Étienne Jacotot

2.3k total citations · 1 hit paper
37 papers, 1.7k citations indexed

About

Étienne Jacotot is a scholar working on Molecular Biology, Oncology and Virology. According to data from OpenAlex, Étienne Jacotot has authored 37 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 11 papers in Oncology and 9 papers in Virology. Recurrent topics in Étienne Jacotot's work include Cell death mechanisms and regulation (10 papers), Peptidase Inhibition and Analysis (10 papers) and HIV Research and Treatment (9 papers). Étienne Jacotot is often cited by papers focused on Cell death mechanisms and regulation (10 papers), Peptidase Inhibition and Analysis (10 papers) and HIV Research and Treatment (9 papers). Étienne Jacotot collaborates with scholars based in France, United States and United Kingdom. Étienne Jacotot's co-authors include Ara G. Hovanessian, Christian Callebaut, Bernard Krust, Henrik Hagberg, Carina Mallard, Pierre Gressèns, Sophie Lebon, Julien Josserand, Bobbi Fleiss and Vincent Degos and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Étienne Jacotot

37 papers receiving 1.7k citations

Hit Papers

Characterization of phenotype markers and neuronotoxic po... 2013 2026 2017 2021 2013 100 200 300 400 500

Peers

Étienne Jacotot
Shongshan Fan United States
Claudio Torres United States
Meng-Liang Zhao United States
Shongshan Fan United States
Étienne Jacotot
Citations per year, relative to Étienne Jacotot Étienne Jacotot (= 1×) peers Shongshan Fan

Countries citing papers authored by Étienne Jacotot

Since Specialization
Citations

This map shows the geographic impact of Étienne Jacotot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Étienne Jacotot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Étienne Jacotot more than expected).

Fields of papers citing papers by Étienne Jacotot

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Étienne Jacotot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Étienne Jacotot. The network helps show where Étienne Jacotot may publish in the future.

Co-authorship network of co-authors of Étienne Jacotot

This figure shows the co-authorship network connecting the top 25 collaborators of Étienne Jacotot. A scholar is included among the top collaborators of Étienne Jacotot based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Étienne Jacotot. Étienne Jacotot is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Soratroi, Claudia, Joel S. Riley, Florian Bellutti, et al.. (2024). Caspase-2 kills cells with extra centrosomes. Science Advances. 10(44). eado6607–eado6607. 3 indexed citations
2.
Kim, Ju Youn, Valentina C. Sladky, Tae Gyu Oh, et al.. (2022). PIDDosome-SCAP crosstalk controls high-fructose-diet-dependent transition from simple steatosis to steatohepatitis. Cell Metabolism. 34(10). 1548–1560.e6. 24 indexed citations
3.
Coric, Pascale, Ju Youn Kim, Ronak Patel, et al.. (2022). Genuine selective caspase-2 inhibition with new irreversible small peptidomimetics. Cell Death and Disease. 13(11). 959–959. 7 indexed citations
4.
Jacotot, Étienne. (2020). Inhibition des caspases. médecine/sciences. 36(12). 1143–1154. 4 indexed citations
5.
Chhor, Vibol, Tifenn Le Charpentier, Sophie Lebon, et al.. (2013). Characterization of phenotype markers and neuronotoxic potential of polarised primary microglia in vitro. Brain Behavior and Immunity. 32. 70–85. 510 indexed citations breakdown →
6.
Sifringer, Marco, Ivo Bendix, Stefanie Endesfelder, et al.. (2012). Prevention of neonatal oxygen-induced brain damage by reduction of intrinsic apoptosis. Cell Death and Disease. 3(1). e250–e250. 34 indexed citations
7.
Carlsson, Ylva, Xiaoyang Wang, Leslie Schwendimann, et al.. (2012). Combined effect of hypothermia and caspase-2 gene deficiency on neonatal hypoxic–ischemic brain injury. Pediatric Research. 71(5). 566–572. 25 indexed citations
8.
Carlsson, Ylva, Leslie Schwendimann, Regina Vontell, et al.. (2011). Genetic inhibition of caspase‐2 reduces hypoxic‐ischemic and excitotoxic neonatal brain injury. Annals of Neurology. 70(5). 781–789. 49 indexed citations
9.
Wang, Xiaoyang, Anna‐Lena Leverin, Wei Han, et al.. (2011). Isolation of brain mitochondria from neonatal mice. Journal of Neurochemistry. 119(6). 1253–1261. 24 indexed citations
10.
Bouët, Valentine, Thomas Fréret, Madeleine Bezault, et al.. (2010). Predicting sensorimotor and memory deficits after neonatal ischemic stroke with reperfusion in the rat. Behavioural Brain Research. 212(1). 56–63. 21 indexed citations
11.
Dzietko, Mark, Marco Sifringer, Bettina Gerstner, et al.. (2008). A critical role for Fas/CD‐95 dependent signaling pathways in the pathogenesis of hyperoxia‐induced brain injury. Annals of Neurology. 64(6). 664–673. 36 indexed citations
12.
Borgne‐Sanchez, Annie, Sylvain Dupont, Alain Langonné, et al.. (2006). Targeted Vpr-derived peptides reach mitochondria to induce apoptosis of αVβ3-expressing endothelial cells. Cell Death and Differentiation. 14(3). 422–435. 41 indexed citations
13.
Chauvier, David, Hervé Lecœur, Alain Langonné, et al.. (2005). Upstream control of apoptosis by caspase-2 in serum-deprived primary neurons. APOPTOSIS. 10(6). 1243–1259. 30 indexed citations
14.
Blanco, Julià, Christian Callebaut, Étienne Jacotot, et al.. (1998). Dipeptidyl‐peptidase IV‐β. European Journal of Biochemistry. 256(2). 369–378. 24 indexed citations
15.
Jacotot, Étienne, Bernard Krust, Christian Callebaut, et al.. (1997). HIV-1 envelope glycoproteins-mediated apoptosis is regulated by CD4 dependent and independent mechanisms. APOPTOSIS. 2(1). 47–60. 23 indexed citations
16.
Valenzuela-Fernández, Agustı́n, Julià Blanco, Christian Callebaut, et al.. (1997). HIV-1 Envelope gp120 and Viral Particles Block Adenosine Deaminase Binding to Human CD26. Advances in experimental medicine and biology. 421. 185–192. 6 indexed citations
17.
Callebaut, Christian, Étienne Jacotot, Julià Blanco, Bernard Krust, & Ara G. Hovanessian. (1997). The Level of CD26 Determines the Rate of HIV Entry in a CD4+ T-Cell Line. Advances in experimental medicine and biology. 421. 179–184. 1 indexed citations
18.
Gougeon, Marie‐Lise, Hervé Lecœur, Christian Callebaut, et al.. (1996). Selective loss of the CD4+/CD26+ T-cell subset during HIV infection. Research in Immunology. 147(1). 5–8. 16 indexed citations
19.
Jacotot, Étienne, Christian Callebaut, Julià Blanco, et al.. (1996). Dipeptidyl‐Peptidase IV‐β, a Novel form of Cell‐Surface‐Expressed Protein with Dipeptidyl‐Peptidase IV Activity. European Journal of Biochemistry. 239(2). 248–258. 44 indexed citations
20.
Jacotot, Étienne, Christian Callebaut, Julià Blanco, et al.. (1996). HIV Envelope Glycoprotein-Induced Cell Killing by Apoptosis Is Enhanced with Increased Expression of CD26 in CD4+T Cells. Virology. 223(2). 318–330. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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