Fumihiro Sugiyama

9.8k total citations · 2 hit papers
156 papers, 6.9k citations indexed

About

Fumihiro Sugiyama is a scholar working on Molecular Biology, Genetics and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Fumihiro Sugiyama has authored 156 papers receiving a total of 6.9k indexed citations (citations by other indexed papers that have themselves been cited), including 81 papers in Molecular Biology, 49 papers in Genetics and 27 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Fumihiro Sugiyama's work include CRISPR and Genetic Engineering (25 papers), Renin-Angiotensin System Studies (22 papers) and Animal Genetics and Reproduction (21 papers). Fumihiro Sugiyama is often cited by papers focused on CRISPR and Genetic Engineering (25 papers), Renin-Angiotensin System Studies (22 papers) and Animal Genetics and Reproduction (21 papers). Fumihiro Sugiyama collaborates with scholars based in Japan, United States and Egypt. Fumihiro Sugiyama's co-authors include Ken-ichi Yagami, Akiyoshi Fukamizu, Katsutoshi Goto, Masashi Yanagisawa, Carsten T. Beuckmann, Takeshi Sakurai, Jon T. Willie, Junko Hara, Ken‐ichi Yagami and Satoru Takahashi and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Fumihiro Sugiyama

151 papers receiving 6.7k citations

Hit Papers

2 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Genetic Ablation of Orexin Neurons in Mice Results in Narcolepsy, Hypophagia, and Obesity

2001 1.1k citations Fumihiro Sugiyama, Katsutoshi Goto et al. profile →
Hypothalamic Orexin Neurons Regulate Arousal According to Energy Balance in Mice

2003 715 citations Fumihiro Sugiyama, Katsutoshi Goto et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Fumihiro Sugiyama Japan	35	2.3k	1.7k	1.6k	1.2k	988	156	6.9k
S. Clay Williams United States	25	3.0k 1.3×	3.5k 2.0×	3.2k 2.0×	646 0.5×	2.0k 2.1×	27	8.3k
Gastone G. Nussdorfer Italy	48	2.8k 1.3×	1.7k 1.0×	628 0.4×	818 0.7×	367 0.4×	398	9.1k
Shuji Hinuma Japan	42	2.9k 1.3×	1.7k 1.0×	316 0.2×	717 0.6×	183 0.2×	89	10.1k
Kaare M. Gautvik Norway	35	2.2k 1.0×	3.0k 1.8×	3.1k 1.9×	96 0.1×	1.9k 1.9×	192	7.6k
Holger M. Reichardt Germany	52	2.5k 1.1×	1.6k 0.9×	283 0.2×	120 0.1×	138 0.1×	135	10.1k
James A. Waschek United States	44	2.4k 1.1×	1.6k 0.9×	488 0.3×	201 0.2×	84 0.1×	166	7.0k
Tomás García‐Caballero Spain	36	1.2k 0.5×	1.2k 0.7×	203 0.1×	297 0.2×	122 0.1×	145	4.4k
Wolfgang Berger Switzerland	46	4.5k 2.0×	413 0.2×	591 0.4×	320 0.3×	476 0.5×	172	7.0k
Marc Laburthe France	59	6.3k 2.8×	1.2k 0.7×	533 0.3×	125 0.1×	232 0.2×	218	10.9k
Annie M. Curtis Ireland	28	1.3k 0.6×	2.5k 1.5×	214 0.1×	231 0.2×	366 0.4×	52	4.9k

Countries citing papers authored by Fumihiro Sugiyama

Since Specialization

Citations

This map shows the geographic impact of Fumihiro Sugiyama's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fumihiro Sugiyama with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fumihiro Sugiyama more than expected).

Fields of papers citing papers by Fumihiro Sugiyama

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fumihiro Sugiyama. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fumihiro Sugiyama. The network helps show where Fumihiro Sugiyama may publish in the future.

Co-authorship network of co-authors of Fumihiro Sugiyama

This figure shows the co-authorship network connecting the top 25 collaborators of Fumihiro Sugiyama. A scholar is included among the top collaborators of Fumihiro Sugiyama based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fumihiro Sugiyama. Fumihiro Sugiyama is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

1.

Kuno, Akihiro, Yoko Daitoku, Yoko Tanimoto, et al.. (2024). Regional random mutagenesis driven by multiple sgRNAs and diverse on-target genome editing events to identify functionally important elements in non-coding regions. Open Biology. 14(4). 240007–240007. 2 indexed citations

2.

Fujita, Ryo, Seiya Mizuno, Taketaro Sadahiro, et al.. (2023). Generation of a MyoD knock-in reporter mouse line to study muscle stem cell dynamics and heterogeneity. iScience. 26(5). 106592–106592. 8 indexed citations

3.

Ogonuki, Narumi, Hirohisa Kyogoku, Toshiaki Hino, et al.. (2022). Birth of mice from meiotically arrested spermatocytes following biparental meiosis in halved oocytes. EMBO Reports. 23(7). e54992–e54992. 3 indexed citations

4.

Kasai, Shuya, Yota Tatara, Hiromi Yamazaki, et al.. (2022). Inducible Systemic Gcn1 Deletion in Mice Leads to Transient Body Weight Loss upon Tamoxifen Treatment Associated with Decrease of Fat and Liver Glycogen Storage. International Journal of Molecular Sciences. 23(6). 3201–3201. 4 indexed citations

5.

Murata, Kazuya, Yuko Hamada, Kanako Kato, et al.. (2021). Efficient induction of proximity-dependent labelling by biotin feeding in BMAL1-BioID knock-in mice. The Journal of Biochemistry. 170(4). 453–461. 10 indexed citations

6.

Sakamoto, Kentarou, Misao Akishiba, Takahiro Iwata, et al.. (2020). Optimizing Charge Switching in Membrane Lytic Peptides for Endosomal Release of Biomacromolecules. Angewandte Chemie International Edition. 59(45). 19990–19998. 44 indexed citations

7.

Matsuda, Shuichi, Jun‐Dal Kim, Fumihiro Sugiyama, et al.. (2020). Transcriptomic Evaluation of Pulmonary Fibrosis-Related Genes: Utilization of Transgenic Mice with Modifying p38 Signal in the Lungs. International Journal of Molecular Sciences. 21(18). 6746–6746. 9 indexed citations

8.

Sakamoto, Kentarou, Misao Akishiba, Takahiro Iwata, et al.. (2020). Optimizing Charge Switching in Membrane Lytic Peptides for Endosomal Release of Biomacromolecules. Angewandte Chemie. 132(45). 20165–20173. 6 indexed citations

9.

Ohmi, Yuhsuke, Takashi Nishikaze, Takako Ito, et al.. (2020). Majority of alpha2,6-sialylated glycans in the adult mouse brain exist inO-glycans: SALSA-MS analysis for knockout mice of alpha2,6-sialyltransferase genes. Glycobiology. 31(5). 557–570. 13 indexed citations

10.

Kanemaru, Kazumasa, Emiko Noguchi, Satoko Tahara‐Hanaoka, et al.. (2019). Clec10a regulates mite-induced dermatitis. Science Immunology. 4(42). 22 indexed citations

11.

Honda, Takato, Tomoyuki Fujiyama, Chika Miyoshi, et al.. (2018). A single phosphorylation site of SIK3 regulates daily sleep amounts and sleep need in mice. Proceedings of the National Academy of Sciences. 115(41). 10458–10463. 43 indexed citations

12.

Oishi, Hisashi, et al.. (2017). Germline recombination in a novel Cre transgenic line, Prl3b1-cre mouse. 2 indexed citations

13.

Mizuno, Seiya, Kanako Kato, Yoko Tanimoto, et al.. (2014). Simple generation of albino C57BL/6J mice with G291T mutation in the tyrosinase gene by the CRISPR/Cas9 system. Mammalian Genome. 25(7-8). 327–334. 86 indexed citations

14.

Sugiyama, Fumihiro, et al.. (2012). Renoprotective mechanisms of telmisartan on renal injury and inflammation in SHRSP.Z-Leprfa/IzmDmcr rats. Clinical and Experimental Nephrology. 17(4). 515–524. 12 indexed citations

15.

Oshima, Motohiko, Mitsuhiro Endoh, Takaho A. Endo, et al.. (2011). Genome-wide analysis of target genes regulated by HoxB4 in hematopoietic stem and progenitor cells developing from embryonic stem cells. Blood. 117(15). e142–e150. 38 indexed citations

16.

Tanimoto, Yoko, Seiya Mizuno, Yoko Daitoku, et al.. (2010). Effect of Different Culture Conditions on Establishment of Embryonic Stem Cells from BALB/cAJ and NZB/BINJ Mice. Cellular Reprogramming. 12(6). 679–688. 11 indexed citations

17.

Miyauchi, Takashi, Yoshihiko Kakinuma, Satoshi Sakai, et al.. (2003). The endothelin receptor antagonist ameliorates the hypertensive phenotypes of transgenic hypertensive mice with renin-angiotensin genes and discloses roles of organ specific activation of endothelin system in transgenic mice. Life Sciences. 74(9). 1105–1118. 8 indexed citations

18.

Sugiyama, Fumihiro, et al.. (2001). Mouse models of blood pressure regulation and hypertension. Current Hypertension Reports. 3(1). 41–48. 23 indexed citations

19.

Ohshima, Takayuki, Makoto Iwama, Takeshi Nakajima, et al.. (1998). Induction of apoptosis in vitro and in vivo by H-1 parvovirus infection.. Journal of General Virology. 79(12). 3067–3071. 37 indexed citations

20.

Miyauchi, Takashi, Tsutomu Kobayashi, Yoshiko Takata, et al.. (1998). Endothelin-1 Expression in Hearts of Transgenic Hypertensive Mice Overexpressing Angiotensin II. Journal of Cardiovascular Pharmacology. 31. S412–S416. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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