F. Deák

10.0k total citations · 1 hit paper
131 papers, 5.3k citations indexed

About

F. Deák is a scholar working on Molecular Biology, Nuclear and High Energy Physics and Radiation. According to data from OpenAlex, F. Deák has authored 131 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 49 papers in Molecular Biology, 37 papers in Nuclear and High Energy Physics and 28 papers in Radiation. Recurrent topics in F. Deák's work include Nuclear physics research studies (37 papers), Nuclear Physics and Applications (26 papers) and Cell Adhesion Molecules Research (15 papers). F. Deák is often cited by papers focused on Nuclear physics research studies (37 papers), Nuclear Physics and Applications (26 papers) and Cell Adhesion Molecules Research (15 papers). F. Deák collaborates with scholars based in Hungary, United States and Japan. F. Deák's co-authors include Ege T. Kavalali, Thomas C. Südhof, William E. Sonntag, Xinran Liu, Yıldırım Sara, Ibolya Kiss, Susanne Schoch, Anna Csiszár, Zoltán Ungvári and Marina G. Mozhayeva and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Physical Review Letters.

In The Last Decade

F. Deák

128 papers receiving 5.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

SNARE Function Analyzed in Synaptobrevin/VAMP Knockout Mice

2001 503 citations Susanne Schoch, F. Deák et al. Science profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
F. Deák Hungary	35	2.4k	1.5k	1.2k	656	641	131	5.3k
P. Sonderegger Switzerland	53	3.6k 1.5×	2.0k 1.3×	2.6k 2.2×	973 1.5×	722 1.1×	161	8.0k
Kazuhiko Kume Japan	43	3.3k 1.4×	447 0.3×	2.2k 1.9×	211 0.3×	1.6k 2.5×	149	9.5k
M. Kœnig France	46	7.1k 3.0×	1.1k 0.8×	2.2k 1.8×	2.8k 4.3×	1.1k 1.7×	301	12.4k
P. G. Barth Netherlands	59	7.1k 3.0×	650 0.4×	1.1k 0.9×	102 0.2×	967 1.5×	300	11.4k
Roland Schüle Germany	53	9.0k 3.8×	545 0.4×	719 0.6×	224 0.3×	523 0.8×	140	12.3k
Markus Rudin Switzerland	53	1.8k 0.8×	193 0.1×	1.5k 1.2×	84 0.1×	823 1.3×	233	8.3k
Russell E. Jacobs United States	45	3.4k 1.4×	266 0.2×	1.1k 0.9×	153 0.2×	1.2k 1.9×	136	8.8k
Mark F. Lythgoe United Kingdom	51	2.2k 0.9×	294 0.2×	1.2k 1.0×	104 0.2×	633 1.0×	244	9.6k
Markus Morawski Germany	35	1.5k 0.6×	1.3k 0.9×	1.2k 1.0×	34 0.1×	778 1.2×	108	4.1k
David Yaffe Israel	46	7.8k 3.3×	1.3k 0.9×	1.1k 1.0×	234 0.4×	1.2k 1.9×	108	9.6k

Countries citing papers authored by F. Deák

Since Specialization

Citations

This map shows the geographic impact of F. Deák's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by F. Deák with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites F. Deák more than expected).

Fields of papers citing papers by F. Deák

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by F. Deák. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by F. Deák. The network helps show where F. Deák may publish in the future.

Co-authorship network of co-authors of F. Deák

This figure shows the co-authorship network connecting the top 25 collaborators of F. Deák. A scholar is included among the top collaborators of F. Deák based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with F. Deák. F. Deák is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Gupta, Sonu Kumar, F. Deák, Xin‐Yun Lu, et al.. (2024). Gut microbiota dysbiosis in Alzheimer's disease (AD): Insights from human clinical studies and the mouse AD models. Physiology & Behavior. 290. 114778–114778. 9 indexed citations

Bryant, William B., Wei Zhang, Weiping Han, et al.. (2023). CRISPR-Cas9 Long-Read Sequencing for Mapping Transgenes in the Mouse Genome. The CRISPR Journal. 6(2). 163–175. 7 indexed citations

Nagaraja, Raghavendra Y., David M. Sherry, Jennifer L. Fessler, et al.. (2021). W246G Mutant ELOVL4 Impairs Synaptic Plasticity in Parallel and Climbing Fibers and Causes Motor Defects in a Rat Model of SCA34. Molecular Neurobiology. 58(10). 4921–4943. 12 indexed citations

Lee, Young Il, Yun Gi Kim, Jin Chul Ahn, et al.. (2019). Dysregulation of the SNARE-binding protein Munc18-1 impairs BDNF secretion and synaptic neurotransmission: a novel interventional target to protect the aging brain. GeroScience. 41(2). 109–123. 12 indexed citations

Sherry, David M., et al.. (2017). Distribution of ELOVL4 in the Developing and Adult Mouse Brain. Frontiers in Neuroanatomy. 11. 38–38. 28 indexed citations

Deák, F., Lajos Mátés, Éva Korpos, et al.. (2014). Extracellular matrilin-2 deposition controls the myogenic program timing during muscle regeneration. Journal of Cell Science. 127(Pt 15). 3240–56. 18 indexed citations

Liu, Chia‐Chen, F. Deák, Justin Rogers, et al.. (2014). Deficiency in LRP6-Mediated Wnt Signaling Contributes to Synaptic Abnormalities and Amyloid Pathology in Alzheimer’s Disease. Neuron. 84(1). 63–77. 163 indexed citations

Deák, F. & William E. Sonntag. (2012). Aging, Synaptic Dysfunction, and Insulin-Like Growth Factor (IGF)-1. The Journals of Gerontology Series A. 67A(6). 611–625. 127 indexed citations

Szabó, Liliána, Éva Korpos, Enkhjargal Batmunkh, et al.. (2008). Expression of Matrilin-2 in Liver Cirrhosis and Hepatocellular Carcinoma. Pathology & Oncology Research. 14(1). 15–22. 27 indexed citations

10.

Bronk, Peter, F. Deák, Michael C. Wilson, et al.. (2007). Differential Effects of SNAP-25 Deletion on Ca²⁺-Dependent and Ca²⁺-Independent Neurotransmission. Journal of Neurophysiology. 98(2). 794–806. 101 indexed citations

11.

Deák, F., et al.. (2006). Structural Determinants of Synaptobrevin 2 Function in Synaptic Vesicle Fusion. Journal of Neuroscience. 26(25). 6668–6676. 119 indexed citations

12.

Korpos, Éva, Andrea Molnár, Péter Papp, et al.. (2005). Expression pattern of matrilins and other extracellular matrix proteins characterize distinct stages of cell differentiation during antler development. Matrix Biology. 24(2). 124–135. 19 indexed citations

13.

Sara, Yıldırım, Tuhin Virmani, F. Deák, Xinran Liu, & Ege T. Kavalali. (2005). An Isolated Pool of Vesicles Recycles at Rest and Drives Spontaneous Neurotransmission. Neuron. 45(4). 563–573. 309 indexed citations

14.

Mátés, Lajos, Claudia M. Nicolae, Matthias Mörgelin, et al.. (2004). Mice lacking the extracellular matrix adaptor protein matrilin-2 develop without obvious abnormalities. Matrix Biology. 23(3). 195–204. 33 indexed citations

15.

Mátés, Lajos, Éva Korpos, F. Deák, et al.. (2002). Comparative analysis of the mouse and human genes (Matn2 and MATN2) for matrilin-2, a filament-forming protein widely distributed in extracellular matrices. Matrix Biology. 21(2). 163–174. 14 indexed citations

16.

Schoch, Susanne, F. Deák, Andreas Königstorfer, et al.. (2001). SNARE Function Analyzed in Synaptobrevin/VAMP Knockout Mice. Science. 294(5544). 1117–1122. 503 indexed citations breakdown →

17.

Horváth, Á., A. Galonsky, K. Ieki, et al.. (2001). Cross section for the astrophysical ^14C(n,γ )^15C reaction via the inverse reaction. APS. 46(2).

18.

Segat, Daniela, Christian Frie, Andreas R. Klatt, et al.. (2000). Expression of matrilin-1, -2 and -3 in developing mouse limbs and heart. Matrix Biology. 19(7). 649–655. 31 indexed citations

19.

Piecha, Dorothea, Selen C. Muratoglu, Matthias Mörgelin, et al.. (1999). Matrilin-2, a Large, Oligomeric Matrix Protein, Is Expressed by a Great Variety of Cells and Forms Fibrillar Networks. Journal of Biological Chemistry. 274(19). 13353–13361. 72 indexed citations

20.

Deák, F., Gábor Nagy, Péter Várnai, Emı́lia Madarász, & András Spät. (1998). Calcium current activated by potassium ions in voltage‐clamped rat hippocampal pyramidal neurones. The Journal of Physiology. 508(3). 735–745. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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