Eva‐Lena Stattin

1.2k total citations
41 papers, 699 citations indexed

About

Eva‐Lena Stattin is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Genetics. According to data from OpenAlex, Eva‐Lena Stattin has authored 41 papers receiving a total of 699 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 17 papers in Cardiology and Cardiovascular Medicine and 17 papers in Genetics. Recurrent topics in Eva‐Lena Stattin's work include Cardiac electrophysiology and arrhythmias (13 papers), Ion channel regulation and function (10 papers) and Connective tissue disorders research (7 papers). Eva‐Lena Stattin is often cited by papers focused on Cardiac electrophysiology and arrhythmias (13 papers), Ion channel regulation and function (10 papers) and Connective tissue disorders research (7 papers). Eva‐Lena Stattin collaborates with scholars based in Sweden, South Africa and United States. Eva‐Lena Stattin's co-authors include Susanne Rudberg, Gisela Dahlquist, Annika Rydberg, Steen M. Jensen, Annika Winbo, Niklas Dahl, Anna Möllsten, Yelverton Tegner, Johan Persson and Magnus Domellöf and has published in prestigious journals such as PLoS ONE, Diabetes Care and Scientific Reports.

In The Last Decade

Eva‐Lena Stattin

39 papers receiving 676 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Eva‐Lena Stattin Sweden 17 245 242 208 83 70 41 699
Masaru Inoue Japan 20 220 0.9× 363 1.5× 133 0.6× 119 1.4× 23 0.3× 40 868
Cheol Woo Ko South Korea 13 244 1.0× 46 0.2× 166 0.8× 136 1.6× 129 1.8× 50 680
Paulo José Lorenzoni Brazil 18 367 1.5× 63 0.3× 78 0.4× 112 1.3× 30 0.4× 92 968
Anabelle Opazo Saez Germany 15 174 0.7× 260 1.1× 84 0.4× 90 1.1× 33 0.5× 23 756
Taku Shirakawa Japan 12 193 0.8× 75 0.3× 51 0.2× 65 0.8× 48 0.7× 33 463
María José Acuña Chile 14 386 1.6× 156 0.6× 91 0.4× 81 1.0× 10 0.1× 22 714
Katsumi Goji Japan 14 324 1.3× 63 0.3× 209 1.0× 88 1.1× 34 0.5× 25 594
Liby Mathew United States 10 364 1.5× 162 0.7× 47 0.2× 53 0.6× 66 0.9× 11 596
Boris Utsch Germany 20 596 2.4× 32 0.1× 391 1.9× 136 1.6× 116 1.7× 32 951
Yuriko Fujinaga Japan 6 352 1.4× 38 0.2× 64 0.3× 57 0.7× 99 1.4× 6 499

Countries citing papers authored by Eva‐Lena Stattin

Since Specialization
Citations

This map shows the geographic impact of Eva‐Lena Stattin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva‐Lena Stattin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva‐Lena Stattin more than expected).

Fields of papers citing papers by Eva‐Lena Stattin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva‐Lena Stattin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva‐Lena Stattin. The network helps show where Eva‐Lena Stattin may publish in the future.

Co-authorship network of co-authors of Eva‐Lena Stattin

This figure shows the co-authorship network connecting the top 25 collaborators of Eva‐Lena Stattin. A scholar is included among the top collaborators of Eva‐Lena Stattin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva‐Lena Stattin. Eva‐Lena Stattin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ameur, Adam, Ignas Bunikis, Eva‐Lena Stattin, et al.. (2024). Long-read sequencing and optical mapping generates near T2T assemblies that resolves a centromeric translocation. Scientific Reports. 14(1). 9000–9000. 3 indexed citations
2.
Svennblad, Bodil, et al.. (2022). Symptoms and ECG changes precede sudden cardiac death in hypertrophic cardiomyopathy—A nationwide study among the young in Sweden. PLoS ONE. 17(9). e0273567–e0273567. 5 indexed citations
3.
Stattin, Eva‐Lena, Emil Hagström, Niklas Dahl, et al.. (2022). Cohort profile: the Swedish study of SUDden cardiac Death in the Young (SUDDY) 2000–2010: a complete nationwide cohort of SCDs. BMJ Open. 12(5). e055557–e055557. 5 indexed citations
4.
Cięszczyk, Paweł, Krzysztof Ficek, Kinga Humińska‐Lisowska, et al.. (2021). Investigation of multiple populations highlight VEGFA polymorphisms to modulate anterior cruciate ligament injury. Journal of Orthopaedic Research®. 40(7). 1604–1612. 8 indexed citations
6.
Thuresson, Ann‐Charlotte, et al.. (2020). A novel heterozygous variant in FGF9 associated with previously unreported features of multiple synostosis syndrome 3. Clinical Genetics. 99(2). 325–329. 7 indexed citations
7.
Wanhainen, Anders, et al.. (2020). Celiprolol Treatment in Patients with Vascular Ehlers-Danlos Syndrome. European Journal of Vascular and Endovascular Surgery. 61(2). 326–331. 28 indexed citations
8.
Körberg, Izabella Baranowska, Daniel Nowinski, Marie-Louise Bondeson, et al.. (2020). A progressive and complex clinical course in two family members with ERF-related craniosynostosis: a case report. BMC Medical Genetics. 21(1). 90–90. 10 indexed citations
9.
Taylan, Fulya, Inger Nennesmo, Göran Annerén, et al.. (2019). Early activating somatic PIK3CA mutations promote ectopic muscle development and upper limb overgrowth. Clinical Genetics. 96(2). 118–125. 9 indexed citations
10.
Ponzetti, Marco, Michael Posthumus, Charlotte K. Häger, et al.. (2019). Functional polymorphisms within the inflammatory pathway regulate expression of extracellular matrix components in a genetic risk dependent model for anterior cruciate ligament injuries. Journal of science and medicine in sport. 22(11). 1219–1225. 18 indexed citations
11.
Stattin, Eva‐Lena, Joakim Klar, Adam Ameur, et al.. (2016). A novel variant in MYLK causes thoracic aortic dissections: genotypic and phenotypic description. BMC Medical Genetics. 17(1). 61–61. 22 indexed citations
13.
Stattin, Eva‐Lena, Annika Winbo, Kristina Cederquist, et al.. (2012). Founder mutations characterise the mutation panorama in 200 Swedish index cases referred for Long QT syndrome genetic testing. BMC Cardiovascular Disorders. 12(1). 95–95. 22 indexed citations
14.
Jensen, Steen M., et al.. (2012). Vectorcardiographic Recordings of the Q-T Interval in a Pediatric Long Q-T Syndrome Population. Pediatric Cardiology. 34(2). 245–249. 7 indexed citations
15.
Wentzel, Christian, Sally Ann Lynch, Eva‐Lena Stattin, et al.. (2010). Interstitial Deletions at 6q14.1–q15 Associated with Obesity, Developmental Delay and a Distinct Clinical Phenotype. Molecular Syndromology. 1(2). 75–81. 25 indexed citations
16.
Winbo, Annika, et al.. (2010). Origin of the Swedish long QT syndrome Y111C/KCNQ1 founder mutation. Heart Rhythm. 8(4). 541–547. 21 indexed citations
17.
Entesarian, Miriam, Birgit Carlsson, Mahmoud Mansouri, et al.. (2009). A chromosome 10 variant with a 12 Mb inversion [inv(10)(q11.22q21.1)] identical by descent and frequent in the Swedish population. American Journal of Medical Genetics Part A. 149A(3). 380–386. 11 indexed citations
18.
Hilton, Emma, Forbes Manson, Jill Urquhart, et al.. (2007). Left-sided embryonic expression of the BCL-6 corepressor, BCOR, is required for vertebrate laterality determination. Human Molecular Genetics. 16(14). 1773–1782. 40 indexed citations
19.
Timby, Niklas, et al.. (2007). Early onset autosomal dominant spinocerebellar ataxia with miosis: Four cases. European Journal of Paediatric Neurology. 12(1). 38–40. 4 indexed citations
20.
Dahlquist, Gisela, Eva‐Lena Stattin, & Susanne Rudberg. (2001). Urinary albumin excretion rate and glomerular filtration rate in the prediction of diabetic nephropathy; a long‐term follow‐up study of childhood onset type‐1 diabetic patients. Nephrology Dialysis Transplantation. 16(7). 1382–1386. 85 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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