Eva‐Pia Reich

685 total citations
11 papers, 609 citations indexed

About

Eva‐Pia Reich is a scholar working on Immunology, Genetics and Surgery. According to data from OpenAlex, Eva‐Pia Reich has authored 11 papers receiving a total of 609 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 5 papers in Genetics and 3 papers in Surgery. Recurrent topics in Eva‐Pia Reich's work include Diabetes and associated disorders (5 papers), T-cell and B-cell Immunology (5 papers) and Immune Cell Function and Interaction (5 papers). Eva‐Pia Reich is often cited by papers focused on Diabetes and associated disorders (5 papers), T-cell and B-cell Immunology (5 papers) and Immune Cell Function and Interaction (5 papers). Eva‐Pia Reich collaborates with scholars based in United States and Germany. Eva‐Pia Reich's co-authors include Charles A. Janeway, Robert S. Sherwin, Osami Kanagawa, Loretta A. Bober, Charles A. Lunn, Paul J. Zavodny, Joseph A. Kozlowski, Long Cui, Mary Ann Cox and Shu‐Cheng Chen and has published in prestigious journals such as Nature, Diabetes and European Journal of Immunology.

In The Last Decade

Eva‐Pia Reich

11 papers receiving 596 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Eva‐Pia Reich United States 9 283 277 190 121 109 11 609
Sofia Mayans Sweden 11 247 0.9× 231 0.8× 199 1.0× 110 0.9× 174 1.6× 15 659
Kinori Kosaka Japan 15 230 0.8× 227 0.8× 303 1.6× 312 2.6× 166 1.5× 40 861
Henry J. DeAizpurua Australia 14 148 0.5× 463 1.7× 424 2.2× 285 2.4× 247 2.3× 18 783
Heshan Peiris Australia 15 96 0.3× 263 0.9× 410 2.2× 199 1.6× 285 2.6× 19 777
Isabelle Millard Belgium 8 67 0.2× 167 0.6× 232 1.2× 90 0.7× 198 1.8× 9 477
Chantal Samson France 12 67 0.2× 226 0.8× 150 0.8× 125 1.0× 303 2.8× 13 592
Melissa P. Allen United States 8 250 0.9× 105 0.4× 60 0.3× 46 0.4× 313 2.9× 9 652
Wanda E. Filipiak United States 8 77 0.3× 234 0.8× 85 0.4× 36 0.3× 407 3.7× 11 906
Laurie J. Kaler United States 8 357 1.3× 101 0.4× 17 0.1× 42 0.3× 106 1.0× 8 731
Byoung‐Hoon Min South Korea 14 83 0.3× 278 1.0× 446 2.3× 97 0.8× 348 3.2× 42 856

Countries citing papers authored by Eva‐Pia Reich

Since Specialization
Citations

This map shows the geographic impact of Eva‐Pia Reich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva‐Pia Reich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva‐Pia Reich more than expected).

Fields of papers citing papers by Eva‐Pia Reich

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva‐Pia Reich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva‐Pia Reich. The network helps show where Eva‐Pia Reich may publish in the future.

Co-authorship network of co-authors of Eva‐Pia Reich

This figure shows the co-authorship network connecting the top 25 collaborators of Eva‐Pia Reich. A scholar is included among the top collaborators of Eva‐Pia Reich based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva‐Pia Reich. Eva‐Pia Reich is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Jenh, Chung‐Her, Mary Ann Cox, Long Cui, et al.. (2012). A selective and potent CXCR3 antagonist SCH 546738 attenuates the development of autoimmune diseases and delays graft rejection. BMC Immunology. 13(1). 2–2. 74 indexed citations
2.
Cox, Mary Ann, Long Cui, Eva‐Pia Reich, et al.. (2012). A selective and potent CXCR3 antagonist SCH 546738 attenuates the development of autoimmune diseases and delays graft rejection. BMC Immunology. 13(1). 2–2. 73 indexed citations
3.
Lunn, Charles A., Eva‐Pia Reich, & Loretta A. Bober. (2006). Targeting the CB2receptor for immune modulation. Expert Opinion on Therapeutic Targets. 10(5). 653–663. 56 indexed citations
4.
Reich, Eva‐Pia, Long Cui, Catherine Pugliese‐Sivo, et al.. (2005). Blocking ion channel KCNN4 alleviates the symptoms of experimental autoimmune encephalomyelitis in mice. European Journal of Immunology. 35(4). 1027–1036. 78 indexed citations
5.
Baumgart, Trageen, Nanette Solvason, Maureen Howard, et al.. (1999). A Conformationally-Constrained MHC Class II I-Ag7-Derived Peptide Protects NOD Mice from the Development of Diabetes. Journal of Autoimmunity. 12(4). 233–242. 11 indexed citations
6.
Reich, Eva‐Pia. (1998). Intervention for diabetes prevention at neonatal age. Diabetes/Metabolism Reviews. 14(1). 107–108. 1 indexed citations
7.
Guerder, Sylvie, et al.. (1995). Expression of the Co-stimulator Molecule B7–1 in Pancreatic β-Cells Accelerates Diabetes in the NOD Mouse. Diabetes. 44(3). 326–329. 61 indexed citations
8.
Janeway, Charles A., et al.. (1993). Summary of Talk in Orlando Protection Against Insulin-Dependent Diabetes Mellitus. Autoimmunity. 15(sup1). 4–5. 2 indexed citations
9.
Reich, Eva‐Pia, Robert S. Sherwin, Osami Kanagawa, & Charles A. Janeway. (1991). Correction: An explanation for the protective effect of the MHC class II I–E molecule in murine diabetes. Nature. 352(6330). 88–88. 13 indexed citations
10.
Reich, Eva‐Pia, Robert S. Sherwin, Osami Kanagawa, & Charles A. Janeway. (1989). An explanation for the protective effect of the MHC class II I–E molecule in murine diabetes. Nature. 341(6240). 326–328. 186 indexed citations
11.
Reich, Eva‐Pia, et al.. (1989). Prevention of Diabetes in NOD Mice by Injection of Autoreactive T-Lymphocytes. Diabetes. 38(12). 1647–1651. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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