Federico Figliolini

2.2k total citations · 1 hit paper
21 papers, 1.8k citations indexed

About

Federico Figliolini is a scholar working on Molecular Biology, Surgery and Nephrology. According to data from OpenAlex, Federico Figliolini has authored 21 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 8 papers in Surgery and 8 papers in Nephrology. Recurrent topics in Federico Figliolini's work include Extracellular vesicles in disease (11 papers), MicroRNA in disease regulation (5 papers) and Acute Kidney Injury Research (5 papers). Federico Figliolini is often cited by papers focused on Extracellular vesicles in disease (11 papers), MicroRNA in disease regulation (5 papers) and Acute Kidney Injury Research (5 papers). Federico Figliolini collaborates with scholars based in Italy, Germany and United States. Federico Figliolini's co-authors include Giovanni Camussi, Ciro Tetta, Maria Chiara Deregibus, Vincenzo Cantaluppi, Luigi Biancone, Davide Medica, Stefania Bruno, Maria Felice Brizzi, Stefano Gatti and Andrea Sordi and has published in prestigious journals such as PLoS ONE, Scientific Reports and International Journal of Molecular Sciences.

In The Last Decade

Federico Figliolini

21 papers receiving 1.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Differential Therapeutic Effect of Extracellular Vesicles Derived by Bone Marrow and Adipose Mesenchymal Stem Cells on Wound Healing of Diabetic Ulcers and Correlation to Their Cargoes

2021 183 citations Margherita Alba Carlotta Pomatto, Chiara Gai et al. International Journal of Molecular Sciences profile →

Peers

Federico Figliolini

SURGE

NEPHR

GENET

Valentina Fonsato Italy

Sebastian Grundmann Germany

Susan C. Hubchak United States

Taek‐Kyun Kim South Korea

Michael A. Kuliszewski Canada

Jasper Koerts Netherlands

Stefano Buttiglieri Italy

Noritoshi Kato Japan

Guanqun Ju China

Jonghoe Byun South Korea

Valentina Fonsato Italy

Federico Figliolini

1.4k ×1.1

813 ×1.1

458 ×1.1

SURGE

233 ×1.1

NEPHR

431 ×2.2

GENET

Citations per year, relative to Federico Figliolini Federico Figliolini (= 1×) peers Valentina Fonsato

Countries citing papers authored by Federico Figliolini

Since Specialization

Citations

This map shows the geographic impact of Federico Figliolini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Federico Figliolini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Federico Figliolini more than expected).

Fields of papers citing papers by Federico Figliolini

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Federico Figliolini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Federico Figliolini. The network helps show where Federico Figliolini may publish in the future.

Co-authorship network of co-authors of Federico Figliolini

This figure shows the co-authorship network connecting the top 25 collaborators of Federico Figliolini. A scholar is included among the top collaborators of Federico Figliolini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Federico Figliolini. Federico Figliolini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Medica, Davide, Rossana Franzin, Alessandra Stasi, et al.. (2021). Extracellular Vesicles Derived from Endothelial Progenitor Cells Protect Human Glomerular Endothelial Cells and Podocytes from Complement- and Cytokine-Mediated Injury. Cells. 10(7). 1675–1675. 33 indexed citations

Pomatto, Margherita Alba Carlotta, Chiara Gai, Federica Negro, et al.. (2021). Differential Therapeutic Effect of Extracellular Vesicles Derived by Bone Marrow and Adipose Mesenchymal Stem Cells on Wound Healing of Diabetic Ulcers and Correlation to Their Cargoes. International Journal of Molecular Sciences. 22(8). 3851–3851. 183 indexed citations breakdown →

Cavallari, Claudia, Federico Figliolini, Marta Tapparo, et al.. (2020). miR-130a and Tgfβ Content in Extracellular Vesicles Derived from the Serum of Subjects at High Cardiovascular Risk Predicts their In-Vivo Angiogenic Potential. Scientific Reports. 10(1). 706–706. 16 indexed citations

Bruno, Stefania, Chiara Pasquino, Maria Beatriz Herrera Sanchez, et al.. (2019). HLSC-Derived Extracellular Vesicles Attenuate Liver Fibrosis and Inflammation in a Murine Model of Non-alcoholic Steatohepatitis. Molecular Therapy. 28(2). 479–489. 109 indexed citations

Figliolini, Federico, Andrea Ranghino, Cristina Grange, et al.. (2019). Extracellular Vesicles From Adipose Stem Cells Prevent Muscle Damage and Inflammation in a Mouse Model of Hind Limb Ischemia. Arteriosclerosis Thrombosis and Vascular Biology. 40(1). 239–254. 69 indexed citations

Cavallari, Claudia, Andrea Ranghino, Marta Tapparo, et al.. (2017). Serum-derived extracellular vesicles (EVs) impact on vascular remodeling and prevent muscle damage in acute hind limb ischemia. Scientific Reports. 7(1). 8180–8180. 60 indexed citations

Cantaluppi, Vincenzo, Davide Medica, Alessandro Domenico Quercia, et al.. (2017). Perfluorocarbon solutions limit tubular epithelial cell injury and promote CD133+ kidney progenitor differentiation: potential use in renal assist devices for sepsis-associated acute kidney injury and multiple organ failure. Nephrology Dialysis Transplantation. 33(7). 1110–1121. 14 indexed citations

Deregibus, Maria Chiara, Federico Figliolini, Sergio D’Antico, et al.. (2016). Charge-based precipitation of extracellular vesicles. International Journal of Molecular Medicine. 38(5). 1359–1366. 247 indexed citations

Navarro-Tableros, Víctor, Maria Beatriz Herrera Sanchez, Federico Figliolini, et al.. (2015). Recellularization of Rat Liver Scaffolds by Human Liver Stem Cells. Tissue Engineering Part A. 21(11-12). 1929–1939. 59 indexed citations

10.

Cantaluppi, Vincenzo, Sergio Dellepiane, Davide Medica, et al.. (2015). Neutrophil Gelatinase Associated Lipocalin Is an Early and Accurate Biomarker of Graft Function and Tissue Regeneration in Kidney Transplantation from Extended Criteria Donors. PLoS ONE. 10(6). e0129279–e0129279. 41 indexed citations

11.

Figliolini, Federico, Vincenzo Cantaluppi, Michela De Lena, et al.. (2014). Isolation, Characterization and Potential Role in Beta Cell-Endothelium Cross-Talk of Extracellular Vesicles Released from Human Pancreatic Islets. PLoS ONE. 9(7). e102521–e102521. 87 indexed citations

12.

Cantaluppi, Vincenzo, Davide Medica, Claudio Mannari, et al.. (2014). Endothelial progenitor cell-derived extracellular vesicles protect from complement-mediated mesangial injury in experimental anti-Thy1.1 glomerulonephritis. Nephrology Dialysis Transplantation. 30(3). 410–422. 65 indexed citations

13.

Cantaluppi, Vincenzo, Stefano Gatti, Davide Medica, et al.. (2012). Microvesicles derived from endothelial progenitor cells protect the kidney from ischemia–reperfusion injury by microRNA-dependent reprogramming of resident renal cells. Kidney International. 82(4). 412–427. 440 indexed citations

14.

Cantaluppi, Vincenzo, Luigi Biancone, Federico Figliolini, et al.. (2012). Microvesicles Derived from Endothelial Progenitor Cells Enhance Neoangiogenesis of Human Pancreatic Islets. Cell Transplantation. 21(6). 1305–1320. 164 indexed citations

15.

Dellepiane, Sergio, et al.. (2012). [New mechanisms and recent insights in the pathogenesis of acute kidney injury (AKI)].. PubMed. 29(5). 535–47. 12 indexed citations

16.

Cantaluppi, Vincenzo, Viktoria Weber, Federico Figliolini, et al.. (2010). Protective effect of resin adsorption on septic plasma-induced tubular injury. Critical Care. 14(1). R4–R4. 43 indexed citations

17.

Figliolini, Federico, et al.. (2009). Biological Activity of Circulating Plasma Exosomes in Acute and Chronic Renal Failure: Identification of New Uremic Toxins?. Blood Purification. 28. 293–293. 1 indexed citations

18.

Cantaluppi, Vincenzo, Luigi Biancone, Gianfranco Romanazzi, et al.. (2008). Macrophage Stimulating Protein May Promote Tubular Regeneration after Acute Injury. Journal of the American Society of Nephrology. 19(10). 1904–1918. 42 indexed citations

19.

Cantaluppi, Vincenzo, Luigi Biancone, Gianfranco Romanazzi, et al.. (2006). Antiangiogenic and Immunomodulatory Effects of Rapamycin on Islet Endothelium: Relevance for Islet Transplantation. American Journal of Transplantation. 6(11). 2601–2611. 59 indexed citations

20.

Biancone, Luigi, Simonetta Geninatti Crich, Vincenzo Cantaluppi, et al.. (2006). Magnetic resonance imaging of gadolinium‐labeled pancreatic islets for experimental transplantation. NMR in Biomedicine. 20(1). 40–48. 76 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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