Ervin Valk

1.9k total citations
28 papers, 1.3k citations indexed

About

Ervin Valk is a scholar working on Molecular Biology, Cell Biology and Oncology. According to data from OpenAlex, Ervin Valk has authored 28 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 15 papers in Cell Biology and 6 papers in Oncology. Recurrent topics in Ervin Valk's work include Microtubule and mitosis dynamics (14 papers), Fungal and yeast genetics research (8 papers) and Cancer-related Molecular Pathways (6 papers). Ervin Valk is often cited by papers focused on Microtubule and mitosis dynamics (14 papers), Fungal and yeast genetics research (8 papers) and Cancer-related Molecular Pathways (6 papers). Ervin Valk collaborates with scholars based in Estonia, United States and Netherlands. Ervin Valk's co-authors include Mart Loog, Rainis Venta, Martin Lepiku, Mardo Kõivomägi, Arnoud van der Laarse, Wilhelmina H. Bax, David O. Morgan, Mihkel Örd, Martin J. Schalij and Douwe E. Atsma and has published in prestigious journals such as Nature, Cell and Nature Communications.

In The Last Decade

Ervin Valk

28 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ervin Valk Estonia 16 735 400 380 223 156 28 1.3k
Ke Yang China 21 823 1.1× 135 0.3× 48 0.1× 47 0.2× 156 1.0× 55 1.3k
Pengxiu Cao China 21 659 0.9× 95 0.2× 168 0.4× 55 0.2× 92 0.6× 47 1.2k
Nikolai Tomilin Russia 22 1.1k 1.5× 208 0.5× 123 0.3× 138 0.6× 170 1.1× 42 1.5k
Gary A. Martin United States 11 812 1.1× 156 0.4× 65 0.2× 81 0.4× 71 0.5× 16 1.3k
Jean‐Claude Cavadore France 18 930 1.3× 91 0.2× 445 1.2× 88 0.4× 128 0.8× 28 1.3k
Alexander A. Stewart United States 12 948 1.3× 71 0.2× 213 0.6× 37 0.2× 162 1.0× 19 1.3k
Laxmikanth Kollipara Germany 15 674 0.9× 32 0.1× 135 0.4× 35 0.2× 107 0.7× 40 982
Qinghua Wang China 20 1.3k 1.8× 55 0.1× 314 0.8× 44 0.2× 156 1.0× 57 1.6k
Takeshi Mizuno Japan 17 763 1.0× 65 0.2× 103 0.3× 99 0.4× 141 0.9× 41 950
Wang Zheng China 19 777 1.1× 161 0.4× 108 0.3× 67 0.3× 80 0.5× 41 1.4k

Countries citing papers authored by Ervin Valk

Since Specialization
Citations

This map shows the geographic impact of Ervin Valk's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ervin Valk with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ervin Valk more than expected).

Fields of papers citing papers by Ervin Valk

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ervin Valk. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ervin Valk. The network helps show where Ervin Valk may publish in the future.

Co-authorship network of co-authors of Ervin Valk

This figure shows the co-authorship network connecting the top 25 collaborators of Ervin Valk. A scholar is included among the top collaborators of Ervin Valk based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ervin Valk. Ervin Valk is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Valk, Ervin, Mihkel Örd, Ilona Faustova, & Mart Loog. (2023). CDK signaling via nonconventional CDK phosphorylation sites. Molecular Biology of the Cell. 34(12). 8–8. 8 indexed citations
3.
Venta, Rainis, Ervin Valk, Mihkel Örd, et al.. (2020). A processive phosphorylation circuit with multiple kinase inputs and mutually diversional routes controls G1/S decision. Nature Communications. 11(1). 1836–1836. 15 indexed citations
4.
Örd, Mihkel, Kaidi Möll, Ilona Faustova, et al.. (2020). Proline-Rich Motifs Control G2-CDK Target Phosphorylation and Priming an Anchoring Protein for Polo Kinase Localization. Cell Reports. 31(11). 107757–107757. 19 indexed citations
5.
Valk, Ervin, et al.. (2019). Synthetic-Evolution Reveals Narrow Paths to Regulation of the Saccharomyces cerevisiae Mitotic Kinesin-5 Cin8. International Journal of Biological Sciences. 15(6). 1125–1138. 5 indexed citations
6.
Örd, Mihkel, Rainis Venta, Kaidi Möll, Ervin Valk, & Mart Loog. (2019). Cyclin-Specific Docking Mechanisms Reveal the Complexity of M-CDK Function in the Cell Cycle. Molecular Cell. 75(1). 76–89.e3. 47 indexed citations
7.
Örd, Mihkel, Kaidi Möll, Ilona Faustova, et al.. (2019). Multisite phosphorylation code of CDK. Nature Structural & Molecular Biology. 26(7). 649–658. 60 indexed citations
8.
Judah, Haim, et al.. (2017). Three Cdk1 sites in the kinesin-5 Cin8 catalytic domain coordinate motor localization and activity during anaphase. Cellular and Molecular Life Sciences. 74(18). 3395–3412. 14 indexed citations
9.
Hõrak, Hanna, Maija Sierla, Kadri Tõldsepp, et al.. (2016). A Dominant Mutation in the HT1 Kinase Uncovers Roles of MAP Kinases and GHR1 in CO2-Induced Stomatal Closure. The Plant Cell. 28(10). 2493–2509. 79 indexed citations
10.
Doncic, Andreas, Ervin Valk, Alan Bush, et al.. (2015). Compartmentalization of a Bistable Switch Enables Memory to Cross a Feedback-Driven Transition. Cell. 160(6). 1182–1195. 37 indexed citations
11.
Valk, Ervin, et al.. (2015). A Docking Interface in the Cyclin Cln2 Promotes Multi-site Phosphorylation of Substrates and Timely Cell-Cycle Entry. Current Biology. 25(3). 316–325. 26 indexed citations
12.
Kõivomägi, Mardo, Mihkel Örd, Ervin Valk, et al.. (2013). Multisite phosphorylation networks as signal processors for Cdk1. Nature Structural & Molecular Biology. 20(12). 1415–1424. 97 indexed citations
13.
Venta, Rainis, Ervin Valk, Mardo Kõivomägi, & Mart Loog. (2012). Double-negative feedback between S-phase cyclin-CDK and CKI generates abruptness in the G1/S switch. Frontiers in Physiology. 3. 459–459. 9 indexed citations
14.
Kõivomägi, Mardo, Ervin Valk, Rainis Venta, et al.. (2011). Dynamics of Cdk1 Substrate Specificity during the Cell Cycle. Molecular Cell. 42(5). 610–623. 120 indexed citations
15.
16.
Hessel, Marleen, Etienne Michielsen, Douwe E. Atsma, et al.. (2008). Release kinetics of intact and degraded troponin I and T after irreversible cell damage. Experimental and Molecular Pathology. 85(2). 90–95. 58 indexed citations
17.
Bax, Wilhelmina H., et al.. (2005). Integrin stimulation induces calcium signalling in rat cardiomyocytes by a NO-dependent mechanism. Pflügers Archiv - European Journal of Physiology. 451(4). 588–595. 24 indexed citations
18.
Hollaar, L., et al.. (1995). Protection of myocytes against free radical-induced damage by accelerated turnover of the glutathione redox cycle. European Heart Journal. 16(4). 553–562. 33 indexed citations
19.
Hollaar, L., et al.. (1994). Desferrioxamine Protects Myocytes Against Peroxide-induced Myocyte Damage Without Affecting Glutathione Redox Cycle Turnover. Journal of Molecular and Cellular Cardiology. 26(7). 877–887. 7 indexed citations
20.
Laarse, A. van der, et al.. (1989). A method to quantitate cell numbers of muscle cells and non-muscle cells in homogenised heart cell cultures. Cardiovascular Research. 23(11). 928–933. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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