Fengxiang Wei

3.5k total citations
49 papers, 823 citations indexed

About

Fengxiang Wei is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Fengxiang Wei has authored 49 papers receiving a total of 823 indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Molecular Biology, 16 papers in Oncology and 6 papers in Genetics. Recurrent topics in Fengxiang Wei's work include Cancer-related Molecular Pathways (9 papers), DNA Repair Mechanisms (8 papers) and Wnt/β-catenin signaling in development and cancer (5 papers). Fengxiang Wei is often cited by papers focused on Cancer-related Molecular Pathways (9 papers), DNA Repair Mechanisms (8 papers) and Wnt/β-catenin signaling in development and cancer (5 papers). Fengxiang Wei collaborates with scholars based in China, Canada and United States. Fengxiang Wei's co-authors include Damu Tang, Lizhi He, Yanyun Xie, Lijian Tao, Xiaozeng Lin, Jenny Yan, Diane Ojo, Judy Yan, Nicholas Wong and Pierre Major and has published in prestigious journals such as Nature Communications, Blood and Cancer Research.

In The Last Decade

Fengxiang Wei

48 papers receiving 813 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Fengxiang Wei China 18 543 192 151 102 101 49 823
Godfrey Grech Malta 15 533 1.0× 154 0.8× 212 1.4× 52 0.5× 75 0.7× 47 855
Xing‐sheng Shu China 16 657 1.2× 107 0.6× 197 1.3× 68 0.7× 57 0.6× 39 929
Jason Kang South Korea 14 679 1.3× 146 0.8× 188 1.2× 77 0.8× 145 1.4× 22 1.0k
Maria Zeniou France 17 619 1.1× 265 1.4× 154 1.0× 61 0.6× 171 1.7× 25 947
Shan Cheng China 18 377 0.7× 109 0.6× 108 0.7× 68 0.7× 49 0.5× 67 734
F. Darío Cuello-Carrión Argentina 17 513 0.9× 164 0.9× 140 0.9× 167 1.6× 44 0.4× 37 831
Maryam Rezaei Iran 19 450 0.8× 137 0.7× 261 1.7× 47 0.5× 83 0.8× 52 840
Aurélie Bedel France 20 888 1.6× 248 1.3× 255 1.7× 61 0.6× 155 1.5× 41 1.3k
Remya Raja India 15 361 0.7× 161 0.8× 175 1.2× 50 0.5× 97 1.0× 29 807
Yanjun Wu China 15 454 0.8× 134 0.7× 188 1.2× 57 0.6× 91 0.9× 42 746

Countries citing papers authored by Fengxiang Wei

Since Specialization
Citations

This map shows the geographic impact of Fengxiang Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fengxiang Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fengxiang Wei more than expected).

Fields of papers citing papers by Fengxiang Wei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fengxiang Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fengxiang Wei. The network helps show where Fengxiang Wei may publish in the future.

Co-authorship network of co-authors of Fengxiang Wei

This figure shows the co-authorship network connecting the top 25 collaborators of Fengxiang Wei. A scholar is included among the top collaborators of Fengxiang Wei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fengxiang Wei. Fengxiang Wei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pan, Teng, Rui Duan, Zihan Xu, et al.. (2025). GDF-15 as a biomarker for diagnosis and prognosis of lung cancer: a meta-analysis. Frontiers in Oncology. 15. 1447990–1447990. 1 indexed citations
2.
Wei, Fengxiang, et al.. (2024). Genome-wide association studies of thyroid-related hormones, dysfunction, and autoimmunity among 85,421 Chinese pregnancies. Nature Communications. 15(1). 8004–8004. 3 indexed citations
3.
Chen, Xiaohang, Yanan Liu, Xiaojin Luo, et al.. (2024). HPV16 E6-induced M2 macrophage polarization in the cervical microenvironment via exosomal miR-204-5p. Scientific Reports. 14(1). 23725–23725. 4 indexed citations
4.
Chen, Xiaohang, Shujia Huang, Xinxin Guo, et al.. (2024). Genetic study of intrahepatic cholestasis of pregnancy in Chinese women unveils East Asian etiology linked to historic HBV epidemic. Journal of Hepatology. 82(5). 826–835. 3 indexed citations
5.
6.
Pei, Yuanyuan, et al.. (2023). Up-regulated GRB7 protein in gastric cancer cells correlates with clinical properties and increases proliferation and stem cell properties. Frontiers in Oncology. 12. 1054976–1054976. 6 indexed citations
7.
Li, Jianxiong, et al.. (2023). Chromosome instability region analysis and identification of the driver genes of the epithelial ovarian cancer cell lines A2780 and SKOV3. Journal of Cellular and Molecular Medicine. 27(21). 3259–3270. 8 indexed citations
8.
Deng, Jinhai, Teng Pan, Lulu Cao, et al.. (2023). Exosomal transfer leads to chemoresistance through oxidative phosphorylation-mediated stemness phenotype in colorectal cancer. Theranostics. 13(14). 5057–5074. 18 indexed citations
9.
Lin, Xiaozeng, Ying Dong, Yan Gu, et al.. (2023). Taxifolin Inhibits the Growth of Non-Small-Cell Lung Cancer via Downregulating Genes Displaying Novel and Robust Associations with Immune Evasion Factors. Cancers. 15(19). 4818–4818. 2 indexed citations
10.
He, Lizhi, Henry Pratt, Mingshi Gao, et al.. (2021). YAP and TAZ are transcriptional co-activators of AP-1 proteins and STAT3 during breast cellular transformation. eLife. 10. 74 indexed citations
11.
Tang, Jia, Meihua Tan, Yihui Deng, et al.. (2021). Two Novel Pathogenic Variants of TJP2 Gene and the Underlying Molecular Mechanisms in Progressive Familial Intrahepatic Cholestasis Type 4 Patients. Frontiers in Cell and Developmental Biology. 9. 661599–661599. 8 indexed citations
12.
Li, Dongxue, Bing Qiu, Xiaohang Chen, et al.. (2020). Expression of CYB5D2 expression is associated with epithelial-mesenchymal transition and survival rates in patients with cervical cancer. Translational Cancer Research. 9(2). 1185–1194. 2 indexed citations
13.
Ojo, Diane, David Rodríguez, Fengxiang Wei, Anita Bane, & Damu Tang. (2019). Downregulation of CYB5D2 is associated with breast cancer progression. Scientific Reports. 9(1). 6624–6624. 11 indexed citations
14.
Yan, Judy, Diane Ojo, Anil Kapoor, et al.. (2016). Neural Cell Adhesion Protein CNTN1 Promotes the Metastatic Progression of Prostate Cancer. Cancer Research. 76(6). 1603–1614. 40 indexed citations
15.
Wei, Fengxiang, Diane Ojo, Xuelei Lin, et al.. (2014). BMI1 attenuates etoposide-induced G2/M checkpoints via reducing ATM activation. Oncogene. 34(23). 3063–3075. 28 indexed citations
16.
Melo, Jason De, Xiaozeng Lin, Lizhi He, et al.. (2014). SIPL1-facilitated PTEN ubiquitination contributes to its association with PTEN. Cellular Signalling. 26(12). 2749–2756. 17 indexed citations
17.
Wei, Fengxiang, Judy Yan, Damu Tang, et al.. (2012). Inhibition of ERK activation enhances the repair of double-stranded breaks via non-homologous end joining by increasing DNA-PKcs activation. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1833(1). 90–100. 23 indexed citations
18.
Wei, Fengxiang, Jenny Yan, & Damu Tang. (2011). Extracellular Signal-Regulated Kinases Modulate DNA Damage Response - A Contributing Factor to Using MEK Inhibitors in Cancer Therapy. Current Medicinal Chemistry. 18(35). 5476–5482. 54 indexed citations
19.
Wei, Fengxiang, Yanyun Xie, Lizhi He, Lijian Tao, & Damu Tang. (2010). ERK1 and ERK2 kinases activate hydroxyurea-induced S-phase checkpoint in MCF7 cells by mediating ATR activation. Cellular Signalling. 23(1). 259–268. 20 indexed citations
20.
Liu, Yuexia, et al.. (2005). Observation on in vitro culture hepatoma apoptosis by TUNEL dying method. 28(1). 10–11. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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