Ezra Wiater

2.5k total citations
24 papers, 1.9k citations indexed

About

Ezra Wiater is a scholar working on Molecular Biology, Oncology and Surgery. According to data from OpenAlex, Ezra Wiater has authored 24 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 6 papers in Oncology and 3 papers in Surgery. Recurrent topics in Ezra Wiater's work include TGF-β signaling in diseases (16 papers), Metabolism, Diabetes, and Cancer (6 papers) and Kruppel-like factors research (6 papers). Ezra Wiater is often cited by papers focused on TGF-β signaling in diseases (16 papers), Metabolism, Diabetes, and Cancer (6 papers) and Kruppel-like factors research (6 papers). Ezra Wiater collaborates with scholars based in United States, Switzerland and Japan. Ezra Wiater's co-authors include Wylie Vale, Peter C. Gray, Senyon Choe, Louise M. Bilezikjian, Kathleen Lewis, Jason Greenwald, Jay C. Groppe, Witek Kwiatkowski, Leigh MacConell and Amy L. Blount and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Ezra Wiater

24 papers receiving 1.9k citations

Peers

Countries citing papers authored by Ezra Wiater

Since Specialization

Citations

This map shows the geographic impact of Ezra Wiater's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ezra Wiater with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ezra Wiater more than expected).

Fields of papers citing papers by Ezra Wiater

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ezra Wiater. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ezra Wiater. The network helps show where Ezra Wiater may publish in the future.

Co-authorship network of co-authors of Ezra Wiater

This figure shows the co-authorship network connecting the top 25 collaborators of Ezra Wiater. A scholar is included among the top collaborators of Ezra Wiater based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ezra Wiater. Ezra Wiater is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	Activation of the adipocyte CREB/CRTC pathway in obesity	Communications Biology	Young‐Sil Yoon, Weiyi Liu et al.	25
2	Crtc modulates fasting programs associated with 1-C metabolism and inhibition of insulin signaling	Proceedings of the National Academy of Sciences	Ezra Wiater, Xinmin Zhang et al.	7
3	CREB Promotes Beta Cell Gene Expression by Targeting Its Coactivators to Tissue-Specific Enhancers	Molecular and Cellular Biology	Sam Van de Velde, Ezra Wiater et al.	24
4	The CREB coactivator CRTC2 promotes oncogenesis in LKB1-mutant non–small cell lung cancer	Science Advances	Laura Rodón, Robert Svensson et al.	33
5	cAMP-inducible coactivator CRTC3 attenuates brown adipose tissue thermogenesis	Proceedings of the National Academy of Sciences	Young‐Sil Yoon, Wen‐Wei Tsai et al.	25
6	CREB coactivators CRTC2 and CRTC3 modulate bone marrow hematopoiesis	Proceedings of the National Academy of Sciences	Jeong‐Ho Kim, Susan Hedrick et al.	15
7	Roles of activin family in pancreatic development and homeostasis	Molecular and Cellular Endocrinology	Ezra Wiater, Wylie Vale	19
8	Cell-type specific modulation of pituitary cells by activin, inhibin and follistatin	Molecular and Cellular Endocrinology	Louise M. Bilezikjian, Nicholas J. Justice et al.	32
9	Inhibin-A Antagonizes TGFβ2 Signaling by Down-Regulating Cell Surface Expression of the TGFβ Coreceptor Betaglycan	Molecular Endocrinology	Brendan D. Looyenga, Ezra Wiater et al.	35
10	4 Activins and Inhibins	Cold Spring Harbor Monograph Archive	Ezra Wiater, Wylie Vale	11
11	Identification of Distinct Inhibin and Transforming Growth Factor β-binding Sites on Betaglycan	Journal of Biological Chemistry	Ezra Wiater, Craig A. Harrison et al.	65
12	Activins and Inhibins and Their Signaling	Annals of the New York Academy of Sciences	Wylie Vale, Ezra Wiater et al.	69
13	Modulation of activin and BMP signaling	Molecular and Cellular Endocrinology	Craig A. Harrison, Ezra Wiater et al.	34
14	The BMP7/ActRII Extracellular Domain Complex Provides New Insights into the Cooperative Nature of Receptor Assembly	Molecular Cell	Jason Greenwald, Jay C. Groppe et al.	221
15	Adenovirus–Mediated Overexpression of Follistatin Enlarges Intact Liver of Adult Rats	Hepatology	Kazuaki Takabe, Lili Wang et al.	35
16	STRUCTURAL BASIS OF BMP SIGNALING INHIBITION BY NOGGIN, A NOVEL TWELVE-MEMBERED CYSTINE KNOT PROTEIN	Journal of Bone and Joint Surgery	Jay C. Groppe, Jason Greenwald et al.	73
17	Inhibin Is an Antagonist of Bone Morphogenetic Protein Signaling	Journal of Biological Chemistry	Ezra Wiater, Wylie Vale	139
18	Residues in the C-terminal region of activin A determine specificity for follistatin and type II receptor binding	Journal of Endocrinology	Cindy Donaldson, Ezra Wiater et al.	18
19	Structural basis of BMP signalling inhibition by the cystine knot protein Noggin	Nature	Jay C. Groppe, Jason Greenwald et al.	421
20	Betaglycan binds inhibin and can mediate functional antagonism of activin signalling	Nature	Kathleen Lewis, Peter C. Gray et al.	473

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact