F. M. Meng

429 total citations
10 papers, 196 citations indexed

About

F. M. Meng is a scholar working on Molecular Biology, Oncology and Epidemiology. According to data from OpenAlex, F. M. Meng has authored 10 papers receiving a total of 196 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 3 papers in Oncology and 3 papers in Epidemiology. Recurrent topics in F. M. Meng's work include Autophagy in Disease and Therapy (3 papers), Cardiac Fibrosis and Remodeling (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). F. M. Meng is often cited by papers focused on Autophagy in Disease and Therapy (3 papers), Cardiac Fibrosis and Remodeling (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). F. M. Meng collaborates with scholars based in China and United States. F. M. Meng's co-authors include Molly Brewer, Yang Yu, Guohui Sun, Guohai Su, Ying Li, Mei Zhong, Yanhong Yu, Hui‐Hui Wu, Jiamin Chen and Shuya Wang and has published in prestigious journals such as Cancer Research, Biochemical and Biophysical Research Communications and British Journal of Cancer.

In The Last Decade

F. M. Meng

9 papers receiving 194 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
F. M. Meng China 7 121 38 31 26 23 10 196
Xuewu Zhang China 8 96 0.8× 21 0.6× 16 0.5× 25 1.0× 36 1.6× 20 239
Huyen Nguyen United States 5 237 2.0× 25 0.7× 33 1.1× 32 1.2× 25 1.1× 6 336
Natsuko Ohtomo Japan 8 180 1.5× 22 0.6× 52 1.7× 28 1.1× 21 0.9× 12 271
Youhong Dong China 10 95 0.8× 42 1.1× 30 1.0× 20 0.8× 47 2.0× 30 208
Yasuhiro Mizushima Japan 8 131 1.1× 52 1.4× 38 1.2× 15 0.6× 7 0.3× 18 219
Carla Navarro Colombia 6 134 1.1× 36 0.9× 18 0.6× 21 0.8× 58 2.5× 7 286
Luis Leiva‐Vega Spain 7 116 1.0× 30 0.8× 91 2.9× 29 1.1× 26 1.1× 11 276

Countries citing papers authored by F. M. Meng

Since Specialization
Citations

This map shows the geographic impact of F. M. Meng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by F. M. Meng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites F. M. Meng more than expected).

Fields of papers citing papers by F. M. Meng

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by F. M. Meng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by F. M. Meng. The network helps show where F. M. Meng may publish in the future.

Co-authorship network of co-authors of F. M. Meng

This figure shows the co-authorship network connecting the top 25 collaborators of F. M. Meng. A scholar is included among the top collaborators of F. M. Meng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with F. M. Meng. F. M. Meng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Li, Ying, Wenjing Li, Huihui Wu, et al.. (2025). Downregulation of PGAM2 alleviates angiotensin II-induced cardiac hypertrophy by destabilizing HSP90 and inactivating the mTOR/IKKα signaling pathway. International Journal of Biological Sciences. 21(3). 1308–1321. 1 indexed citations
2.
Zhao, Jun, Tingting Zhang, Guomin Chen, et al.. (2022). Non-Structural Protein 3 of Duck Tembusu Virus Induces Autophagy via the ERK and PI3K–AKT–mTOR Signaling Pathways. Frontiers in Immunology. 13. 746890–746890. 19 indexed citations
3.
Wang, Wei, F. M. Meng, Shuya Wang, et al.. (2021). Nicotine Causes Mitochondrial Dynamics Imbalance and Apoptosis Through ROS Mediated Mitophagy Impairment in Cardiomyocytes. Frontiers in Physiology. 12. 650055–650055. 25 indexed citations
4.
Wu, Huihui, Ting-Ting Meng, Jiamin Chen, et al.. (2021). Asenapine maleate inhibits angiotensin II-induced proliferation and activation of cardiac fibroblasts via the ROS/TGFβ1/MAPK signaling pathway. Biochemical and Biophysical Research Communications. 553. 172–179. 13 indexed citations
5.
Wang, Shuya, F. M. Meng, Min Li, et al.. (2020). Cilostazol alleviate nicotine induced cardiomyocytes hypertrophy through modulation of autophagy by CTSB/ROS/p38MAPK/JNK feedback loop. International Journal of Biological Sciences. 16(11). 2001–2013. 24 indexed citations
6.
Meng, F. M., et al.. (2020). Procalcitonin, C-reactive Protein, and Neutrophil Ratio Contribute to the Diagnosis and Prognosis of Severe Acute Pancreatitis. Iranian Journal of Public Health. 48(12). 2177–2186. 15 indexed citations
7.
Yang, Cui, et al.. (2016). [Effects of docosahexaenoic acid on cell apoptosis, invasion and migration of cervical cancer cells in vitro].. PubMed. 36(6). 848–56. 6 indexed citations
8.
9.
Meng, F. M., et al.. (2013). Anticancer efficacy of cisplatin and trichostatin A or 5-aza-2′-deoxycytidine on ovarian cancer. British Journal of Cancer. 108(3). 579–586. 61 indexed citations
10.
Meng, F. M., Mei Zhong, Yanhong Yu, et al.. (2012). Abstract 1406: A new ovarian cancer metastasis model using multicellular spheroids generated from human ovarian cancers tissues. Cancer Research. 72(8_Supplement). 1406–1406.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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