F. van der Ham

825 total citations
15 papers, 629 citations indexed

About

F. van der Ham is a scholar working on Molecular Biology, Surgery and Epidemiology. According to data from OpenAlex, F. van der Ham has authored 15 papers receiving a total of 629 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 4 papers in Surgery and 4 papers in Epidemiology. Recurrent topics in F. van der Ham's work include Osteoarthritis Treatment and Mechanisms (4 papers), Neuroblastoma Research and Treatments (2 papers) and Genetic Neurodegenerative Diseases (2 papers). F. van der Ham is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (4 papers), Neuroblastoma Research and Treatments (2 papers) and Genetic Neurodegenerative Diseases (2 papers). F. van der Ham collaborates with scholars based in Netherlands, United States and United Kingdom. F. van der Ham's co-authors include Jeroen DeGroot, Alice Maroudas, Sarit Sivan, Sally Roberts, Ellen Wachtel, Ruud A. Bank, Tony M. Keaveny, Christopher J. Hernandez, Simon Y. Tang and A.N. Sakkee and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and The Journal of Clinical Endocrinology & Metabolism.

In The Last Decade

F. van der Ham

13 papers receiving 614 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
F. van der Ham Netherlands 9 170 149 145 140 128 15 629
Shigeru Soshi Japan 11 141 0.8× 233 1.6× 102 0.7× 201 1.4× 31 0.2× 22 632
Edward J. Puzas United States 13 231 1.4× 88 0.6× 63 0.4× 153 1.1× 60 0.5× 18 550
Qinghe Zeng United States 10 174 1.0× 149 1.0× 84 0.6× 77 0.6× 33 0.3× 19 439
Glen Blake United Kingdom 4 282 1.7× 356 2.4× 66 0.5× 119 0.8× 37 0.3× 5 645
Tadamasa Hanyu Japan 16 100 0.6× 130 0.9× 284 2.0× 291 2.1× 97 0.8× 44 774
Takeshi Fujita Japan 14 322 1.9× 92 0.6× 65 0.4× 77 0.6× 20 0.2× 43 722
Carole Bougault France 19 313 1.8× 95 0.6× 514 3.5× 159 1.1× 86 0.7× 34 921
Na-Young Bae South Korea 5 216 1.3× 44 0.3× 292 2.0× 65 0.5× 30 0.2× 20 644
L. Laadhar Tunisia 16 181 1.1× 29 0.2× 285 2.0× 96 0.7× 71 0.6× 47 593
Shunichiro Nagahata Japan 13 191 1.1× 41 0.3× 81 0.6× 159 1.1× 21 0.2× 43 532

Countries citing papers authored by F. van der Ham

Since Specialization
Citations

This map shows the geographic impact of F. van der Ham's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by F. van der Ham with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites F. van der Ham more than expected).

Fields of papers citing papers by F. van der Ham

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by F. van der Ham. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by F. van der Ham. The network helps show where F. van der Ham may publish in the future.

Co-authorship network of co-authors of F. van der Ham

This figure shows the co-authorship network connecting the top 25 collaborators of F. van der Ham. A scholar is included among the top collaborators of F. van der Ham based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with F. van der Ham. F. van der Ham is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Kim, Hyeongju, Sophie Lenoir, Angela Helfricht, et al.. (2022). A pathogenic proteolysis–resistant huntingtin isoform induced by an antisense oligonucleotide maintains huntingtin function. JCI Insight. 7(17). 7 indexed citations
2.
Kozijn, A.E., F. van der Ham, David A. Barrett, et al.. (2018). Human C-reactive protein aggravates osteoarthritis development in mice on a high-fat diet. Osteoarthritis and Cartilage. 27(1). 118–128. 25 indexed citations
3.
Klein, Pontus, Lodewijk J.A. Toonen, Hyeongju Kim, et al.. (2018). I01 QRX-704, a novel antisense oligonucleotide therapy, designed to prevent hd pathology while maintaining htt function. A88.2–A88. 1 indexed citations
4.
Kozijn, A.E., Lobke Gierman, F. van der Ham, et al.. (2017). Variable cartilage degradation in mice with diet-induced metabolic dysfunction: food for thought. Osteoarthritis and Cartilage. 26(1). 95–107. 20 indexed citations
5.
Gierman, Lobke, B. van El, F. van der Ham, et al.. (2013). Profiling the Secretion of Soluble Mediators by End Stage Osteoarthritis Synovial Tissue Explants Reveals a Reduced Responsiveness to an Inflammatory Trigger. PLoS ONE. 8(5). e62634–e62634. 5 indexed citations
6.
Gierman, Lobke, F. van der Ham, Angela Koudijs, et al.. (2011). Metabolic stress–induced inflammation plays a major role in the development of osteoarthritis in mice. Arthritis & Rheumatism. 64(4). 1172–1181. 61 indexed citations
7.
Sivan, Sarit, Ellen Wachtel, F. van der Ham, et al.. (2008). Collagen Turnover in Normal and Degenerate Human Intervertebral Discs as Determined by the Racemization of Aspartic Acid. Journal of Biological Chemistry. 283(14). 8796–8801. 106 indexed citations
8.
Sivan, Sarit, Ellen Wachtel, Peter J. Roughley, et al.. (2006). Aggrecan Turnover in Human Intervertebral Disc as Determined by the Racemization of Aspartic Acid. Journal of Biological Chemistry. 281(19). 13009–13014. 73 indexed citations
9.
Hernandez, Christopher J., Simon Y. Tang, A.N. Sakkee, et al.. (2005). Trabecular microfracture and the influence of pyridinium and non-enzymatic glycation-mediated collagen cross-links. Bone. 37(6). 825–832. 171 indexed citations
10.
Wal, Bart C. H. van der, Ronald R. de Krijger, Wouter W. de Herder, et al.. (2000). Adult hyperinsulinemic hypoglycemia not caused by an insulinoma: a report of two cases. Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin. 436(5). 481–486. 22 indexed citations
11.
Hofland, Leo J., P. M. van Koetsveld, Joke M. Zuijderwijk, et al.. (1999). Immunohistochemical Detection of Somatostatin Receptor Subtypes sst1 and sst2A in Human Somatostatin Receptor Positive Tumors. The Journal of Clinical Endocrinology & Metabolism. 84(2). 775–780. 105 indexed citations
12.
Baan, Carla C., Teun van Gelder, F. van der Ham, et al.. (1998). Contribution of the Inflammatory Response to Cardiac Allograft Rejection: Histopathologic Analysis of Serial Endomyocardial Biopsies. Transplantation Proceedings. 30(4). 1104–1106. 3 indexed citations
13.
Baan, Carla C., Cécile Holweg, Teun van Gelder, et al.. (1998). ANALYSIS OF THE INTRAGRAFT CYTOKINE NETWORK IN HEART TRANSPLANT PATIENTS TREATED WITH AN ANTI-CD25 ANTIBODY. Transplantation. 65(Supplement). 111–111. 1 indexed citations
14.
Meijer, Piet, F. van der Ham, & Cornelis Kluft. (1996). The use of Stabilyte® plasma may cause changes in pH in the assay of some fibrinolysis analytes and might effect results. Fibrinolysis and Proteolysis. 10. 155–157. 1 indexed citations
15.
Gijbels, Marion J., F. van der Ham, Ariëtte M. van Bennekum, H. Hendriks, & Paul Roholl. (1992). Alterations in cytokeratin expression precede histological changes in epithelia of vitamin A-deficient rats. Cell and Tissue Research. 268(1). 197–203. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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