Eric J. G. Pollitt

854 total citations
12 papers, 595 citations indexed

About

Eric J. G. Pollitt is a scholar working on Molecular Biology, Infectious Diseases and Genetics. According to data from OpenAlex, Eric J. G. Pollitt has authored 12 papers receiving a total of 595 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 5 papers in Infectious Diseases and 3 papers in Genetics. Recurrent topics in Eric J. G. Pollitt's work include Bacterial biofilms and quorum sensing (5 papers), Antimicrobial Resistance in Staphylococcus (5 papers) and Evolution and Genetic Dynamics (3 papers). Eric J. G. Pollitt is often cited by papers focused on Bacterial biofilms and quorum sensing (5 papers), Antimicrobial Resistance in Staphylococcus (5 papers) and Evolution and Genetic Dynamics (3 papers). Eric J. G. Pollitt collaborates with scholars based in United Kingdom, United States and Netherlands. Eric J. G. Pollitt's co-authors include Stephen P. Diggle, Simon J. Foster, Piotr Szkuta, Shanika A. Crusz, Stuart A. West, Maxwell N. Burton-Chellew, Stephen A. Renshaw, Bas G. J. Surewaard, Paul Morris and Manli Na and has published in prestigious journals such as Development, Nature Reviews Microbiology and Scientific Reports.

In The Last Decade

Eric J. G. Pollitt

11 papers receiving 590 citations

Peers

Eric J. G. Pollitt
Armando R. Caballero United States
Madeleine G. Moule United States
Wenqi Yu Germany
Joshua R. Fletcher United States
Eleni Tsompanidou Netherlands
Doreen Hooi United Kingdom
Chungyu Chang United States
Armando R. Caballero United States
Eric J. G. Pollitt
Citations per year, relative to Eric J. G. Pollitt Eric J. G. Pollitt (= 1×) peers Armando R. Caballero

Countries citing papers authored by Eric J. G. Pollitt

Since Specialization
Citations

This map shows the geographic impact of Eric J. G. Pollitt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eric J. G. Pollitt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eric J. G. Pollitt more than expected).

Fields of papers citing papers by Eric J. G. Pollitt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eric J. G. Pollitt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eric J. G. Pollitt. The network helps show where Eric J. G. Pollitt may publish in the future.

Co-authorship network of co-authors of Eric J. G. Pollitt

This figure shows the co-authorship network connecting the top 25 collaborators of Eric J. G. Pollitt. A scholar is included among the top collaborators of Eric J. G. Pollitt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eric J. G. Pollitt. Eric J. G. Pollitt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
2.
Pollitt, Eric J. G., et al.. (2021). Lamb1a regulates atrial growth by limiting second heart field addition during zebrafish heart development. Development. 148(20). 9 indexed citations
3.
Carnell, Oliver, Joe Gray, Jacob Biboy, et al.. (2021). Staphylococcus aureus cell wall structure and dynamics during host-pathogen interaction. PLoS Pathogens. 17(3). e1009468–e1009468. 57 indexed citations
4.
Tessadori, Federico, Eric J. G. Pollitt, Aaron M. Savage, et al.. (2021). Asymmetric Hapln1a drives regionalized cardiac ECM expansion and promotes heart morphogenesis in zebrafish development. Cardiovascular Research. 118(1). 226–240. 25 indexed citations
5.
Gibson, Josie F., Grace R. Pidwill, Oliver Carnell, et al.. (2021). Commensal bacteria augment Staphylococcus aureus infection by inactivation of phagocyte-derived reactive oxygen species. PLoS Pathogens. 17(9). e1009880–e1009880. 15 indexed citations
6.
Pollitt, Eric J. G., et al.. (2018). Staphylococcus aureus infection dynamics. PLoS Pathogens. 14(6). e1007112–e1007112. 152 indexed citations
7.
Surewaard, Bas G. J., Manli Na, Ying Fei, et al.. (2018). Human skin commensals augment Staphylococcus aureus pathogenesis. Nature Microbiology. 3(8). 881–890. 84 indexed citations
8.
Pollitt, Eric J. G. & Stephen P. Diggle. (2017). Defining motility in the Staphylococci. Cellular and Molecular Life Sciences. 74(16). 2943–2958. 64 indexed citations
9.
Pollitt, Eric J. G., Shanika A. Crusz, & Stephen P. Diggle. (2015). Staphylococcus aureus forms spreading dendrites that have characteristics of active motility. Scientific Reports. 5(1). 17698–17698. 63 indexed citations
10.
Popat, Roman, Eric J. G. Pollitt, Freya Harrison, et al.. (2015). Conflict of interest and signal interference lead to the breakdown of honest signaling. Evolution. 69(9). 2371–2383. 23 indexed citations
11.
Pollitt, Eric J. G., Stuart A. West, Shanika A. Crusz, Maxwell N. Burton-Chellew, & Stephen P. Diggle. (2013). Cooperation, Quorum Sensing, and Evolution of Virulence in Staphylococcus aureus. Infection and Immunity. 82(3). 1045–1051. 90 indexed citations
12.
Youle, Merry, Forest Rohwer, Apollo Stacy, et al.. (2012). The Microbial Olympics. Nature Reviews Microbiology. 10(8). 583–588. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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