap-junctional intercellular comnunication in transformation-sensitive and transformation-resistant BALB/c 3T3 cell variants. Carcinogenesis. 12(10). 1923–1926. 7 indexed citations
13.
Katoh, Fumitaka, et al.. (1990). Okadaic Acid and Phorbol Esters: Comparative Effects of These Tumor Promoters on Cell Transformation, Intercellular Communication and Differentiation in vitro. Japanese Journal of Cancer Research. 81(6-7). 590–597. 39 indexed citations
14.
Masui, Tsuneo, Shoji Fukushima, Fumitaka Katoh, Hiroshi Yamasaki, & Nobuyuki Ito. (1988). Effects of sodium L-ascorbate, uracil, butylated hydroxyanisole and extracellular pH on junctional intercellular communication of BALB /c 3T3 cells. Carcinogenesis. 9(7). 1143–1146. 11 indexed citations
15.
Bignami, Margherita, Silvia De Rosa, Germana Falcone, et al.. (1988). Specific viral oncogenes cause differential effects on cell‐to‐cell communication, relevant to the suppression of the transformed phenotype by normal cells. Molecular Carcinogenesis. 1(1). 67–75. 49 indexed citations
16.
Katoh, Fumitaka, et al.. (1988). Novel method for selective killing of transformed rodent cells through intercellular communication, with possible therapeutic applications.. PubMed. 48(11). 3203–7. 24 indexed citations
17.
Yamasaki, Hiroshi, et al.. (1988). Role of intercellular communication in the control of critical gene expression during multistage carcinogenesis.. PubMed. 57–75. 13 indexed citations
18.
Katoh, Fumitaka, et al.. (1988). Transforming growth factor beta as a potent promoter in two-stage BALB/c 3T3 cell transformation.. PubMed. 48(10). 2832–6. 32 indexed citations
19.
Yamasaki, Hiroshi & Fumitaka Katoh. (1988). Further evidence for the involvement of gap-junctional intercellular communication in induction and maintenance of transformed foci in BALB/c 3T3 cells.. PubMed. 48(12). 3490–5. 59 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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