Eric de Kant

520 total citations
18 papers, 421 citations indexed

About

Eric de Kant is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Eric de Kant has authored 18 papers receiving a total of 421 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 10 papers in Oncology and 6 papers in Pathology and Forensic Medicine. Recurrent topics in Eric de Kant's work include Genetic factors in colorectal cancer (6 papers), Cancer-related Molecular Pathways (6 papers) and Drug Transport and Resistance Mechanisms (3 papers). Eric de Kant is often cited by papers focused on Genetic factors in colorectal cancer (6 papers), Cancer-related Molecular Pathways (6 papers) and Drug Transport and Resistance Mechanisms (3 papers). Eric de Kant collaborates with scholars based in Germany, Switzerland and Netherlands. Eric de Kant's co-authors include Christoph Rochlitz, Richard Herrmann, Christian Thiede, Richard Herrmann, Marco C. DeRuiter, Marit J. Boot, Heleen Lie‐Venema, Adriana C. Gittenberger–de Groot, D. Huhn and D. Huhn and has published in prestigious journals such as Circulation Research, Cancer and Journal of Lipid Research.

In The Last Decade

Eric de Kant

18 papers receiving 412 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Eric de Kant Germany 14 272 154 75 68 68 18 421
H C Hsu Taiwan 10 155 0.6× 106 0.7× 29 0.4× 71 1.0× 91 1.3× 16 419
Hanjun Qin United States 15 245 0.9× 164 1.1× 53 0.7× 60 0.9× 63 0.9× 36 561
Francis Wright United States 10 162 0.6× 168 1.1× 139 1.9× 49 0.7× 110 1.6× 21 437
Artit Jinawath Thailand 12 221 0.8× 133 0.9× 64 0.9× 68 1.0× 84 1.2× 32 451
C Rimbaut France 9 133 0.5× 75 0.5× 53 0.7× 183 2.7× 37 0.5× 26 391
Hiroyuki Maeta Japan 11 191 0.7× 146 0.9× 31 0.4× 79 1.2× 96 1.4× 24 419
Uwe-Jochen Göhring Germany 13 163 0.6× 199 1.3× 42 0.6× 105 1.5× 74 1.1× 35 531
J.P. Farcet France 9 76 0.3× 77 0.5× 63 0.8× 31 0.5× 31 0.5× 10 399
Motonori Futatsugi Japan 9 293 1.1× 246 1.6× 75 1.0× 123 1.8× 79 1.2× 13 526
Mauro Iannopollo Italy 7 133 0.5× 306 2.0× 35 0.5× 83 1.2× 111 1.6× 10 454

Countries citing papers authored by Eric de Kant

Since Specialization
Citations

This map shows the geographic impact of Eric de Kant's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eric de Kant with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eric de Kant more than expected).

Fields of papers citing papers by Eric de Kant

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eric de Kant. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eric de Kant. The network helps show where Eric de Kant may publish in the future.

Co-authorship network of co-authors of Eric de Kant

This figure shows the co-authorship network connecting the top 25 collaborators of Eric de Kant. A scholar is included among the top collaborators of Eric de Kant based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eric de Kant. Eric de Kant is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Lie‐Venema, Heleen, et al.. (2003). Ets-1 and Ets-2 Transcription Factors Are Essential for Normal Coronary and Myocardial Development in Chicken Embryos. Circulation Research. 92(7). 749–756. 78 indexed citations
2.
Dallinga‐Thie, Geesje M., et al.. (2001). Genetic heterogeneity in the apolipoprotein C-III promoter and effects of insulin. Journal of Lipid Research. 42(9). 1450–1456. 23 indexed citations
3.
Popescu, Răzvan, Andreas Lohri, Eric de Kant, et al.. (1998). bcl-2 expression is reciprocal to p53 and c-myc expression in metastatic human colorectal cancer. European Journal of Cancer. 34(8). 1268–1273. 16 indexed citations
4.
Thiede, Christian, Tanja Sonntag, Eric de Kant, et al.. (1997). The status of p53 in the metastatic progression of colorectal cancer. European Journal of Cancer. 33(8). 1314–1322. 25 indexed citations
5.
Kant, Eric de, et al.. (1996). MDR1 expression correlates with mutantp53 expression in colorectal cancer metastases. Journal of Cancer Research and Clinical Oncology. 122(11). 671–675. 16 indexed citations
6.
Rochlitz, Christoph, Richard Herrmann, & Eric de Kant. (1996). Overexpression and Amplification of c-<i>myc</i> during Progression of Human Colorectal Cancer. Oncology. 53(6). 448–454. 55 indexed citations
7.
Rochlitz, Christoph, et al.. (1995). Evidence for a mutual regulation of p53 and c-myc expression in human colorectal cancer metastases. Annals of Oncology. 6(10). 981–986. 11 indexed citations
8.
Valentijn, A. Rob P.M., et al.. (1995). Synthesis of a potential enzyme‐specific inhibitor of squalene synthase. Recueil des Travaux Chimiques des Pays-Bas. 114(7). 332–336. 8 indexed citations
9.
Thiede, Christian, et al.. (1994). Expression and mutational analysis of Nm23-H1 in liver metastases of colorectal cancer. British Journal of Cancer. 70(6). 1267–1271. 20 indexed citations
10.
Kant, Eric de, Christoph Rochlitz, & Richard Herrmann. (1994). Gene expression analysis by a competitive and differential PCR with antisense competitors.. PubMed. 17(5). 934–42. 29 indexed citations
11.
Rochlitz, Christoph, Eric de Kant, R.‐M. Böhmer, et al.. (1993). Position Specificity of Ki-ras Oncogene Mutations during the Progression of Colorectal Carcinoma. Oncology. 50(1). 70–76. 19 indexed citations
12.
Kant, Eric de, et al.. (1993). Molecular alterations in a patient with Turcot's syndrome. British Journal of Cancer. 68(3). 519–523. 10 indexed citations
13.
Neubauer, Andreas, Eric de Kant, Christoph Rochlitz, et al.. (1993). Altered expression of the retinoblastoma susceptibility gene in chronic lymphocytic leukaemia. British Journal of Haematology. 85(3). 498–503. 20 indexed citations
14.
Rochlitz, Christoph, H. Lobeck, Stefan Peter, et al.. (1992). Multiple drug resistance gene expression in human renal cell cancer is associated with the histologic subtype. Cancer. 69(12). 2993–2998. 21 indexed citations
15.
Rochlitz, Christoph, Eric de Kant, Andreas Neubauer, et al.. (1992). PCR-determined expression of the MDR1 gene in chronic lymphocytic leukemia. Annals of Hematology. 65(6). 241–246. 19 indexed citations
16.
Rochlitz, Christoph, et al.. (1992). Mutations in the ras protooncogenes are rare events in renal cell cancer. European Journal of Cancer. 28(2-3). 333–336. 21 indexed citations
17.
Peters, Godefridus J., Jorge Nadal, E. Laurensse, Eric de Kant, & Herbert M. Pinedo. (1990). Retention of in vivo antipyrimidine effects of brequinar sodium (DUP-785; NSC 368390) in murine liver, bone marrow and colon cancer. Biochemical Pharmacology. 39(1). 135–144. 25 indexed citations
18.
Kant, Eric de, Herbert M. Pinedo, E. Laurensse, & Godefridus J. Peters. (1989). The relation between inhibition of cell growth and of dihydroorotic acid dehydrogenase by Brequinar Sodium. Cancer Letters. 46(2). 123–127. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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