Fangfang Jin

2.1k total citations
39 papers, 1.5k citations indexed

About

Fangfang Jin is a scholar working on Molecular Biology, Cancer Research and Immunology. According to data from OpenAlex, Fangfang Jin has authored 39 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 15 papers in Cancer Research and 8 papers in Immunology. Recurrent topics in Fangfang Jin's work include MicroRNA in disease regulation (12 papers), RNA modifications and cancer (8 papers) and Cancer-related molecular mechanisms research (5 papers). Fangfang Jin is often cited by papers focused on MicroRNA in disease regulation (12 papers), RNA modifications and cancer (8 papers) and Cancer-related molecular mechanisms research (5 papers). Fangfang Jin collaborates with scholars based in China, United States and Pakistan. Fangfang Jin's co-authors include Ke Zen, Chen‐Yu Zhang, Yanbo Wang, Hongwei Liang, Mingzhen Li, Yanan Zhu, Xi Chen, Yong Guan, Xia Yin and Anna Zheng and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Nature Communications.

In The Last Decade

Fangfang Jin

37 papers receiving 1.4k citations

Peers

Fangfang Jin
Fangfang Jin
Citations per year, relative to Fangfang Jin Fangfang Jin (= 1×) peers Lichao Sun

Countries citing papers authored by Fangfang Jin

Since Specialization
Citations

This map shows the geographic impact of Fangfang Jin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fangfang Jin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fangfang Jin more than expected).

Fields of papers citing papers by Fangfang Jin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fangfang Jin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fangfang Jin. The network helps show where Fangfang Jin may publish in the future.

Co-authorship network of co-authors of Fangfang Jin

This figure shows the co-authorship network connecting the top 25 collaborators of Fangfang Jin. A scholar is included among the top collaborators of Fangfang Jin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fangfang Jin. Fangfang Jin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yan, Shu, Jun Ren, Jing Yang, et al.. (2025). NRF2 maintains redox balance via ME1 and NRF2 inhibitor synergizes with venetoclax in NPM1-mutated acute myeloid leukemia. Cancer & Metabolism. 13(1). 32–32.
2.
Yang, Ping, Gaoli Liang, Yangyue Ni, et al.. (2025). Investigating the role of intratumoral Streptococcus mitis in gastric cancer progression: insights into tumor microenvironment. Journal of Translational Medicine. 23(1). 126–126. 3 indexed citations
3.
Ren, Jun, Junpeng Huang, Zailin Yang, et al.. (2024). Cytoplasmic TP53INP2 acts as an apoptosis partner in TRAIL treatment: the synergistic effect of TRAIL with venetoclax in TP53INP2-positive acute myeloid leukemia. Journal of Experimental & Clinical Cancer Research. 43(1). 176–176. 4 indexed citations
4.
Yu, Wei, Dian Zhang, He Shi, et al.. (2023). PDK1 promotes breast cancer progression by enhancing the stability and transcriptional activity of HIF-1α. Genes & Diseases. 11(4). 101041–101041. 8 indexed citations
5.
Li, Changtao, Lijuan Pang, Fangfang Jin, et al.. (2023). Integrated Network Analysis to Determine CNN1, MYL9, TAGLN, and SORBS1 as Potential Key Genes Associated with Prostate Cancer. Clinical Laboratory. 69(07/2023). 2 indexed citations
6.
Jin, Fangfang, et al.. (2023). The function of decidua natural killer cells in physiology and pathology of pregnancy. American Journal of Reproductive Immunology. 90(3). 6 indexed citations
7.
Jin, Fangfang, Qian Zeng, Husun Qian, et al.. (2022). Dual-Targeted Self-Assembled DNA Hydrogels Decorated With Multivalent Aptamers Loaded With DOX for Anticancer Therapy. Frontiers in Pharmacology. 13. 807498–807498. 16 indexed citations
8.
Zhan, Shoubin, Ping Yang, Ye Xu, et al.. (2022). Serum mitochondrial tsRNA serves as a novel biomarker for hepatocarcinoma diagnosis. Frontiers of Medicine. 16(2). 216–226. 35 indexed citations
9.
Meng, Huan, Shuang Wang, Xiaomeng Tang, et al.. (2022). Respiratory immune status and microbiome in recovered COVID-19 patients revealed by metatranscriptomic analyses. Frontiers in Cellular and Infection Microbiology. 12. 1011672–1011672. 3 indexed citations
10.
Wang, Yanbo, Zhang‐Peng Chen, Huanhuan Hu, et al.. (2021). Sperm microRNAs confer depression susceptibility to offspring. Science Advances. 7(7). 78 indexed citations
11.
Fang, Wenli, Ting Zhou, He Shi, et al.. (2021). Progranulin induces immune escape in breast cancer via up-regulating PD-L1 expression on tumor-associated macrophages (TAMs) and promoting CD8+ T cell exclusion. Journal of Experimental & Clinical Cancer Research. 40(1). 4–4. 158 indexed citations
12.
Jin, Fangfang, Rong Yang, Wei Yao, et al.. (2018). HIF-1α-induced miR-23a∼27a∼24 cluster promotes colorectal cancer progression via reprogramming metabolism. Cancer Letters. 440-441. 211–222. 49 indexed citations
13.
Jin, Fangfang, Yanbo Wang, Yanan Zhu, et al.. (2017). The miR-125a/HK2 axis regulates cancer cell energy metabolism reprogramming in hepatocellular carcinoma. Scientific Reports. 7(1). 3089–3089. 64 indexed citations
14.
Wei, Yao, Dong Wang, Fangfang Jin, et al.. (2017). Pyruvate kinase type M2 promotes tumour cell exosome release via phosphorylating synaptosome-associated protein 23. Nature Communications. 8(1). 14041–14041. 239 indexed citations
15.
Wang, Yanbo, Zhenyu Wu, Minghai Wang, et al.. (2016). miR-124-3p functions as a tumor suppressor in breast cancer by targeting CBL. BMC Cancer. 16(1). 826–826. 86 indexed citations
16.
Li, Limin, Tao Zhang, Wenli Diao, et al.. (2015). Role of Myeloid-Derived Suppressor Cells in Glucocorticoid-Mediated Amelioration of FSGS. Journal of the American Society of Nephrology. 26(9). 2183–2197. 29 indexed citations
17.
Diao, Wenli, Fangfang Jin, Bing Wang, et al.. (2014). The protective role of myeloid-derived suppressor cells in concanavalin A-induced hepatic injury. Protein & Cell. 5(9). 714–724. 29 indexed citations
18.
Xu, Yongjie, Yanbo Wang, Fangfang Jin, et al.. (2013). Porcine skeletal muscle differentially expressed gene ATP5B: molecular characterization, expression patterns, and association analysis with meat quality traits. Mammalian Genome. 24(3-4). 142–150. 25 indexed citations
19.
Wu, Deyan, Fangfang Jin, Weiqiang Lü, et al.. (2012). Synthesis, Structure–Activity Relationship, and Pharmacophore Modeling Studies of Pyrazole‐3‐Carbohydrazone Derivatives as Dipeptidyl Peptidase IV Inhibitors. Chemical Biology & Drug Design. 79(6). 897–906. 25 indexed citations
20.
Jin, Fangfang, Chunhua Lu, Xianqiang Sun, et al.. (2011). Insights into the binding modes of human β3-adrenergic receptor agonists with ligand-based and receptor-based methods. Molecular Diversity. 15(4). 817–831. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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