Emma Langley

830 total citations · 1 hit paper
14 papers, 507 citations indexed

About

Emma Langley is a scholar working on Oncology, Molecular Biology and Cancer Research. According to data from OpenAlex, Emma Langley has authored 14 papers receiving a total of 507 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Oncology, 6 papers in Molecular Biology and 6 papers in Cancer Research. Recurrent topics in Emma Langley's work include Cancer Cells and Metastasis (3 papers), Pancreatic and Hepatic Oncology Research (3 papers) and Cancer Genomics and Diagnostics (3 papers). Emma Langley is often cited by papers focused on Cancer Cells and Metastasis (3 papers), Pancreatic and Hepatic Oncology Research (3 papers) and Cancer Genomics and Diagnostics (3 papers). Emma Langley collaborates with scholars based in United States, United Kingdom and France. Emma Langley's co-authors include Jean‐Marc Reichhart, David Gubb, Elena A. Levashina, Clare Green, Jules A. Hoffmann, Michael Ashburner, Isabelle Groß, Philippe Georgel, Jules A. Hoffmann and Émmanuelle Nicolas and has published in prestigious journals such as Science, Nucleic Acids Research and Journal of Clinical Oncology.

In The Last Decade

Emma Langley

12 papers receiving 498 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Constitutive Activation of Toll-Mediated Antifungal Defense in Serpin-Deficient Drosophila

1999 369 citations Elena A. Levashina, Emma Langley et al. Science profile →

Peers

Emma Langley

IMMUN

CMN

MICRO

József Antal Hungary

Bo Gustav Lindberg Sweden

M. Otte Germany

Sunny Zachariah United States

Xiaomeng An China

Daniele Chaves‐Moreira Brazil

Sharissa L. Latham Australia

Srdjan M. Dragovic United States

Shoji Iwagami Japan

Randolf J. Kerschbaumer Austria

József Antal Hungary

Emma Langley

214 ×0.6

IMMUN

137 ×0.7

31 ×0.2

35 ×0.3

CMN

15 ×0.2

MICRO

Citations per year, relative to Emma Langley Emma Langley (= 1×) peers József Antal

Countries citing papers authored by Emma Langley

Since Specialization

Citations

This map shows the geographic impact of Emma Langley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emma Langley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emma Langley more than expected).

Fields of papers citing papers by Emma Langley

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Emma Langley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emma Langley. The network helps show where Emma Langley may publish in the future.

Co-authorship network of co-authors of Emma Langley

This figure shows the co-authorship network connecting the top 25 collaborators of Emma Langley. A scholar is included among the top collaborators of Emma Langley based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emma Langley. Emma Langley is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Langley, Emma, et al.. (2023). Prolactin-induced neuroprotection against excitotoxicity is mediated via PI3K/AKT and GSK3β/NF-κB in primary cultures of hippocampal neurons. Peptides. 166. 171037–171037. 6 indexed citations

Langley, Emma, et al.. (2021). 713 Novel protease activatable linker with tumor targeting motifs enhances the retention of cytokine prodrug and active cytokine at disease site and demonstrates improved efficacy in preclinical model. Regular and Young Investigator Award Abstracts. A742–A742. 1 indexed citations

Lim, Kian‐Huat, Emma Langley, Feng Gao, et al.. (2017). A clinically feasible multiplex proteomic immunoassay as a novel functional diagnostic for pancreatic ductal adenocarcinoma. Oncotarget. 8(15). 24250–24261. 5 indexed citations

McConnell, Melanie J., Emma Langley, Arthur Zelent, et al.. (2015). Post transcriptional control of the epigenetic stem cell regulator PLZF by sirtuin and HDAC deacetylases. Epigenetics & Chromatin. 8(1). 38–38. 10 indexed citations

Park, Silvia, Emma Langley, Jong‐Mu Sun, et al.. (2015). Low EGFR/MET ratio is associated with resistance to EGFR inhibitors in non-small cell lung cancer. Oncotarget. 6(31). 30929–30938. 10 indexed citations

Shah, Payal D., Shanu Modi, Mary Ellen Moynahan, et al.. (2014). Phase I trial of daily PI3Kα inhibitor BYL719 plus letrozole (L) or exemestane (E) for patients (pts) with hormone receptor-positive (HR+) metastatic breast cancer (MBC).. Journal of Clinical Oncology. 32(15_suppl). 2605–2605. 9 indexed citations

Pastorino, Sandra, Sandeep C. Pingle, Emma Langley, et al.. (2014). Abstract 4652: Cerebrospinal fluid pharmacokinetics and pharmacodynamics following high-dose erlotinib treatment in brain cancer patients. Cancer Research. 74(19_Supplement). 4652–4652. 1 indexed citations

Pastorino, Sandra, Emma Langley, Ying S. Chao, et al.. (2014). Mechanisms of resistance to PDGFR inhibition in glioblastoma.. Journal of Clinical Oncology. 32(15_suppl). e13030–e13030. 5 indexed citations

Wang‐Gillam, Andrea, Sachin Wani, Emma Langley, et al.. (2013). Identifying targetable pathways in pancreatic cancer from endoscopic ultrasound-guided fine-needle aspirates (EUS/FNA): Providing a personalized approach to targeted therapy.. Journal of Clinical Oncology. 31(15_suppl). 4051–4051.

10.

Wani, Sachin, Andrea Wang‐Gillam, Dayna S. Early, et al.. (2012). Sa1520 Comparison of Phosphorylation Signatures in Patients With Suspected Pancreatic Cancer Undergoing Endoscopic Ultrasound-Fine Needle (EUS-FNA) With and Without Malignant Cytology: A Pilot Study. Gastrointestinal Endoscopy. 75(4). AB189–AB190. 1 indexed citations

11.

Wani, Sachin, Andrea Wang‐Gillam, Dayna S. Early, et al.. (2012). Su1909 Evaluation of Phosphorylation Signatures in Suspected Pancreatic Cancer Using Endoscopic Ultrasound-Guided Fine Needle (EUS-FNA) Aspirates: A Feasibility Study. Gastroenterology. 142(5). S–533. 1 indexed citations

12.

Dong, Jianying, Stephen J. Demarest, Arlene Sereno, et al.. (2010). Combination of Two Insulin-Like Growth Factor-I Receptor Inhibitory Antibodies Targeting Distinct Epitopes Leads to an Enhanced Antitumor Response. Molecular Cancer Therapeutics. 9(9). 2593–2604. 48 indexed citations

13.

Levashina, Elena A., Emma Langley, Clare Green, et al.. (1999). Constitutive Activation of Toll-Mediated Antifungal Defense in Serpin-Deficient Drosophila. Science. 285(5435). 1917–1919. 369 indexed citations breakdown →

14.

Georgel, Philippe, Christine Kappler, Emma Langley, et al.. (1995). Drosophilaimmunity. A sequence homologous to mammalian interferon consensus response element enhances the activity of the diptericin promoter. Nucleic Acids Research. 23(7). 1140–1145. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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